Posted on 10/29/2020 2:09:47 AM PDT by Oshkalaboomboom
A professor who in an experiment infected himself with Covid-19 to become ill with the virus for a second time says hopes for herd immunity are overblown.
Dr Alexander Chepurnov, 69, first caught coronavirus on a skiing trip to France in February.
After recovering back home in Siberia without requiring hospitalisation, he and his team at the Institute of Clinical and Experimental Medicine in Novosibirsk launched a study into coronavirus antibodies.
They studied 'the way antibodies behaved, how strong they were, and how long they stayed in the body' and found they decrease rapidly, he said.
He said: 'By the end of the third month from the moment I felt sick, the antibodies were no longer detected.
(Excerpt) Read more at dailymail.co.uk ...
What is being ignored is that
Antibodies also come with an expiration date: Because they are inanimate proteins and not living cells, they can’t replenish themselves, and so disappear from the blood just weeks or months after they are produced. Hordes of antibodies appear shortly after a virus has breached the body’s barriers, then wane as the threat dissipates. Most of the B cells that produce these early antibodies die off as well.
But even when not under siege, the body retains a battalion of longer-lived B cells that can churn out virus-fighting antibodies en masse, should they prove useful again. Some patrol the bloodstream, waiting to be triggered anew; others retreat into the bone marrow, generating small amounts of antibodies that are detectable years, sometimes decades, after an infection is over. Several studies, including those led by Dr. Bhattacharya and Dr. Pepper, have found antibodies capable of incapacitating the coronavirus lingering at low levels in the blood months after people have recovered from Covid-19.
Multiple studies, including one published on Friday in the journal Cell, have also managed to isolate coronavirus-attacking T cells from the blood of recovered individuals — long after symptoms have disappeared. When provoked with bits of the coronavirus in the lab, these T cells pumped out virus-fighting signals, and cloned themselves into fresh armies ready to confront a familiar foe. [https://www.nytimes.com/2020/08/16/health/coronavirus-immunity-antibodies.html]
FauXi. Or he at least funds the creation of same.
Nobody ever invented a vaccine for the common cold.
Vaccines have a low efficacy against the flu
It’s the nature of these virus’s
Very fake news-y but my understanding is that it’s fine if antibodies fade ... body remembers ... B and T cells.
I think you’re right. The answer is therapeutics, and dealing with the co-morbidities that make it fatal. That brings us back to heart disease, diabetes, obesity... the real cause of death for so many.
But if it provides five or six months of immunity, an annual shot in Oct or Nov could be useful.
Anyone who wants to travel internationally will have to show proof of immunity via vaccination. That much is a certainty, also nearly every private insurance company who sponsors private health-care for incorporations will almost certainly require their policy holders to require it as a precondition of employment and also in right to work states a condition of continuing employment. There will be no avoiding it. Domestically access to stadiums, mass transit, aircraft could and most likely will require proof for unrestricted access. London Heathrow Airport is already testing digital immunity passports with other EU member states it will be the norm.
Vaccines are the hope? Lol! You go ahead, I’ll pass thanks.
Yes, and how do they even test for something if they can’t even isolate it?
How does this physician know his new infection was of the identical strain as his original? Every year, virologists GUESS about which strain of influenza will predominate, and they issue vaccines based on that guess. Why would this doctor expect his native immunity against one strain of the virus to be effective against a different strain?
His T-cells learned from the antibodies built during the 1st infection and they retained what they learned. They then helped create post-infection new antibodies to fight the virus the second time. Some folks have T-cells that can identity Covid-19 from the last Sars virus that preceded it, and help hit the Covid-19 with new antibodies on an initial infection.
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