Cognitive Dysfunction Reversed in Mouse Model of Down Syndrome

ScienceDaily ^ | Nov. 19, 2009 | ScienceDaily

[bogusname]

Now, findings from the Stanford University School of Medicine and Lucile Packard Children's Hospital shed light on the neural basis of memory defects in Down syndrome and suggest a new strategy for treating the defects with medication. The study, which was conducted in mice, is the first to show that boosting norepinephrine signaling in the brains of mice genetically engineered to mimic Down syndrome improves their cognition. Norepinephrine is a neurotransmitter that nerve cells use to communicate...

[Leftism is Mentally Deranged]

Mouse brain is not human brain (current members of congress excluded)