"Well I didn't have the money and I thought to myself, "Join the Army"! It's free. So I figured while I'm here I'll lose a few pounds. And you got what, a 6 to 8 week training program here? A real tough one. Which is perfect for me."
Posted on 01/06/2022 5:31:07 PM PST by Hojczyk
An athlete’s muscles need a lot of oxygen when they’re playing, and the heart has to pump more blood to meet the oxygen demand, and it has to beat faster. The rapid heart rate also increases the heart muscles’ oxygen need, and the heart’s blood vessels have to dilate to allow more blood to flow through.
If the heart’s oxygen demand is not met, the cardiac muscles cannot pump blood to the whole body, including the brain, and the brain stops functioning, and the player collapses.
If the player does not get immediate medical attention, the heart muscle dies, and so can the player. Heart attacks result from the lack of oxygen to the heart.
Dr. Santiano MD then explains how “endothelial cells and pericytes in the cardiac blood vessels have to play
Athletes exerting themselves will enter numerous or even continuous phases when their hearts need more oxygen to meet the demand, where increased blood flow is critical.
The study was from the Bristol Medical School in the UK and published in Clinical Science. In brief, the paper showed that the spike protein of the SARS-CoV-2 can disrupt cardiac pericyte functions independent of a COVID19 infection.
The research found that spike proteins by eliciting pro-inflammatory cytokines such as tumor necrosis factor-alpha (TNF-α), interleukin-1 beta, IL-1β, interleukin-6 IL-6, and monocyte chemoattractant protein-1 MCP1. The same cytokines are increased in patients with severe COVID-19 that have cytokine storms. The cytokines then cause damage to the heart muscles and form blood clots.
The spike proteins also stimulate ERK1 and ERK2 and impair the “teamwork” of the pericytes and endothelial cells. The result is the death of endothelial cells. Once it dies, the cardiac blood vessels can not dilate to meet the demands of a rapidly beating heart.
(Excerpt) Read more at granitegrok.com ...
“So stop quoting idiotic theories and acting like an ignorant fool... it is unbecoming.”
Name me a single America college or pro football, baseball or basketball player even track and field who has died of a heart attack while playing in the last 2 years of covid.
Most are vaccinated.
“If you do not understand the difference in activity levels in players of soccer as compared to baseball and football where they are sitting or standing around 90% of the time... it illustrates only your extreme willful ignorance.”
You just answered your own question. It has nothing to do with covid itself but the game.
Explain why soccer players have been dying of heart attacks for the last 50 years. I’m fairly certain covid was not around then.
They also lost me when they showed countess vaccines being administered without aspiration. We're not smart enough to realize it, but we're regressing back to our simian ancestors.
Or shift back into the amateur sports, abandon the status of being a professional.
Except that it is affecting amateurs, kids, high school, college just as much:
https://freerepublic.com/focus/chat/3988541/posts?page=324#324
People generally throw away the part of fruit and vegetables that contain the quercetin anyway. For apples, the major amount is in the skins, and is diminished by processing. Applesauce or pies? Not much in them, the literature indicates.
How many apples do I have to eat every day with the skins on to get the 500 mg of quercetin in the pill I purchase for 11 cents a day? I dunno, but I like to vary my diet a little bit.
I guess I don't see eating as a form of planned self-medication as some folks do.
But thanks for your observations!
Really? Are you that ignorant? Talk about intentionally ignoring reality! There have been hundreds, but I have time this morning to list just a few for you.
Joe Bradshaw, 19, a football player at Charleston Southern University. Collapsed with cardiac arrest.
Emmanual Antwi (18) a Kennedy High footballer collapsed on the field in Sacramento. On-field CPR attempts failed and he died.
Ernesto Ramirez Jr: 16-Year-Old Boy collapsed while playing basketball, and died with a double-size enlarged heart 5 days after his first Pfizer COVID-19 Vaccine on 19 April. From Texas.
Nickolas Lawrinas (17), Footballer died suddenly and unexpectedly after practice. - Florida
Ivan Hicks (16) Footballer died during scrimmage. Coroner found an enlarged heart.
Joshua Ivory (15) Football player collapsed and died during game. Coroner's report said cardiac dysrhythmia triggered sudden cardiac arrest.
Stephen Sylvester (15), Detroit Central Catholic High School football and track athlete collapsed and died during conditioning practice.
Tirrell Williams (19) Fort Scott freshman lineman died after collapsing with a stroke on field during practice.
Quandarius Wilburn (19), Football player collapsed during a Panthers conditioning practice and later died.
Azorean Tatum (16), high school football player suddenly collapsed at school. Died on August 21.
Dimitri McKee (17) Lee High School Football player passed out and died after practice.
Jack Alkhatib (18), Columbia High school footballer collapsed on the field and died.
