Posted on 03/23/2020 3:59:51 AM PDT by DAC21
For those scratching their head how an antibiotic is showing promise vs. Covid-19 virus here is some science behind it. Interesting and not to difficult of a read versus some Medical studies.
And for the Leftist haters accusing PDT of peddling snake oil by promoting Chloroquine and Azithromycin as a promising treatment go screw yourselves. But before you do, place your order in advance of how you like your Crow prepared.
https://erj.ersjournals.com/content/45/2/428
Thanx for posting facts
One other thing: the ‘mycin’ family of drugs work, for those allergic to penicillin and its derivatives, like me.
Could it be that the most severe pneumonia-like effects of the disease are from a plasmodium secondary infection for which the virus is a gateway?
I would doubt it, malaria is not endemic here.
Nice find.
I believe this is the study that was referenced in this WSJ article.
https://www.wsj.com/articles/these-drugs-are-helping-our-coronavirus-patients-11584899438?mod=hp_opin_pos_3
I understood the general principle right off the bat - it’s not rocket science. The anti-inflammatory properties reduce the symptoms - it doesn’t cure the virus.
All i know, that i know, that i know is that anything penicillin is dangerous for me to ingest.
There is no ‘mycin’ family of drugs. It was a marketing term. For further reading: Penicillin
Macrolide antibiotics are commonly referred to as mycins . Not sure what your point is.
But pneumonia is.
Pneumonia has many causes. Gram negative bacteria, Strep, Legionella, to name a few. By itself pneumonia is meaningless or nearly so. If you were only interested in studying all respiratory illnesses asking Medical Records to give you a list of all cases of pneumonia. In that case we have that blank in the form.
Thanks!!
From 2016...
This is so cool...
Macrolides in critically ill patients with Middle East Respiratory Syndrome
Different bug than WuFlu fortunately.
Azithromycin potentially works by stimulating the host antiviral responses through the induction of interferons (IFNs) and IFN-stimulated genes (ISGs).
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