Posted on 02/16/2021 9:24:32 AM PST by SeekAndFind
Scientists have found seven separate but similar variants of the coronavirus that have emerged within the United States and appear share a similar genetic mutation to more contagious U.K. and South African types, raising concerns that the U.S. may have to deal with multiple variants of the virus that likely spread more easily.
A preprint study released on Sunday, found that the seven variants—all of which evolved independently—have been spotted in several states.
It is not yet clear if their mutations—which are similar to the more contagious foreign variants—make either of the seven types more contagious, but scientists suspect this is likely the case.
One of the variants, Q677P, was first detected in the U.S. on October 23, yet it accounted for 27.8% cases in Louisiana between December 1 and January 19.
It is, however, unclear if increased presence of the variants was due to its higher transmissibility or if it was aided by holiday travel or other superspreader events.
“There’s clearly something going on with this mutation. I think there’s a clear signature of an evolutionary benefit,” Jeremy Kamil, a virologist at Louisiana State University and one of the study’s co-author told the New York Times.
As the U.S. witnesses a sustained decline in Covid-19 cases, the more contagious U.K. and South African variants remain the biggest concern. Dr. Anthony Fauci, the country’s top infectious disease official, told the New York Times Saturday that the variants were the “big wild card” and warned if people failed to adhere to public health measures, those variants could “take off on us.”
(Excerpt) Read more at forbes.com ...
It seems like it should only be able to mutate so far before it is unable to utilize the ACE2 receptor/and or it kills its host too quickly to transmit it to a new host. At some point its spike protein will achieve maximum affinity for the receptor. At that point, it would be vulnerable to a death blow. As it becomes deadlier and deadlier, it will kill its host before he/she has a chance to transmit it to a bunch of other people and it will burn itself out. Its transmissibility is what makes it deadly right now, not it’s innate lethality. As lethality increases, transmissibility decreases. It will probably mutate to a happy medium where it has increased affinity for the ACE2 receptor and, therefore, a decreased infectious dose, but still isn’t so deadly that it can’t manage to reach 1, 2, or 3 new hosts.
The irony is that the most dangerous thing for these med students is that they won’t get enough sleep—and then they will get into an auto accident!
It looks like the numbers are dropping rapidly...like a normal flu...and does not fit the vaccine model.
we already knew there were many many coronavirus/common cold types so this is nothing new, jus repackaged info to keep people afraid, distant, wesaring masks etc.
What is ADE?
Since the vaccine is related to the protein that permits the virus to attach to the human body, the vaccine is good for all virus minor mutations.
RE: What is ADE?
Antibody-dependent enhancement, sometimes less precisely called immune enhancement or disease enhancement, is a phenomenon in which binding of a virus to suboptimal antibodies enhances its entry into host cells, followed by its replication
And this is why we need that southern border closed!
https://www.thelancet.com/journals/lancet/article/PIIS0140-6736(21)00183-5/fulltext#%20
Resurgence of COVID-19 in Manaus, Brazil, despite high seroprevalence
person gets a virus and develops antibodies
Antibodies kill off the virus and then, over the years, diminish in number
Person comes into contact with the virus again a few years later and, perhaps, it has mutated a bit.
The antibodies now only weakly attach to the virus and, possibly, enhance its ability to get into our cells.
This is seen with Dengue Fever Virus. The second Dengue infection is typically worse than the first and is even sometimes fatal. there are four Dengue phenotypes. It has been postulated that subsequent infection by a different phenotype leads to ADE as the antibodies don't properly bind to the different phenotype and, instead of killing it, they facilitate its entry into human cells.
Recent research has shown with a great degree of certainty that dengue virus ADE occurs when the antibody titer falls to levels within a narrow range. On the other hand, protective benefit is gained from a high antibody titer.
That and for a whole lot of other reasons. P.1. has already been isolated here though. All of the big 3 mutations have been. Recombination is probably inevitable.
Scientists Detect Seven More Coronavirus Variants In U.S. With Similar Mutation To More Infectious U.K. Type
Finally... I was getting sick of the ones we had.
Give everyone Kool-Aid now.
I only here about the new strains being more virulent.
I have never heard any discussion about morbidy differences, if any.
The most successful viruses become more virulent, but less deadly over time, i.e. keep the host alive so he can spread it.
Can someone smart..(and I mean that you know 100% with total certainty)...tell me if the genetic sequence of the SarsCOV-2 virus has been isolated?
I am hearing from more and more sources that it HAS NOT been fully sequenced. In that case, how in the world would they be able to tell one variant from another?
For example, the human genome has been fully sequenced and we can tell one individual from another. I have been told by more than one source that we HAVE NOT sequenced or even isolated the genome of the coronavirus. We are diagnosing infection by the presence of certain protein snippets or certain snippets. How then, could they tell one variant from another.
Someone please set me straight.
‘...but vast knowledge of minor details can definitely cause needless worry’
especially in a gynocentric society; yesterday I’m sitting in a tiny vestibule waiting to get into the gym...while a large gathering room is on the other side of the door, for some reason we can’t go there to wait. I asked a woman, clad in two masks why we can’t sit in the larger room, and she said ‘the virus will spread, and its a death sentance for me...’
I replied to the negative, that if she were healthy, she wouldn’t die, and whe got irritated, saying ‘I don’t want to argue about it.’ I told her we can’t get anywhere if we refuse to discuss things, and she snarled ‘you’re arguing...’ and walked outside to avoid sitting in the vestibule with the likes of me ...
I’m sick to death of these people...
geez... stuck on the kung flu today aren’t you...
covid covid covid is the new russia russia russia...
btw... thanks for helping me create a new tagline...
RE: geez... stuck on the kung flu today aren’t you...
What do you mean today? This thread was started 3 days ago.
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