Scientists have shown that cells in the heart's outer layer can migrate deeper into a failing organ to carry out essential repairs. The migration of progenitor cells is controlled by a protein called thymosin beta 4, already known to help reduce muscle cell loss after a heart attack. The discovery opens up the possibility of using the protein to develop more effective treatments for heart disease. The University College London (UCL) study appears in the journal Nature. Finding out how this protein helps to heal the heart offers enormous potential in fighting heart disease Professor Colin Blakemore Medical Research Council...