Posted on 08/07/2007 9:30:37 AM PDT by GodGunsGuts
RIVERSIDE, Calif. – A research team, including UC Riverside biologists, has found experimental evidence that supports a controversial theory of genetic conflict in the reproduction of those animals that support their developing offspring through a placenta.
The conflict has been likened to a “battle of the sexes” or an “arms race” at the molecular level between mothers and fathers. At stake: the fetus’s growth rate and how much that costs the nutrient-supplying mother.
The new research supports the idea of a genetic “arms race” going on between a live-bearing mother and her offspring, assisted by the growth-promoting genes of the father...
(Excerpt) Read more at eurekalert.org ...
Why would the pseudogene be broken in the same place in chimpanzees and humans? Why would the pseudogenes, ERV’s and genetic homology all point to common descent in that they are more similar in species that evidence suggests share a recent common ancestor and more divergent in species that evidence suggests have a less recent common ancestor?
There is no good reason why there would be pseudogenes or ERV’s at all if one is assuming de-novo design. You have yet to come up with a compelling reason why the entire Vitamin C synthase pseudogene sequence wouldn’t simple be absent, let alone why it would be identical between humans and chimpanzees.
Because the article states such.
The study also provides independent support for the theory of conflict between the male and female genetic material in producing offspring.
Starvation is the lack of food.
Common circumstance has not EVER been shown to cause identical mutations.
That is due to the fact that common descent is accepted as the cause. But acceptance cannot logically exclude common circumstance.
Yes. Starvation is the lack of food. What does this have to do with Scurvy.
No. Common circumstance has not been shown to cause identical mutations because many experiments with mutagenesis has repeatedly shown that mutations are random. A recent experiment in mutation and selection for heat tolerance showed that the mutations described reached saturation (i.e. every possible mutation was produced).
Sure it does. The gene that promotes growth is tied directly to the male. You even state so in your "rebuttal."
What does this have to do with Scurvy.
You previously stated the following.
Did God want Brit’s to be called “Limies”, did God think sailors needed scurvy?
Substitute "starvation".
Common circumstance has not been shown to cause identical mutations because many experiments with mutagenesis has repeatedly shown that mutations are random.
So are the rolls of dice. But "7" shows up more times than "2".
TAC CCC GTG GAG GTG CGC TTC ACT CGG GCG GAC GAC ATC CTG CTG AGC CCC PIG TAC CCC GTG GAG GTA CGC TTC ACT CGC GGG GAC GAC ATC CTG CTG AGC CCC BOS TAC CCC GTA GAG GTG CGC TTC ACC CGA GGC GAT GAC ATT CTG CTG AGC CCC RAT TAC CCC GTG GAG GTG CGC TTC ACC CGA GGT GAT GAC ATC CTG CTG AGC CCG MOUSE TAC CCT GTG GGG GTG CGC TTC ACC CGG GGG GAC GAC ATC CTG CTG AGC CCC GUIN PIG TAC CTG GTG GGG GTA CGC TTC ACC TGG AG* GAT GAC ATC CTA CTG AGC CCC HUMAN TAC CTG GTG GGG CTA CGC TTC ACC TGG AG* GAT GAC ATC CTA CTG AGC CCC CHIMPANZEE TAC CCG GTG GGG GTG CGC TTC ACC CAG AG* GAT GAC GTC CTA CTG AGC CCC ORANGUTAN TAA CCG GTG GGG GTG CGC TTC ACC CAA GG* GAT GAC ATC ATA CTG AGC CCC MACAQUE
I did an analysis of the vitamin C mutations quite a while ago. A certain individual, who shall remain unnamed, attempted a rebuttal. But he couldn't count so I have left it to hang splendedly for years. Please note at the position of the *(asterisk), which denotes the missing nucleotide base, there are three letters and the asterisk. Since there are three letters, at least two mutations have occurred at that position. The asterisk is also a mutation, but it can hide more than one mutation. Thus, despite the "rebutters" claim, there are at least 3 mutations at that point(there are only 4 bases so three mutations run the gamut). It is a hot spot.
If humans could make their own vitamin C they wouldn’t get scurvy.
There is a single deletion in that triplet pair denoted by the astrix which leads to a frameshift mutation, making a protein unable to make Vitamin C. The other examples provided do not have a frameshift mutation and make a perfectly functional Vitamin C Synthase protein.
Notice that there are several differences in the sequences (although the chimp and human sequences are identical) not just the astrix. However all Ape sequences have the same frame shift mutation.
What is it about "the genetic material he provided is hard-wired to provide fast fetal growth," that you have difficulty seeing?
If humans could make their own vitamin C they wouldn’t get scurvy.
If humans could make their own food they wouldn't starve.
Notice that there are several differences in the sequences (although the chimp and human sequences are identical) not just the astrix. However all Ape sequences have the same frame shift mutation.
Look again, the chimp and human sequence are different. And I know that there are several differences in the sequences, since I am the one that first introduced the comparison years ago. And as I point out, the frame shift occurs at a hot spot. The rodents differ at that point.
Y L V G V R F T W R Met T S Y Stop= Human and Chimp
Notice that several of the mutations not at the purported “hot spot” did not lead to a difference in the protein sequence? Did you notice that the differences in amino acids due to the mutations NOT at your purported “hot spot” will still make a functional Vitamin C Synthase protein? Notice that the frameshift mutation present in all the Ape species, but not in the other animals, will lead to a STOP codon so that Humans and Chimps cannot make a Vitamin C Synthase gene and are therefore vulnerable to scurvy?
Not one of these codons is identical between all the mentioned species. Are they all mutation hot spots?
Providing fast fetal growth determines the sex of the offspring? Who knew?
Except that you are still wrong on the Human/chimp sequence, of course I did. I analyzed this years ago. The hot spot is still a hot spot. And I know that some mutations appear to be harmless. But those mutations could still code for something that we are, as of this moment, unaware.
The mutation which causes a frame shift in the human sequence occurs at a hot spot. It is obvious.
Having a nice time in LA? The growth factor is tied to the male. He provides the gene. Thus the growth gene is tied to the maleness gene.
BTW -- Bos is cows.
Signals for placental growth has nothing to do with sex determination.
Based upon the fact that a single base position was mutated at least 3 times.
No, not unless you make the statement that blue eyes come from fathers and brown eyes come from mothers, which is equivalent to what this article is stating.
If it made no difference which parent provided which genes than reciprocal crosses would show no difference and a Lyger and a Tigon would look the same. They do not.
Well, this article is not about lygers and tigons. It is about fish. And your above argument seems to indicate that you do tie the parent to the gene.
Look, your argument is that the mutation at point "A" could only come about because of common descent. I show that the particular point "A" is a spot rife with change, so that mutation at point "A" could come about because of environment and not common descent. It is a hot spot.
Disclaimer: Opinions posted on Free Republic are those of the individual posters and do not necessarily represent the opinion of Free Republic or its management. All materials posted herein are protected by copyright law and the exemption for fair use of copyrighted works.