Posted on 10/18/2021 7:48:32 AM PDT by Red Badger
The right diet is pretty much protection against anything.
https://www.endalznow.org/news/research-shows-intermittent-fasting-may-prevent-alzheimers-disease
We need this process, but it appears to have become dysfunctional in Alzheimer's patients.
I come back, again and again, to the gradual, imperceptual changes a bad diet gives each of us. We may gain weight, or have additional issues inside, which we chalk up to “getting older,” when it was from multiple daily insults to our bodies which we did not overcome.
“Any one care to put that medical science speak into simple English?”
Need a therapy to clean the gunk causing alzheimers off of the nerve endings.
So no real solution except some mysterious unnamed therapy, then?
No argument there. My gma died from it, meaning my father is prime for it (he’s already exhibiting some memory issues).
I can’t tell you how frustrating it is to have both wife & father decline in health because they’re both too stubborn to listen to common sense (perhaps you know).
hmmmm gene therapies
I hope this is for real and not a guise to put forth a new alzheimers vax that does nothing but degrade our t cells. Sorry but how can we trust after this covid debacle
I think they are doing their research in a fashion where they are trying to find a drug to patent. This is not really useful in my eyes. The reality is that Alzheimer’s is both genetically and habitually predictable. In other words you can figure out if you are at risk by looking at your siblings and other family members who are older. If they got it, you are more likely. In my family, every single female on my fathers side (a very large family) has died of Alzheimer’s. And secondly, you can look at those with fatty liver or diabetes 2. They are far more likely to get Alzheimer’s.
Alzheimer’s is a condition where avoidance is far better than a cure. The rate of Alzheimer’s has increased around the world as people start eating the Standard American Diet.
Given that, try not to contract fatty liver. That means staying away from Fructose, maybe alcohol and carbs. High Fructose Corn Starch was the most common way to ingest fructose for the past 40 years. Also fruit juices are very high in fructose.
These researchers are trying to let you eat wrong, get fat, continue your horribly unhealthy life style and take a pill that allows you to avoid Alzheimer’s but get your knees replaced and stay on numerous other pills designed to keep the unhealthy alive.
Oh that explains everything so simply even a cave man could understand!
Here’s the bottom line. The cell organelle that makes the protein that destroys neurons is controlled by an enzyme known as SIGMAR-1. Increase SIGMAR-1 blocks a critical receptor and the toxic protein is blocked from production. Healthy people produce SIGMAR-1 most of their life but it declines with age and severely declines with Alzheimer’s patients.
Currently the leading drug candidate that does exactly this is Anavex Lifesciences AVX-273 clinical trial.
Bingo.
Yeah. Start eating keto, and take it seriously. Every day.
No sugar, no carbs, no alcohol,
Get your weight within guidelines. Then lose 10% more.
Do your own research.
Eat and drink like an idiot and get Alzheimer’s, diabetes, heart disease, debilitating arthritis, kidney failure, etc, OR take your nutrition seriously and live 20 years longer, or more.
Jesus is Lord.
Big patented drug sales potential!
Dr. Robert Malone has talked about the promise of mRNA therapies, but he’s also cautioned of its misapplication. He seems to veer heavily toward the misapplication side of the argument with the COVID stuff, but in time, it’s likely many of our medical therapies will make use of mRNA. There’s promise in it for cancers and things like Alzheimer’s, for sure.
I’ll put it in simple English for you. Wait . . . what was I saying?
“The reality is that Alzheimer’s is both genetically “
I think far yoo many times genetics get blamed when it is really families tend to eat similarly, for better or worse. I also believe Establishment Medicine pushes the genectics angle constantly so that the sheeple think many diseases are inevitable and you can do nothing but rely on them to save you.
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“Any one care to put that medical science speak into simple English?”
They have identified the details of specific steps that occur, in producing the gunk that builds up in brains with Alzheimer’s. They also showed that by blocking one specific step, it would block production of that gunk.
However, that does not necessarily mean that blocking the gunk buildup, will prevent mental decline. Others have reduced the gunk, without reversing the mental decline.
In addition to the buildup of this gunk (Amyloid Beta), there is also a tangling that occurs among brain cells during Alzheimer’s, and inflammatory effects. It is possible that the gunk is just a secondary effect (symptom) of Alzheimer’s, and not the cause.
It was good, detailed research, that convincingly detailed some of the processes involved. So it is a significant step forward in understanding Alzheimer’s - but it is still a long way from a cure, and has not been shown to prevent it.
Researchers want to figure out how to block a key protein called the sigma-1 receptor (S1R) from acting to create amaloid plaque in the brain.
----------------------------------------------------------------------------------------------------------------------- a bit longer version.
Researchers around the world have worked intensely to find ways to block the formation of amyloid beta by preventing cleavage by beta secretase and gamma secretase. However, these approaches have been hampered by safety issues.
a form of APP that has undergone a process called palmitoylation (palAPP) gives rise to amyloid beta.
The new investigation revealed that palAPP is stabilized and prepared for cleavage by beta secretase in special lipid rafts within the neuron known as mitochondria-associated endoplasmic reticulum membranes (MAMs). “We showed for the first time not only that the MAM is where palAPP is processed by beta secretase to make Abeta, but that this happens exclusively in axons and neuronal processes where Abeta does most of its damage,”
They showed for the first time that preventing assembly of MAMs, either with gene therapy or a drug that blocked a key protein called the sigma-1 receptor (S1R), dramatically decreased beta secretase cleavage of palAPP in axons and lowered Abeta production.
(and therefore Amloid plaque.)
Until they figure this out use Turmeric and fish oil!
“Any one care to put that medical science speak into simple English?”
Alzheimer’s is caused by a neurotoxin
😆
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