[Myrddin]

SARS and MERS had poor binding affinity for ACE2 receptors in humans. It was primarily lung tissue ACE2. SARS-CoV-2 was engineered with an "improved" spike protein with better binding affinity to ACE2 in all human tissues. In addition, the protein sequence generated by the viral RNA was updated with a cleavage site (a location on a protein where a protease cuts a protein) that utilize of "furin-like" geometry to take advantage the more ubiquitous availability of that protease in human cells.

[grey_whiskers]

Its also noteworthy that the sequence of nucleotides in COVID, used the codons more common in humans, than in coronaviruses, to signal for the amino acids in the cleavage site.

Nobel Prize winner David Baltimore (discovered reverse transcriptase at MIT) called this a “smoking gun” for bioengineering of the coof.

[Paco]

Uh huh, yeah, the next time you explain something try using the English language...