Posted on 04/08/2023 9:11:16 AM PDT by ConservativeMind
Since the emergence of COVID-19, much remains unknown, including the widespread organ damage beyond just the lungs. Increasingly, scientists are learning organ dysfunction results from damage to the blood vessels, but why is unclear. Now a team has discovered what they believe is the key molecular pathway.
Results show COVID-19 damages the cells lining the smallest blood vessels, choking off blood flow.
"We were surprised by the little overlap between our adult and pediatric patients," says Cheryl Maier, MD, Ph.D. "One pathway that stood out in adults was related to vessel health and blood flow."
Maier says this was particularly interesting given their observations that blood from patients severely ill with COVID-19 was unusually viscous: think maple syrup rather than water.
Since the earliest days of the pandemic, physicians have seen that a blood protein called fibrinogen was extremely elevated in patients with severe COVID-19. The body forms blood clots in part by cutting fibrinogen to form fibrin, a key component of clots, but fibrinogen itself is not thought to form clots and levels are not affected by anticlotting medications.
The researchers found that in COVID-19 patients, the sky-high levels of fibrinogen cause red blood cells to clump together, altering blood flow and directly damaging the endothelial glycocalyx, a gelatinous protective layer lining the microvessels. "Fibrinogen is one of the top proteins in plasma," Maier says. "It's been hiding in plain sight."
Taken together, these data suggest fibrinogen-induced red blood cell aggregation and resulting microvascular damage could be the major pathway by which COVID causes organ damage and even death.
There's presently no medications targeting high fibrinogen in the blood. However the team has done exploratory research using therapeutic plasma exchange: removing plasma with high fibrinogen from COVID-19 patients and replacing it with donor plasma that has normal fibrinogen levels.
(Excerpt) Read more at medicalxpress.com ...
I’ll post a study on each, next.
Nattokinase decreases plasma levels of fibrinogen, factor VII, and factor VIII in human subjects
https://pubmed.ncbi.nlm.nih.gov/19358933/
Serratiopeptidase, A Serine Protease Anti-Inflammatory, Fibrinolytic, and Mucolytic Drug, Can Be a Useful Adjuvant for Management in COVID-19
https://www.frontiersin.org/articles/10.3389/fphar.2021.603997/full
Thanks for this article.
Thank you for all this great information you provide.
Interesting. I do seem to remember that there was debate about whether patients should be put on blood thinners in early stages of the disease. I can understand concerns about risk. Maybe those supplements would lower the typical risks.
Thanks for the links.
So just like they said in “Died Suddenly.”
Damn you authoritarian globalist corporatist scum to hell. This is mass murder.
Pathways, apparently made more agressive, per multiple studies, via the risky $hots :-(
FLCCC (real) docs/scientists have been recommending nattokinase, as part of their post-jab (and, china virus) protocol, for months.
https://covid19criticalcare.com/treatment-protocols/i-recover/
It is a shame that these $hots do not prevent/protect, but, appear to increase odds of contracting china (and, other) viruses.
The mechanisms of action of ivermectin against SARS-CoV-2—an extensive review
https://www.nature.com/articles/s41429-021-00491-6
a surprising number of ways ivermectin combats covid...
After reading the article I surfed the internet looking for the supplements and found this one which contains nattokinase and serrapeptase.
Ivermectin for Prevention and Treatment of COVID-19 Infection: A Systematic Review, Meta-analysis, and Trial Sequential Analysis to Inform Clinical Guidelines
Conclusions: Moderate-certainty evidence finds that large reductions in COVID-19 deaths are possible using ivermectin. **Using ivermectin early in the clinical course may reduce numbers progressing to severe disease.** The apparent safety and low cost suggest that ivermectin is likely to have a significant impact on the SARS-CoV-2 pandemic globally
https://pubmed.ncbi.nlm.nih.gov/34145166/
Great find!
Will buy some for dear friends/fam who took these horrid $hots.
Dr. Roger Seheult was exploring and discussing the role of fibrinogen, fibrin, and endothelial cells back in April 2020. His daily “COVID Updates” covered it. He recognized it is more of a vascular disease than a respiratory disease. The damage to the alveoli in the lungs inhibits gas exchange.
If serrapeptase is too strong for someone, bromelain or quercetin can also do this to a degree.
I couldn’t tell, did they segregate those who had the Jab and covid from the non Jabbed covid group. IOW, were they looked at separately or just anyone who had it?
Is there a way to determine of an individual is actually effected?
It was vaccine agnostic.
Then for me at least it is of no value. There is very reasonable evidence of blood issues with the shot. There needs to be that separation of the virus with and without the shot or the research loses much of its validity. Simply put, it's bad science not to make more effort to isolate pertinent variables.
That's one of the most important things in research, isolating for potentially relevant variables. Otherwise the conclusions are not much better than anecdotal. How do we know that "unJabed" people who had covid are equally experiencing these blood issues?
Thanks.
BKMK
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