Posted on 08/26/2011 1:12:38 PM PDT by Swordmaker
Thanks for the info. However, have they actually tested GSE?
The only reason to use toothpaste is for the fluoride, to strengthen your enamel. So the other purposes have not been investigated. The claims for Neem are exaggerated as people in India where they chew The twigs, don't have any better teeth than else where. The head dentist at my office was the keynote speaker at the Indian Society of Oral Implantology in Mumbai, India, this January. He found the dentist there were fighting the same bacteria and far worse cases of Yaws, carries, and gum disease.
2. Any reason I couldn’t mix my toothpaste with the baking soda at each brushing instead of using straight baking soda?
3. My gums don’t ever bleed while brushing, but when I go for my cleanings they always tell me that my gums bleed “excessively”. Do you know if there’s a difference between the two as far as possible damage from spirochetes?
Mixing the baking soda with toothpaste is not a good Idea. The baking soda needs to be packed into the gum line. Mixing with toothpaste would not do that. read my answer just before this reply as to how it kills the bacteria.
If your hygienist is telling you have bleeding gums, it is most important that you do this. Just because you don't see blood when you brush does not mean your gums are not bleeding. The area of your gums being raw and oozing is the equivalent to the palm of your hand being a raw open wound! Think about that. That's how much potential area is exposed to allowing these bacteria into your bloodstream!
Thank you for the info. I will not use fluoride; I think in general people are getting too much of it between toothpaste and water supply. I stopped using fluoridated tooth paste about a year ago and also have a filter that I use with the tap water at work that removes fluoride.
The taste of the baking soda makes me gag, probably because I’m pregnant. So I’ve been putting a thin line of toothpaste on the brush and then piling on the baking soda, work in to one area and then repeat. The little bit of mint flavor really helps.
Congratulations on the coming baby! My elder daughter is about to have my first grandchild... so I share your excitement.
You will get used to the flavor and grow to like it, but in the meantime, that is a good approach. Just make sure you work the baking soda into the gums between your gums and teeth and also floss it in between... they like to hide there, too. DO IT for your children, so you'll be around and aware for their babies.
Thanks! And congratulations to you!
Researchers and pharma companies have tried to attack this disease by reducing amyloid plaques, but inflammation may be the real culprit.
To investigate whether IL-1 might play a role in Lewy body pathology seen in Alzheimer's, we tested the role of IL-1 in alpha-synuclein fiber production using three methods: in tissue culture, in IL-1-pellet implanted rat brains, and in brain slices from Alzheimer’s patients. All three methods gave the same results, showing that IL-1 overexpression was associated with increased production of alpha-synuclein
http://the-scientist.com/2011/09/01/what-causes-alzheimers/
A nice link IL-1 and spirochetes:
Interleukin- 1 gene polymorphisms as assessed in a 10-year study of patients with early-onset periodontitis.
http://www.ncbi.nlm.nih.gov/pubmed/17575917 and
Salivary interleukin-1beta concentration and the presence of multiple pathogens in periodontitis.
Thanks for the links. I appreciate them. Dr. Miklossy points out in her paper that these oral spirochetes are capable of triggering the human immune response for their OWN defense. After reading the first paper on inflammation, and it’s involvement, I wonder if that is not part of the AD modality?
Yes, it is a strong possibility
Just to let you know, I started your protocol immediately upon reading this thread when it was first posted along with a friend to whom I sent the link, and we have BOTH had awesome checkups since then. The protocol may surely help with AD and other diseases, but it is fantastic for plain old regular daily oral hygiene.
We both are VERY grateful. I can’t imagine brushing my teeth with toothpaste ever again.
Guess I missed this thread the first time around...but your statement sounds like a classic coincidental correlation. Correlation is not causation, I'm sure you've heard that lol. Think of all the other things in diet, etc. that changed about the same time.
Steve, yes, there were other factors, but nothing else changed that correlates as well.
We’ve tracked this as the use of toothpaste as a dentifrice was exported to foreign markets, and its substitution for dental powders (90% sodium bicarbonate and its totally coincidental bactericidal benefits) spread and the ensuing 20-30 year lagging growth of the same plaque related diseases in those populations where they did not change diets or other aspects of their lifestyles. . . and where those diseases were not endemic before.
You can’t tell me that these diseases have followed this same pattern everywhere that tasty, pasty sweetened pumice dentifrice (which has zero anti-bacterial ability) has supplanted the use of baking soda as a dentifrice with no major change in cultural dietary norms, yet 20-30 years later that population experiences an identical increase on heart disease, adult onset diabetes, age related senility, and alzheimers and that is not significant???
Steve, yes, there were other factors, but nothing else changed that correlates as well.
We’ve tracked this as the use of toothpaste as a dentifrice was exported to foreign markets, and its substitution for dental powders (90% sodium bicarbonate and its totally coincidental bactericidal benefits) spread and the ensuing 20-30 year lagging growth of the same plaque related diseases in those populations where they did not change diets or other aspects of their lifestyles. . . and where those diseases were not endemic before.