Donadrian Robinson (Donnie) (17), Columbia High school footballer died on August 21 after practice.
There are hundreds more reported by the media and despite most being taken down by Google you can still find them. They pretty much all have one thing in common... Covid vaccines shortly before.
And it's not just kids and athletes... my little brother in his mid 50s who had been an airline captain for decades with no history of heart issues was grounded after getting the Pfizer. He and other airline pilots all get physicals twice a year. As soon as he mentioned the Pfizer the “flight surgeon” went looking for difficulties with his heart. He had no symptoms but it was found that he had a swollen and inflamed heart. He was told that he would never fly again because the damage already done was permanent and made it much more likely to die from sudden cardiac failure.
He went to several other doctors and specialists, and they all told him the same thing. But when his airline had to start cancelling hundreds of flights because so many pilots had been grounded... he received a call and told to go to a different flight surgeon in another state. Bingo, he was back in the air a week later. If you feel completely comfortable as a passenger in a jet aircraft piloted by people with heart conditions that could kill them at any moment but especially when a stressful situation arises... I salute you. I love my brother and am glad that he is happily pulling in the big bucks again, but something feels a little weird about it.
There has been an increase of over 200% in a year. Some sources say much higher than that.
Oh and I am sorry that most of the kids that I listed were in high school, but I am not going to bother to go back and list the college and beyond aged players. You are just not worth the effort. Sorry.
“Oh and I am sorry that most of the kids that I listed were in high school, but I am not going to bother to go back and list the college and beyond aged players”
You do not think the rampant drug use of youth today is not a factor? Youth who drink energy drinks like water?
The obesity in todays kids? I played HS football. A handful of the guys were fat and they were the linemen.
Have you seen photos of kids in HS sports today..heck even the cheerleaders?..they are all fatsos.
Have you seen American youth? Fat fat fat.
They are finding teens and young adults today have artery disease like a 60 year old. They have grown up on nothing but pizza, fast food. and processed foods.
There is more to this story than covid.
One more thing about todays youth.
Have you see where the military is toying with a pre boot camp?
They cannot find enough fit normal weight recruits so they are looking at allowing the fat to join but first they will go through a weight watchers style of boot camp to lose the weight before going onto normal boot camp.
sad.
"Well I didn't have the money and I thought to myself, "Join the Army"! It's free. So I figured while I'm here I'll lose a few pounds. And you got what, a 6 to 8 week training program here? A real tough one. Which is perfect for me."
“Hey Eisenhower which way to the mess hall?”
One of the funniest lines in all military movies.
My BIL was stationed at Ft Benning at the time and he took us to see Stripes at a base movie theatre
I can still remember the crowd laughing uncontrollably over this line.
Dude, they’re WRONG. All the time. The mRNA doesn’t stay in the arm.
The ‘side effects’ are deadly and common. Wake up. LOL
I have been a huge proponent of vaccines for my entire life. But these are an obvious failure. Far more death and injury than all others combined in 30 years by far and efficacy that is in the toilet when it comes to omicron or any new other new “variant”. But all these problems are from fat kids, yada, yada, yada. Keep living in fantasy land.
The mRNA is injected into your body encapsulated by a lipid nanopartical. If it weren’t protected by this enclosure, the mRNA particle would be quickly destroyed by antibodies. The lipid nanopartical has been proven by clinical trials to be safe. If you understood its chemistry, you would say it was safe. It is what makes the mRNA technology work.
Yaaaan! I must get some sleep . . .
—jonrickvanwinkle46 😉
I’m not sure if you’re serious or not. No one is saying the lipid capsule isn’t safe.
It’s the mRNA code transported by the lipid nanoparticle that isn’t safe, particularly when it infects cells in sensitive parts of the body, like the ovaries, brain, heart etc. The code forces your cells to produce a toxin — the spike protein.
Do you get it? Not sure why you’re struggling with this.
Endothelial expression of MHC class II molecules in autoimmune disease
https://pubmed.ncbi.nlm.nih.gov/14754393/
Excerpt from the abstract:
Increased endothelial MHC class II expression has been demonstrated in several autoimmune diseases, including myocarditis with dilated cardiomyopathy, rheumatoid arthritis (RA) and systemic lupus erythematosus (SLE).
Recent data suggest that there is an association between endothelial expression of MHC class II molecules and diffuse endothelial dysfunction, which may be part of the explanation of the increased risk of cardiovascular disease in patients with RA, SLE and other chronic inflammatory conditions.
MHC class II transcription is in part genetically determined. Cytokine induced up-regulation of MHC class II molecules can be inhibited in vitro by antioxidants and different drugs, such as cyclosporin and statins.
Research on the development of new treatments for systemic autoimmune diseases and cardiovascular disease should include evaluation of effects on endothelial activation, including MHC class II expression.
