Posted on 11/27/2009 9:01:25 AM PST by opentalk
THREE Australian experts are making waves in the medical community with a report suggesting swine flu may have developed because of a lab error in making vaccines.
"It could have happened in a lab where somebody became affected and then travelled with it," virologist Dr Adrian Gibbs said yesterday.
Conjuring up a vision of Frankenstein's fictional monster fleeing the laboratory, he added: "Things do get out of labs and this has to be explored. There needs to be more research done in this area.
"At the moment there is no way of distinguishing where swine flu has come from."
The research, published in the Virology Journal on Tuesday, was compiled by two former researchers at the Australian National University - Dr Gibbs and programmer John S. Armstrong.
Dr Jean Downie, once the head of HIV research at Westmead Hospital, was also involved.
The article claimed the swine-origin influenza A (H1N1) virus that appeared in Mexico in April has at least three parent genes which originated in the US, Europe and Asia.
"The three parents of the virus may have been assembled in one place by natural means, such as by migrating birds, however the consistent link with pig viruses suggests that human activity was involved," the research found.
Within two days of them publishing their findings there were more than 16,000 downloads of the article.
"What we wanted to do was instigate debate about this again because we still don't know the source of this virus," Dr Gibbs said.
The research suggested more tests be done on laboratories "which share and propagate a range of swine influenza viruses".
It said that if the virus was generated by laboratory activity it would explain why it had "escaped surveillance for over a decade".
Dr Gibbs said it was not the first time lab errors had been made, with evidence foot and mouth disease in England had been born out of a lab mistake and circumstantial evidence that Spanish influenza in 1918 and Asian influenza in 1957 reappeared decades later because of mistakes.
"Measures to restore confidence include establishing an international framework co-ordinating surveillance, research and commercial work with this virus and a registry of all influenza isolates held for research and vaccine production," the report concluded.
Those are thoughts worth considering...thanks.
Yep, this is also the reason I refuse to get the vaccination or allow any member of my family to get it. A significant number of people who get that vaccine are GOING to get the flu, AND the proteins that are in it are GOING to create more people who develop Anaphylactic Allergic reactions to things like peanuts, because peanut oil is used in the manufacture of it. Its the main reason we have so many kids allergic to Peanuts, the VACCINATIONS and the proteins that are IN them to create a stable compound.
The seasonal flu doesn’t attack the lungs like H1N1 can in some cases....
Swine flu hitting young, healthy adults hardest (Canada)
http://www.theglobeandmail.com/news/national/swine-flu-hitting-young-healthy-adults-hardest/article1203826/
The young and healthy who feel invincible from the H1N1 swine flu influenza pandemic may not be as bulletproof as they think, warn public health experts.
Nearly two-thirds of Canadians hospitalized due to swine flu, and half of those who have died, had no underlying health conditions.
Experts do not yet understand why the new strain affects some healthy people so severely, ravaging their lungs with an aggressive pneumonia and forcing them to spend weeks in hospital, attached to breathing machines.-
Lungs of fatal swine flu patients badly damaged
http://www.ctv.ca/servlet/ArticleNews/story/CTVNews/20090903/h1n1_lungs_090903/20090903
Sep. 3 2009
The lungs of people who have died from swine flu look more like those of the victims of H5N1 avian influenza than those of people who succumb to regular flu, the chief of infectious diseases pathology at the U.S. Centers for Disease Control says.
Study of about 70 fatal H1N1 cases so far also reveals there may be more incidences of co-infections with bacteria than was earlier thought, Dr. Sherif Zaki told The Canadian Press in an interview.
The damage to lung tissue is consistent with that inflicted by ARDS or acute respiratory distress symptom, Zaki says, referring to an often-fatal, difficult-to-treat syndrome that can have a number of causes. The U.S. National Heart, Lung and Blood Institute estimates about 30 per cent of people who develop ARDS die.
In terms of the disease, yes, it (H1N1) is remarkably different than seasonal flu, Zaki says. The pathology looks very similar to H5(N1).
If they were going to go to all this trouble, wouldn't they create and release a super-flu virus?
a report suggesting
swine flu may
could have happened
this has to be explored
needs to be more research
no way of distinguishing
may have been assembled in one place by natural means
instigate debate”
What I see here is a couple of researchers fishing for grant money. It sound more like “In Search Of” starring Leonard Nimoy that a scientific article.
“It could have happened in a lab where somebody became affected and then travelled with it,” virologist Dr Adrian Gibbs said yesterday
&&&
Affected? Sorry, I could not get past that huge error to read any further with confidence in the reporting.
Oh? No one can guess where the lab is that H1N1 originated?
Follow the money.It may be that H1N1 is a national socialist “test” run, for later population control.
( Yes, the nasties now in the White House are perfectly capabable of it, they want to “win” their leftist Utopia that badly.)
Obama investment , curious:
http://keepittrill.com/online/2009/11/barack-obama-invest-shares-company-swine-flu-vaccine/
I am perfectly aware of the peer review process. The language they use is not declarative and they are not reporting scholarly findings. This reads like a letter to the editor which means that it is likely neither peer reviewed nor vetted.
You pinged me, I assumed you wanted my opinion, again, this reads like someone fishing for grant money.
The phylogenetic information presently available does not identify the source of S-OIV, however it provides some clues, which can be translated into hypotheses of where and how it might have originated. Two contrasting possibilities have been described and discussed in this commentary, but more data are needed to distinguish between them. It would be especially valuable to have gene sequences of isolates filling the time and phylogenetic gap between those of S-OIV and those closest to it.
We believe that these important sequences are most likely to be found in isolates from as-yet-unsampled pig populations or as-yet-unsampled laboratories, especially those holding isolates of all three clusters of viruses closest to those of S-OIV, and involved in vaccine research and production. Quarantine and trade records of live pigs entering North America could probably focus the search for the unsampled pig population. It is likely that further information about S-OIV’s immediate ancestry will be obtained when the unusual features of its PB1-F2 gene are understood.
Why create one? Just dig one up.
Like the 1918 variant of H1N1 that was exhumed with victims who were buried in permafrost, and re-created in Jurassic Park like style from RNA found in the victims' bodies.
Tell that to the Chinese that still die from the drug-resistant, super-virulent, cholera that Japan nailed them with before WWII.
Disclaimer: Opinions posted on Free Republic are those of the individual posters and do not necessarily represent the opinion of Free Republic or its management. All materials posted herein are protected by copyright law and the exemption for fair use of copyrighted works.