Does Hillary have Progressive Supranuclear Palsy? -vanity

Yahoo ^ | Sept 22, 2016 | Self

[Future Useless Eater]

Hillary demonstrates here that she can move her eyes quickly side-to-side, but she may have a GREAT DEAL OF TROUBLE rolling her EYES UP, (or down).

That could be why she had to arch her back, and tip her head side-to-side to watch the balloons falling.

In contrast to her, Bill has no problem looking up just by rolling his eyes UP in his eye sockets.

Hillary's eye problem may also help explain why, when reporters shoved a recording device beneath Hillary's chin she flinched and flung her head backwards, because she couldn't move her eyes DOWN to see the recorder, so it surprised her.

Her vertical eye movement disorder may be "Supranuclear Ophthalmoplegia", which is the cardinal manifestation of "Progressive Supranuclear Palsy" (PSP), which is a degenerative brain disorder.

I suggest that many of you COLLECT and SAVE whatever pertinent findings you can about this condition, because Hillary's troops have a habit of scrubbing the internet of any medical information that is damaging to her cause.

Here is some text I lifted from Parkinsons.org...

Progressive Supranuclear Palsy (PSP) is slightly more common than ALS (also called Lou Gehrig disease). Symptoms usually begin in the early 60s. Common early symptoms include forgetfulness, personality changes, and loss of balance while walking that results in unexplained falls. The visual problems associated with PSP generally occur three to five years after the walking problems and involve the inability to aim the eyes properly because of weakness or paralysis of the muscles that move the eyeballs.

I have also saved all of the Wikipedia text as-of today since her storm-troopers will likely tamper with that soon.
Here is a portion of that text, with some possibly-pertinent portions that I highlighted. [my comments in brackets and italics]

Progressive Supranuclear Palsy

Progressive Supranuclear Palsy (PSP) is a degenerative disease involving the gradual deterioration and death of specific volumes of the brain.

Symptoms and signs

The initial symptoms in two-thirds of cases are loss of balance, lunging forward when mobilizing, fast walking, bumping into objects or people, and falls.

Other common early symptoms are changes in personality, general slowing of movement, and visual symptoms.

Later symptoms and signs are dementia (typically including loss of inhibition and ability to organize information), slurring of speech, difficulty swallowing, [which can lead to coughing fits and aspiration pneumonia]
and difficulty moving the eyes, particularly in the vertical direction. The latter accounts for some of the falls experienced by these patients as they are unable to look up or down.

Some of the other signs are poor eyelid function, contracture of the facial muscles, a backward tilt of the head with stiffening of the neck muscles, sleep disruption, urinary incontinence and constipation.

The visual symptoms are of particular importance in the diagnosis of this disorder. Patients typically complain of difficulty reading due to the inability to look down well. ... Involuntary eye movement [side-to-side as the head is turned],

may be closer to normal. On close inspection, eye movements called "square-wave jerks" may be visible when the patient fixes at distance. These are fine movements, that can be mistaken for nystagmus, except that they are saccadic in nature, with no smooth phase. [This may explain the limp/overshoot side-to-side eye movements she displayed in Philly]

Difficulties with convergence (convergence insufficiency), where the eyes come closer together while focusing on something near, like the pages of a book, is typical. Because the eyes have trouble coming together to focus at short distances, the patient may complain of diplopia (double vision) when reading. [Remember: her fresnel lens glasses were for double vision]

Cardinal manifestations:
  Supranuclear ophthalmoplegia [the inability to roll her eyes UP]
  Neck dystonia [involuntary muscle contractions that can result in slow repetitive movements, like her bobble-head imitation]
  Parkinsonism [a group of neurological disorders that mimics Parkinson’s disease, tends to progress more rapidly than Parkinson’s, and presents with additional symptoms such as early falling, dementia or hallucinations. Drug-induced Parkinsonism is usually a side effect of drugs that affect dopamine levels in the brain, such as antipsychotics, some calcium channel blockers and stimulants like amphetamines and cocaine.]
  Pseudobulbar palsy
  Behavioral and cognitive impairment
  Imbalance and walking difficulty
  Frequent falls

Differential diagnosis
PSP is one of a number of diseases collectively referred to as Parkinson plus syndromes. Also, patients with the Richardson variant tend to have an upright or arched-back posture as opposed to the stooped-forward posture of other Parkinsonian disorders. Early falls are characteristic, especially with Richardson-syndrome.

Pathophysiology
The principal areas of the brain affected are:
  the basal ganglia, particularly the subthalamic nucleus, substantia nigra and globus pallidus;
  the brainstem, particularly the portion of the midbrain where "supranuclear" eye movement resides;
  the cerebral cortex, particularly that of the frontal lobes;
  the dentate nucleus of the cerebellum;
  and the spinal cord, particularly the area where some control of the bladder and bowel resides.

Classification and treatment
There is no known cure for PSP and management is primarily supportive. PSP cases are often split into two subgroups, PSP-Richardson, the classic type, and PSP-Parkinsonism, where a short-term response to levodopa can be obtained. After a few years the Parkinsonian variant tends to take on Richardson features.

Due to the progressive nature of this disease, all individuals eventually lose their ability to walk and will need to progress to using a wheelchair. Severe dysphagia [difficulty swallowing that leads to coughing fits and aspiration pneumonia] often follows, and at this point death is often a matter of months.

Prognosis
There is currently no effective treatment or cure for PSP, although some of the symptoms can respond to nonspecific measures. The average age at symptoms onset is 63 and survival from onset averages 7 years with a wide variance. Pneumonia is a frequent cause of death.

[EVO X]

Not saying it is Parkinson’s, but that freeze at the bollard sure follows the script. If there is a bunch of commotion going on and the person is in an unfamiliar area, it may prolong the freeze according to literature.

[Psalm 144]

“This fits her symptoms, and implies she is close to death’s door.”

Well, she needs to hang on to the door frame for a few more weeks.

[lizma2]

I’m still betting on post stroke or Parkinson’s or both.

PSP often look’s like Parkinson’s in the beginning. But PSP as far as I remember has rigidity of the spine and neck, jerky eye movements and eye lid problems. Haven’t seen that yet but I’ll keep watching.

They also don’t have tremors but she keeps her hand to her chest a lot. People with tremors do that. (Parkinson)

She went from emergency prisms in her glasses, to a pair of glasses with permanent prisms (The eyes didn’t seem to match up but the light reflects did) to no glasses with occasional a wonky eye. What ever she has it’s seems to be improving. (DANG!) That points to more a cerebral-vascular event.

Please keep me in the loop and I’ll keep pickin my brain.

[sport]

I agree.

[RockyTx]

Useless...

Thanks for posting this!

I think you nailed it.

Rocky

[Darnright]

Thank you for posting this. A beloved elderly relative was just diagnosed with this disease. As it is extremely rare, there is very little research and no effective treatment. It falls into the “orphan” group of rare diseases.

Prayers appreciated for this dear, 90 year old lady.