This review also discusses the genetic basis of MHC class II expression and its implications for understanding MHC genotype associations with autoimmune diseases.
The severe acute respiratory syndrome coronavirus 2 (SARS-CoV-2) causes a broad range of clinical responses including prominent microvascular damage.What are pericytes?
The capacity of SARS-CoV-2 to infect vascular cells is still debated.
Additionally, the SARS-CoV-2 Spike (S) protein may act as a ligand to induce non-infective cellular stress.
We tested this hypothesis in pericytes (PCs), which are reportedly reduced in the heart of patients with severe coronavirus disease-2019 (COVID-19).
Here we newly show that the in vitro exposure of primary human cardiac PCs [pericytes] to the SARS-CoV-2 wildtype strain or the α [alpha] and δ [Delta] variants caused rare infection events.
Exposure to the recombinant S protein alone elicited signalling and functional alterations, including:
(1) increased migration,
(2) reduced ability to support endothelial cell (EC) network formation on Matrigel ["Matrigel resembles the laminin/collagen IV-rich basement membrane extracellular environment found in many tissues and is used by cell biologists as a substrate (basement membrane matrix) for culturing cells" - Wikipedia],
(3) secretion of pro-inflammatory molecules typically involved in the cytokine storm, and
(4) production of pro-apoptotic factors [mostly proteins having a role in apoptosis] causing EC [endothelial cell] death. Next, adopting a blocking strategy against the S protein receptors angiotensin-converting enzyme 2 (ACE2) and CD147, we discovered that the S protein stimulates the phosphorylation/activation of the extracellular signal-regulated kinase 1/2 (ERK1/2) through the CD147 receptor, but not ACE2, in PCs [pericytes].
The neutralisation of CD147, either using a blocking antibody or mRNA silencing, reduced ERK1/2 activation, and rescued PC [pericyte] function in the presence of the S protein.
Immunoreactive S protein was detected in the peripheral blood of infected patients.
In conclusion, our findings suggest that the S protein may prompt PC [pericyte] dysfunction, potentially contributing to microvascular injury.
This mechanism may have clinical and therapeutic implications.
Pericytes are cells present at intervals along the walls of capillaries (and post-capillary venules).What is CD147?
In the CNS, they are important for blood vessel formation, maintenance of the blood–brain barrier, regulation of immune cell entry to the central nervous system (CNS) and control of brain blood flow.
Classically, the blood flow increase evoked by neuronal activity, which focuses the oxygen and glucose supply on active neurons and generates BOLD functional imaging signals, was thought to reflect relaxation of smooth muscle cells around arterioles.
Recently, however, it has become clear that much of the flow increase is generated by dilation of capillaries, rather than of arterioles (which dilate more slowly), and that this is achieved by a relaxation of contractile pericytes.
Pericytes also play a key role in pathology: in ischaemia [restriction of blood supply,] pericytes constrict capillaries, trapping blood cells, which prevents microcirculatory reperfusion [restoration of blood flow] after clot removal in stroke.
This has made pericytes an important therapeutic target.
In late 2019, the betacoronavirus SARS-CoV-2 was identified as the viral agent responsible for the coronavirus disease 2019 (COVID-19) pandemic.
Coronaviruses Spike proteins are responsible for their ability to interact with host membrane receptors and different proteins have been identified as SARS-CoV-2 interactors, among which Angiotensin-converting enzyme 2 (ACE2), and Basigin2/EMMPRIN/CD147 (CD147).
CD147 plays an important role in human immunodeficiency virus type 1, hepatitis C virus, hepatitis B virus, Kaposi's sarcoma-associated herpesvirus, and severe acute respiratory syndrome [SARS] coronavirus infections.
In particular, SARS-CoV recognizes the CD147 receptor expressed on the surface of host cells by its nucleocapsid protein binding to cyclophilin A (CyPA), a ligand for CD147.
However, the involvement of CD147 in SARS-CoV-2 infection is still debated.
Interference with both the function (blocking antibody) and the expression (knock down) of CD147 showed that this receptor partakes in SARS-CoV-2 infection and provided additional clues on the underlying mechanism:CD147 binding to CyPA does not play a role;Altogether, these findings suggest that CD147 is involved in SARS-CoV-2 tropism and represents a possible therapeutic target to challenge COVID-19. ["tropism refers to the way in which different viruses/pathogens have evolved to preferentially target specific host species, specific tissue, or specific cell types within those species" - Wikipedia]
CD147 regulates ACE2 levels and both receptors are affected by virus infection.
That is because they tend to blood clot and covid (virus or vaccine) is a blood clotting agent. There are relatively thin low metabolism people, and one sees athletes with high metabolism dying of the vaccine just as the obese do.
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