Interesting. My dad was diagnosed with MS in his late 40s with no prior indications. After it progressed rapidly at first it stablized and has seen no major advance for 20 years.
They still have not figured out what initiates it. Though there has been conjecture on pockets of MS - genetic and geographic, they haven’t been able to pinpoint anything definitively. (unless I’ve missed research I’m unaware of).
My dads history and this research to me indicates that there is something causing the attack on the myelin to switch on and then when the initiator is dormant or gone it turns off the attack.
Great news for MS sufferers, including Mrs. LS.
My father-in-law was diagnosed in his 80s. The doctors didn’t seem sure that’s what he had or maybe we didn’t understand the diagnosis, but it was really late in life to develop that illness, we thought. He would have a flare, and then get better, but never quite as well as he was before each flare.
What a terrible disease.
I cal BS. We don't cure diseases anymore. There's too much $$ in treating things.
Wonder if the American Love affair with statins and low fat diets are possible factors here?:
In a recently published article in Biochimica et Biophysica Acta (BBA) Molecular Basis of Disease, Isobel A. Scarisbrick, Ph.D., director of the Neuroregeneration and Neurorehabilitation Laboratory at Mayo Clinic’s campus in Rochester, Minnesota, and co-authors investigate the relationship between fat intake, exercise and myelin production in mice.
The roles that dietary fat intake and other external factors play in the production of oligodendrocytes and OPCs is not well-understood.
Although brain lipids have high fat content, consumption of a diet containing excess fats and sugars has been shown to be detrimental to CNS function. However, myelin assembly requires a significant amount of lipids, and lipids play an important role in glial cell myelination.
Multiple sclerosis will prove to be another disease caused by a particular combination of factors .....a leaky intestinal wall and a porous vagus nerve deliver negative-mimicking bacteria combined with food antigens into the brain’s fourth ventricle. Food antigens cling to nerve myelin, and the immine system creates antibodies to attack them. The bacteria evolve to create negative impressions of the food antigens. The immune system creates antibodies to attack the bacteria. The immune system also attacks myelin because its protein signature is identical to the bacteria’s protein signature.
Incidently, the bacteria and the leaky gut are both caused by ingestion of carbohydrates. Ingesting fat instead goes a long way toward eliminating the problem.
A substantial accumulation of epidemiological and other evidence suggests that varicella zoster, the chicken pox and shingles virus, is responsible for MS. My layman’s guess is that this is correct and that the case will be proved within five years.
I hear the photo of the researches was taken by a Freeper