You can’t tell me that these diseases have followed this same pattern everywhere that tasty, pasty sweetened pumice dentifrice (which has zero anti-bacterial ability) has supplanted the use of baking soda as a dentifrice with no major change in cultural dietary norms, yet 20-30 years later that population experiences an identical increase on heart disease, adult onset diabetes, age related senility, and alzheimers and that is not significant???
If you have had Lyme disease, I implore you to read the books by Dr. Ritchie Shoemaker (works on the Eastern Shore of Maryland, MD from Duke University).
He says there are a number of syndromes (including mold toxicity and some lyme diseases) that are due to fat-soluble toxins released from the foreign organism which induce cytokine and other hormonal responses in the body, and affect visual nerves first.
He has quite a bit of clinical data to back it up, including detailed HLA (human leukocyte antigen, which controls the human immune response) mappings for some 25% of the population who are more susceptible to the toxins and generally have difficultly clearing them.
His remedy is to prescribe (off-label usage) the old cholesterol drug cholestyramine which binds bile salts and causes them to be removed in the waste. The idea being that as your body attempts to replace the bile salts, the fat-soluble toxins (which would otherwise be uselessly recirculated throughout the body and cause problems) become bound to the cholestyramine and eliminated.
I know it sounds like quackery but he uses rigorous testing (case-by-case basis, not double-blind experiments followed by statistical massage) and has a wealth of clinical *results* backing him up.
Cheers!
Oral spirochetes implicated in dental diseases are widespread in normal human subjects and carry extremely diverse integron gene cassettes.
http://www.ncbi.nlm.nih.gov/pubmed/22635997
The psychoimmunology of lyme/tick-borne diseases and its association with neuropsychiatric symptoms.
http://www.ncbi.nlm.nih.gov/pubmed/23091569
The oral microbiome in health and disease.
http://www.ncbi.nlm.nih.gov/pubmed/23201354
Evidence for graft colonization with periodontal pathogens in lung transplant recipients. A pilot study.
http://www.ncbi.nlm.nih.gov/pubmed/22203528
Simultaneous detection of periodontal pathogens in subgingival plaque and placenta of women with hypertension in pregnancy.
http://www.ncbi.nlm.nih.gov/pubmed/21830010
Emerging roles of pathogens in Alzheimer disease.
http://www.ncbi.nlm.nih.gov/pubmed/21933454
Exploring new biological functions of amyloids: bacteria cell agglutination mediated by host protein aggregation.
http://www.ncbi.nlm.nih.gov/pubmed/23133388
Periodontal vaccine.
http://www.ncbi.nlm.nih.gov/pubmed/22406716
A “nice” video:
Want to see how bad it can get? Spirochetes in a dying mouth.
http://www.youtube.com/watch?v=gxQZFZzc2Dw
That's one of our videos with our head dentist narrating. . .
Determining the presence of periodontopathic virulence factors in short-term postmortem Alzheimer's disease brain tissue.
Demonstration of a known chronic oral-pathogen-related virulence factor reaching the human brains suggests an inflammatory role in the existing AD pathology.
http://www.ncbi.nlm.nih.gov/pubmed/23666172
Effect of periodontal therapy on the subgingival microbiota over a 2-year monitoring period. I. Overall effect and kinetics of change.
CONCLUSION:
Periodontal therapy leads to a rapid reduction in periodontal pathogens, followed by a slower reduction in other taxa that can be sustained for at least 2 years.
http://www.ncbi.nlm.nih.gov/pubmed/23710672
Altered oral viral ecology in association with periodontal disease.
http://www.ncbi.nlm.nih.gov/pubmed/24846382
The next is an important study
The connection between ruptured cerebral aneurysms and odontogenic bacteria.
CONCLUSIONS:
This is the first report showing evidence that dental infection could be a part of pathophysiology in intracranial aneurysm disease
http://www.ncbi.nlm.nih.gov/pubmed/23761916
Oral biofilms, oral and periodontal infections, and systemic disease.
http://www.ncbi.nlm.nih.gov/pubmed/24479275
Invasion of oral and aortic tissues by oral spirochete Treponema denticola in ApoE(-/-) mice causally links periodontal disease and atherosclerosis.
This is the first study examining the potential causative role of chronic T. denticola periodontal infection and vascular atherosclerosis in vivo in hyperlipidemic ApoE(-/-) mice. T. denticola is closely associated with periodontal disease and the rapid progression of atheroma in ApoE(-/-) mice. These studies confirm a causal link for active oral T. denticola infection with both atheroma and periodontal disease.
http://www.ncbi.nlm.nih.gov/pubmed/24566627
Atherosclerosis and Alzheimer - diseases with a common cause? Inflammation, oxysterols, vasculature
We postulate that AD and ATH are both caused by chronic immunologic challenge that induces CH25H expression and protection against particular infectious agents, but at the expense of longer-term pathology.
free article
http://www.ncbi.nlm.nih.gov/pmc/articles/PMC3994432/
And you read it here first. Didn’t know FR was a scientific site, did you? Heheheh
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