Posted on 01/06/2022 5:31:07 PM PST by Hojczyk
An athlete’s muscles need a lot of oxygen when they’re playing, and the heart has to pump more blood to meet the oxygen demand, and it has to beat faster. The rapid heart rate also increases the heart muscles’ oxygen need, and the heart’s blood vessels have to dilate to allow more blood to flow through.
If the heart’s oxygen demand is not met, the cardiac muscles cannot pump blood to the whole body, including the brain, and the brain stops functioning, and the player collapses.
If the player does not get immediate medical attention, the heart muscle dies, and so can the player. Heart attacks result from the lack of oxygen to the heart.
Dr. Santiano MD then explains how “endothelial cells and pericytes in the cardiac blood vessels have to play
Athletes exerting themselves will enter numerous or even continuous phases when their hearts need more oxygen to meet the demand, where increased blood flow is critical.
The study was from the Bristol Medical School in the UK and published in Clinical Science. In brief, the paper showed that the spike protein of the SARS-CoV-2 can disrupt cardiac pericyte functions independent of a COVID19 infection.
The research found that spike proteins by eliciting pro-inflammatory cytokines such as tumor necrosis factor-alpha (TNF-α), interleukin-1 beta, IL-1β, interleukin-6 IL-6, and monocyte chemoattractant protein-1 MCP1. The same cytokines are increased in patients with severe COVID-19 that have cytokine storms. The cytokines then cause damage to the heart muscles and form blood clots.
The spike proteins also stimulate ERK1 and ERK2 and impair the “teamwork” of the pericytes and endothelial cells. The result is the death of endothelial cells. Once it dies, the cardiac blood vessels can not dilate to meet the demands of a rapidly beating heart.
(Excerpt) Read more at granitegrok.com ...
It is the nature of the retrovirus. The virus cannot multiply by itself. It must send its RNA to your cells, and then your cells will multiply its RNA and will also turn its RNA into more virus proteins.
In my case, ever since I have had my thyroid removed, I have had zero colds or flu bouts for the past 20 years. All of a sudden, the dreaded stomach flu of winter or feverish bouts are no more. Reactions to flu vaccines, none, and same with small pox injections which create zero scabbing my case.
The medical field has this colonist mentality. It won’t want to hear about your medicine or patient testimony, they want to sell you THEIR pills. This is so obvious.
But we know that now. There are pre-treatments:
-PJ
Read later.
That’s very interesting.
Medical science knows a whole lot less about the human body than it pretends to and when they encounter someone with a really unusual condition, they don’t even want to bother.
Been there, doing that.
During my searching around, I have not run across any reports of the symptoms for Coxsackievirus infection.
I do think, that there is some probability that, some of the vaccines are leading to the SARS-CoV-2 Spike Protein being “expressed” on the surface of some cells that
a) are NOT supposed to be participating as host cells, and/or
b) are participating in locations (heart, kidney examples),
that may lead to significant concentrations of inflammation.
That inflammation happening, because the immune system *record keeping* (some portion thereof, that stores patterns) may be
c) considered by the immune system, to be at fault along with,
or
d) found by the immune system, to be a bit confused when discovering,
said participants sporting the Spike Protein(s).
Scenario:
Immune system finds a SARS-CoV-2 Spike Protein at the surface of what otherwise has been recorded to be a Known Good One [type of] Cell.
Immune system re that Cell, does one of the following
e) tosses out the “Known Good One” record status, or
f) temporarily sets a bypass switch to ignore the “Known Good One” record status, or
g) makes no change to the “Known Good One” record status;
and then, the immune system either
h) attends to the Spike Protein, or
i) attends to the combination of the Spike Protein and participant cell.
IF the participant cell is NOT protected by some recorded status (Known Good One), then, possibly, an auto-immune event may occur, either temporarily or as an initial incident for a longer haul.
For conditions (c) and (d) above, the immune system memory storage system may have some Storage First Aid process that cleans, resets/restores the particular memory storage area.
This scenario, is a way for me to explain what I have been observing among reports by people re their personal experience and symptoms.
How does the Spike Protein leave the cell after it is created by the ribosomes? The ribosomes of the dendrite cells will translate the messenger RNA into the Spike Protein which is broken up by the protezome and presented on the MHC-1 and MHC-2 complex molecules on the surface of the host cell’s membrane. The Spike Protein is completely contained and no way does it float around in the blood stream. Those who say the Spike Proteins are running around in the blood stream don’t know what they are talking about.
Sit down, relax and watch the video:
https://www.youtube.com/watch?v=SQztlqblgVI
How does the Spike Protein leave the cell after it is created by the ribosomes?
Hmm . . . Re that consideration, I did not propose that; nor have I proposed that the Spike Proteins are running loose.
I set up conditions for my Scenario . . . “and then, the immune system either”
[clarifying a bit, my writing:]
h) attends to the Spike Protein (ie ignoring the host cell), or
i) attends to the combination of the Spike Protein and participant cell (not ignoring the host cell).
That said, I am watching Mobeen Syed, MD’s video - tx.
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