Coronavirus Takes Aim At Insulin-Producing Cells in the Pancreas

The Stanford Scope ^ | June 15, 2021 | Bruce Goldman

[grey_whiskers]

Diabetics are at heightened risk for COVID-19 complications, it's known. But the connection might run in two directions: COVID-19 may itself kick off or exacerbate diabetes.

A new study shows that SARS-CoV-2, the virus that causes COVID-19, selectively attacks the cells in the pancreas that produce insulin, the critical hormone that regulates our circulating blood-sugar levels. The loss of these pancreatic cells, known as beta cells, is the defining feature of type 1 diabetes, understood to be triggered when the body's immune system, for unknown reasons, launches an unprovoked attack on those cells.

[Bikkuri]

Can’t forget Apple... (or even Micro$uck)

[Jane Long]

LOL...where’s the FAIL cat, when we need it?

https://www.youtube.com/watch?v=Awf45u6zrP0

[Bikkuri]

Huh.. I didn’t know that..

[Bikkuri]

I thought it's name was Paddywhacker, or something like that 😁

[Jane Long]

Looks like this was know, back in Feb.....

https://news.yahoo.com/covid-19-attacks-pancreas-one-212259808.html

[logi_cal869]

As an OCD autodidact, I've done extensive research pursuant to my book to elaborate in layman's terms the true nature of lifestyle disease, not this BS promoted by the AMA et al under the banner of multiple disease conditions.

Without diving down that bunny hole and/or revealing too much: Based upon what I can discern, the virus severely affects haemodynamics by directly inhibiting the normal cellular health of red blood cells, likely some effect of the spike protein, just as we've seen with the adverse effects of those who've gotten the 'shots. Blood clotting is merely a symptom, the true problem being the inability to provide oxygen to the organs: Without good laminar flow, the organs die of hypoxia. i.e. 'organ failure'.

In short, it's all about capillary blood flow or, in the case of literal COVID, lack thereof. Think about it: A possible bioweapon which literally targets the blood in a stealthy manner...a facet which took over a year to discern (or perhaps so-called 'scientists' were too biased to look).

I believe that a link between the virus and the diseased state of those with so-called 'comorbidities' (my preferred term is 'lifestyle disease') will be found to fully explain the causation as it pertains to red blood cells, but it's becoming patently clear that the negligent actions of the medical establishment - enacted by regulatory authorities - killed hundreds of thousands - if not millions - by inhibiting early, effective treatment (e.g. Ivermectin, etc.) to prevent the toxic effects upon diseased individuals, be they diagnosed with a comorbidity or otherwise 'asymptomatic'.

Furthermore, it's all but proven that limiting viral load with effective treatments overwhelmingly prevents the problems associated with the lungs in advanced stage COVID, the lungs merely being another pair of organs being inhibited proper blood flow.

The success of India in the past 2 months - TOTALLY ignored by the media, especially in the US - lends credence to my thoughts, their rate of 'vaccination' not corresponding AT ALL to the incredible drop in both 'cases' and daily deaths.

Furthermore, the observed adverse effects of the 'vaccines' demonstrate that it's less about the virus and the ACE2 receptors than it is about the spike protein itself.

Frankly, I do not believe it is coincidental that a signature clue has been found in the sequencing of the spike protein:

If 'science' actually pulls its head out of its backside and follows the clues the size of - as opposed to viral particles - A HOUSE, somebody please wake me up. Until then, I assure you that I won't be holding my breath...

[tired&retired]

Does the vaccine do this too?

[grey_whiskers]

The article said the spike protein does; so I guess it depends on how much spike protein gets into the pancreas when you get the jab.

[ConservativeMind]

The genetic structure of SARS‐CoV‐2 does not rule out a laboratory origin

https://www.ncbi.nlm.nih.gov/pmc/articles/PMC7744920/