[grey_whiskers]

Money quote, from further down:

""Traditional first-line therapy has been based on the belief that the bacteria are bad, so we have to get rid of them as quickly as possible," McCullers said. "But what we are finding is that maybe it is the inflammation we need to worry about first, and the bacteria second. Protein synthesis inhibitors shut down the bacterial protein-making factory, and they can avoid the inflammatory burst by killing them over days instead of quickly lysing them."

This may help limit deaths due to pneumonia following infection by flu...

Cheers!

[grey_whiskers]

Like, *PING*, folks.

Some good news from an unexpected quarter...

Cheers!

[metmom]

ping

[SunkenCiv]

Thanks grey_whiskers.

[yefragetuwrabrumuy]

This has some problems. To start with, secondary bacterial pneumonia is more typical of normal influenza, and they specify young children and the elderly-infirm which again are typical.

However, the radically different influenza for which there is little or no immunity or partial immunity, has different rules. Its primary victims are those from about age 20-45, with healthy and strong immune systems. This is because their immune system itself attacking the virus, not secondary bacteria, is what is destroying their lungs.

For these young and healthy people, it is essential that lethal Acute Respiratory Distress be prevented in the first place. And there are some very positive things that can be done right now to fend off ARD during an epidemic.

The first thing is to eliminate possible sources of Arsenic from your environment. Small amounts of Arsenic, over time, have been found to inhibit the immune system pathogen recognition system. So in the case of influenza, first the body doesn’t recognize the disease, and then it overreacts to it, causing ARD.

Fortunately, the EPA has strict limits on Arsenic in city water, so only rural wells present a drinking water - influenza risk. But 90% of the other Arsenic in our environment is used as wood preservative.

Statin drugs, normally used for high cholesterol, like Lipitor and Crestor, have been found to strongly inhibit ARD.

[Myrddin]

My first serious pneumonia following flu was treated with erythromycin. It certainly hit the bacterial population hard, but the secondary effects were a fever spike to 103.5 that persisted for 3 weeks. Some of the bacteria that persisted were proteolytic and left the back of my throat looking like it had been cut deeply with a knife along the drainage path from sinuses to lungs. The garbage in my lungs was a mix of blood, pus and mucous with a very acidic characteristic. It took almost 3 months for my lungs to completely clear. After that pneumonia, my lung capacity was never very good again. I tried very hard to excel at inline speed skating, but always ended up hypoxic when I pushed my limits.

The study results seem to mirror the advice for treatment of anthrax as well. A slow, steady kill of the germinated organisms achieves the objective without dumping a large volume of toxins into the patient at one time. The antibiotics that cause a breach of the bacterial wall dump the endotoxins out in one broad step. Many of the fever producing toxins are endotoxins. That likely explains my own experience.