That will be good, if it works.
Could me amazing for so many people!
This would be amazing having an HF autistic daughter, but I wonder about/any long term effects.
Thanks SeekAndFind.
*PING* to LJ
Just wait until the “Anti-Abelist” Lobby finds out about this!
I have heard of families where both parents were deaf, they were born that way, and it could not be changed. They give birth to a child with the same defect, only now, in year 2023, this can be easily ‘fixed’. I’ve heard of such parents who will refuse to allow that child to get their hearing repaired, because then the child won’t be a part of the deaf community, and maybe the child won’t love them anymore. Sad, but believable.
It’s Lamictal there’s a pic on dailymailuk
Used for bipolar disorder and schizophrenia as mood stabilizer
My own research showed that the brain mutation happened during the third month of pregnancy. A shift in consciousness processing ability in the thalamus blocks amygdala processing and diverts it all intp the hippocampus. It’s a shift from feminine consciousness, normal for a developing child, prematurely to the masculine which normally happens around age seven.
It’s interesting as it often causes the person to turn their head to the left to optimize input from the right peripheral optical field into the left cerebral hemisphere, thus increasing comprehension.
bttt
I wonder if it would impact folks who might be borderline Aspergers.
Marked for later reading
$3 per pill today. If and when FDA approval comes for human use...that will skyrocket. Yes R&D is expensive. But so is greed.
Ironically, Zoomers are trying to find doctors to diagnose them with Autism.
Bookmarking
I bet the pharma-bro, Martin Shkreli, is wishing he’d waited for this one.
It has been said that autism is the undeveloped brain’s form of schizophrenia. Therefore when one encounters a drug which works against autism one should ask if it also works against schizophrenia. Sure enough, a search of “schizophrenia lamotrigine” turns up some promising results.
https://link.springer.com/article/10.1007/s00213-005-0020-9?
Conclusions
The efficacy of lamotrigine is most likely explained within the framework of a glutamate neuron dysregulation hypothesis, and may arise primarily through the drugs ability to influence glutamate transmission and neural activity in the cortex. The drug is likely to act through inhibition of voltage-gated sodium channels, though other molecular interactions cannot be ruled out. Lamotrigine may add to or synergise with some atypical antipsychotic drugs acting on glutamate transmission; alternatively, they may act independently on glutamate and dopamine systems to bring about a combined therapeutic effect. We propose new strategies for the treatment of schizophrenia using a combination of anti-dopaminergic and anti-glutamatergic drugs.
I have an autistice son.
He’s on lamatrigine for seizers already.
Guess what?
He still has autism.
Don’t get your hopes up.
I have an autistice son.
He’s on lamotrigine for seizers already.
Guess what?
He still has autism.
Don’t get your hopes up.
Jill places order