Orly Taitz did it first.
And with exactly the same success rate.
#birftards = #Qtards
PING
Fauci funded this bioweapon.
As another Freeper said.....”THE VACCINE WAS NOT CREATED FOR COVID......COVID WAS CREATED FOR THE VACCINE”!!!!!!THINK ABOUT THAT!!!!!
Fred Corbin did not kill himself next week......................
Orly Taitz did it first.
And with exactly the same success rate.
#birftards = #Qtards
Isn’t bitchute Q stuff?
“Two mutations were critical for bat to human transmission of Middle East Respiratory Syndrome (MERS) Coronavirus.”
“We recognize HKU4 spike, aiming to build its capacity to mediate viral entry into human cells. To this end, we introduced two single mutations.”
“S76R and S76A into HKU4 spike."
I would like to read that document in its entirety.
A racecar engineer in Barbados?
Did this guy also get the “Data Packets” the Q team has been looking for?
This smells like a big pile of BS.
.
The Covidians are already hard at work on the keywords...
Why isn’t that database posted online then?
Quite a claim. He’d best be prepared to prove it (which will get him cancelled and banned.)
bump
* * *
PING
PING
Sterilized and/or dead.
Looks like we have leaked documents from Pfizer and a china data base?
bkmk
However, we also have MUCH more, including US scientific reports with clear descriptions of exactly who and how new SARS-COV viruses were engineered in the lab, with deliberate production of SARS-COV viruses that were extremely capable of infection in humans.
For example, here is an article published in PNAS (Proceedings of the National Academy of Sciences of the United States of America) March 14, 2016
https://www.pnas.org/content/113/11/3048
“This manuscript describes efforts to extend surveillance beyond sequence analysis, constructing chimeric and full-length zoonotic coronaviruses to evaluate emergence potential. Focusing on SARS-like virus sequences isolated from Chinese horseshoe bats, the results indicate a significant threat posed by WIV1-CoV. Both full-length and chimeric WIV1-CoV readily replicated efficiently in human airway cultures and in vivo, suggesting capability of direct transmission to humans.”
And from the results summary in this article:
Using the SARS-CoV infectious clone as a template (7), we designed and synthesized a full-length infectious clone of WIV1-CoV consisting of six plasmids that could be enzymatically cut, ligated together, and electroporated into cells to rescue replication competent progeny virions (Fig. S1A). In addition to the full-length clone, we also produced WIV1-CoV chimeric virus that replaced the SARS spike with the WIV1 spike within the mouse-adapted backbone (WIV1-MA15, Fig. S1B). WIV1-MA15 incorporates the original binding and entry capabilities of WIV1-CoV, but maintains the backbone changes to mouse-adapted SARS-CoV. Importantly, WIV1-MA15 does not incorporate the Y436H mutation in spike that is required for SARS-MA15 pathogenesis (8). Following electroporation into Vero cells, robust stock titers were recovered from both chimeric WIV1-MA15 and WIV1-CoV.
And the connection to Wuhan Labs in China is highlighted in the Acknowledgments:
We thank Dr. Zhengli-Li Shi of the Wuhan Institute of Virology for access to bat CoV sequences and plasmid of WIV1-CoV spike protein. Research was supported by the National Institute of Allergy and Infectious Disease and the National Institute of Aging of the NIH under Awards U19AI109761 and U19AI107810 (to R.S.B.), AI1085524 (to W.A.M.), and F32AI102561 and K99AG049092 (to V.D.M.). Human airway epithelial cell cultures were supported by the National Institute of Diabetes and Digestive and Kidney Disease under Award NIH DK065988 (to S.H.R.). Support for the generation of the mice expressing human ACE2 was provided by NIH Grants AI076159 and AI079521 (to A.C.S.).
And an interesting side note: National Institute of Allergy and Infectious Disease (NIAID) is managed by Dr. Anthony Fauci, in case you were wondering if he had any connection to this.
My assessment is that the PNAS article indicates strongly that NIH National Institute of Allergy and Infectious Disease, managed br Dr. Anthony Fauci, and the Wuhan Institute of Virology, managed by Dr. Zhengli-Li Shi cooperated to create what ultimately became the chimeric virus SARS-CoV-2 (Covid-19) pandemic.
And finally: Chinese and US scientists genetically engineered bat coronaviruses in dangerous gain-of-function research stretching back years.
Excerpt from this article:
Research was omitted from landmark paper claiming natural origin of SARS-CoV-2. Report: Claire Robinson.
Chinese and US scientists have been collaborating for years in dangerous gain-of-function experiments that involve genetically engineering coronaviruses from bats and other animals, as revealed by a series of scientific publications.
The coronaviruses are related to the SARS viruses that cause severe respiratory diseases in humans. The scientists were based at the Wuhan Institute of Virology (WIV) in China, the lab suspected by some of accidentally releasing the SARS-CoV-2 virus that caused the COVID-19 pandemic, and at the University of North Carolina (UNC) in the US.
Oddly, however, this long and high-profile research history was entirely omitted from the scientific paper, published in Nature in February this year, in which Shi Zhengli and her team at the WIV claimed to have identified a natural origin for SARS-CoV-2. The origin, according to the WIV scientists, was a bat virus, RaTG13, that was thought to have jumped from an animal to a human at a Wuhan seafood and wildlife market (the “zoonotic” theory – that is, coming from animals by a natural spillover event).
Why the omission? To understand the possible reason, we need to first understand the nature of the research work that was done by the WIV scientists and their US collaborators.
The purported benign aim of this line of research was to investigate the potential of bat coronaviruses to infect humans, to improve scientists’ ability to predict pandemics, and to develop vaccines or other therapies.
However, this is also gain-of-function research, which aims to make viruses more infective or transmissible. Such research has come under increasing criticism by scientists for many years, due to its tendency to pose huge risks for little benefit.
This fear is borne out by the results of a particularly risky gain-of-function experiment carried out in the US and published in 2015 by scientists from the UNC in collaboration with WIV scientists, including Shi Zhengli, dubbed China’s “bat woman” for her work with bat coronaviruses. The work was funded by: * The National Institute of Allergy & Infectious Disease (NIAID) of the US National Institutes of Health (NIH). The director of the NIAID is Dr Anthony Fauci, who currently heads up the US COVID-19 response. The NIH’s money was directed through the US-based Eco-Health Alliance, headed by Dr Peter Daszak; * USAID; and * Chinese institutions.
In the published paper reporting the risky experiment, the scientists state that they began their work before the 2014 US temporary moratorium on virus gain-of-function studies, which was prompted by several high-profile biosafety failures at US labs. But in spite of the moratorium, as stated in the paper, the NIH gave permission for the study to continue. Dr Fauci of the NIAID “outsourced” the research to the WIV in China, in the words of one media article.
Alarming finding
In the experiment, the scientists took a mouse coronavirus and exchanged its spike protein – the part on the surface of the virus that determines infectivity – for one from a bat coronavirus that was similar to the virus that causes the human epidemic disease SARS. They kept the mouse virus “backbone” – its basic RNA and protein molecular structure. The bat coronavirus, in its natural state, was unable to infect humans as its spike protein was inadequate – it was not able to dock onto the ACE2 receptor on human cells.
Infectivity is supposed to be determined just by the spike protein. So joining the bat spike protein with the mouse virus backbone should have resulted in a virus that was non-infectious to humans
. But the resulting genetically engineered chimeric virus unexpectedly turned out to be highly infectious to humans. In fact, its infectivity, tested in human airway cells, was comparable to the human epidemic-causing virus strain SARS-CoV Urbani.
(end of excerpt, but there is much more...)