Posted on 04/13/2007 5:23:54 PM PDT by wagglebee
The organization points to a new report from the American Cancer Society that cancer deaths declined slightly in 2003 and 2004.
But Karen Malec, the head of the Coalition, told LifeNews.com "There would be fewer cancer cases and deaths if women had been told the truth in the 1980s when conclusive evidence became available" about the abortion-breast cancer link.
She said studies at that time pointed to the association between breast cancer and abortion as well as hormone replacement therapy and oral contraceptives.
The new ACS report said it expects 40,460 female deaths due to breast cancer and 240,510 total breast cancer cases for 2007.
However, it continues to mislead women about the abortion-breast cancer link saying "Several studies have provided very strong data that induced abortions have no overall effect on the risk of breast cancer."
Yet, Malec points to a scientific review in 2005 that concluded the studies ACS mentions are seriously flawed and cannot be used to dismiss the larger body of evidence supporting an abortion-breast cancer link.
No scientist has challenged these conclusions, she told LifeNews.com in a statement.
She pointed to research from Baruch College professor Dr. Joel Brind, who is one of the leading scientists examining the data. Brind has found that having an induced abortion leaves a woman with a higher long-term risk of breast cancer, compared to carrying the pregnancy to term.
"We call on the Society and other cancer businesses to put their priorities in order," said Malec. "Women's lives and cancer prevention are more important than making money, doing cancer walks, and protecting the abortion and pharmaceutical industries."
She indicated that more abortions means more cancer cases and also condemned the Susan G. Komen Foundation for its donations of hundreds of thousands of dollars to Planned Parenthood, which is the nation's leading abortion business.
"It's unthinkable that groups that claim to want to eradicate the disease would help fund a cancer-causing organization, especially when the funds could be directed to legitimate health organizations," Malec said.
Related web sites:
Coalition on Abortion/Breast Cancer - http://www.abortionbreastcancer.com
At least two of these links are from women’s health groups that also support abortion.
One of you links was the “Feminist Women’s Health Center” which I would hardly call an “impartial” source. They also provide all sorts of abortion information.
http://www.fwhc.org/abortion/index.htm
You originally posted, "Strange how smoking, which doesnt show any direct link to cancer gets so much attention,"
Now you change your statement/argument to the following.
"Most studies have found no link between active cigarette smoking and breast cancer."
So which is it? Is there NO link between smoking and cancer, or just smoking and breast cancer?
Wagglebee, as you know, the website organizations did not do the research themselves. They are quoting from unrelated researchers.
I can’t speak to the veracity of each study - you said you were unaware of any studies refuting the link, and I gave you some that do so.
However, you are going to believe whatever you want in spite of not having good evidence to back up you opinion.
Scientists have determined that boys exposed to estrogen hormones in the abortifacient birth control pill while in utero are at a greater risk of developing prostate cancer and other urinary tract problems later in life.
Estrogen-like hormones deform the prostates in developing embryos, which can be a precursor to developing prostate cancer or bladder problems, according to research conducted by Frederick vom Saal of the University of Missouri in Columbia, Missouri. These chemicals [mimic] extremely potent synthetic sex hormones, strong enough to completely control an adult womens reproductive system, vom Saal told New Scientist.com. The developing fetus is extremely sensitive to chemical disturbance so exposing a male baby to them is a very bad idea.
Vom Saal and his team exposed mice to ethinylestradiol, the hormone found in the birth-control pill. He found that the prostate glands had an increase in the number and size of prostate ducts and a narrowing of the bladder neck. They compared the effect of the hormone to diethylstilbestrol (DES) a known (banned) cancer-causing agent once used to treat women at risk for miscarriages.
As many as two million women become pregnant each year while taking the abortifacient birth-control pill, many of whom continue to take the drug for several months before realizing they are pregnant.
Are you suggesting that abortion is “safe” for women and carries no health risks?
You should read some of the information from the link at the bottom of the story.
Oral contraceptives (OCs) first became available to American women in the early 1960s. The convenience, effectiveness, and reversibility of action of birth control pills (popularly known as the pill) have made them the most popular form of birth control in the United States. However, concerns have been raised about the role that the hormones in OCs might play in a number of cancers, and how hormone-based OCs contribute to their development. Sufficient time has elapsed since the introduction of OCs to allow investigators to study large numbers of women who took birth control pills for many years.
This fact sheet addresses only what is known about OC use and the risk of developing cancer. It does not deal with other serious side effects of OC use, such as the increased risk of cardiovascular disease for certain groups of women. Recently, alternative methods of delivering hormones for contraception have been developed, including a topical patch, vaginal ring, and intrauterine delivery system, but these products are too new to have been tested in clinical trials (research studies) for long-term safety and other effects (1). They also are not covered in this fact sheet.
Currently, two types of OCs are available in the United States . The most commonly prescribed OC contains two man-made versions of natural female hormones (estrogen and progesterone) that are similar to the hormones the ovaries normally produce. This type of pill is often called a combined oral contraceptive. The second type of OC available in the United States is called the minipill. It contains only a type of progesterone.
Estrogen stimulates the growth and development of the uterus at puberty, causes the endometrium (the inner lining of the uterus) to thicken during the first half of the menstrual cycle, and influences breast tissue throughout life, but particularly from puberty to menopause.
Progesterone, which is produced during the last half of the menstrual cycle, prepares the endometrium to receive the egg. If the egg is fertilized, progesterone secretion continues, preventing release of additional eggs from the ovaries. For this reason, progesterone is called the pregnancy-supporting hormone, and scientists believe that it has valuable contraceptive effects. The man-made progesterone used in OCs is called progestogen or progestin.
Because medical research suggests that some cancers depend on naturally occurring sex hormones for their development and growth, scientists have been investigating a possible link between OC use and cancer risk. Researchers have focused a great deal of attention on OC users over the past 40 years. This scrutiny has produced a wealth of data on OC use and the development of certain cancers, although results of these studies have not always been consistent. The risk of endometrial and ovarian cancers is reduced with the use of OCs, while the risk of breast and cervical cancers is increased (1). A summary of research results for each type of cancer is given in Questions 25.
A womans risk of developing breast cancer depends on several factors, some of which are related to her natural hormones. Hormonal factors that increase the risk of breast cancer include conditions that may allow high levels of hormones to persist for long periods of time, such as beginning menstruation at an early age (before age 12), experiencing menopause at a late age (after age 55), having a first child after age 30, and not having children at all.
A 1996 analysis of worldwide epidemiologic data conducted by the Collaborative Group on Hormonal Factors in Breast Cancer found that women who were current or recent users of birth control pills had a slightly elevated risk of developing breast cancer. The risk was highest for women who started using OCs as teenagers. However, 10 or more years after women stopped using OCs, their risk of developing breast cancer returned to the same level as if they had never used birth control pills, regardless of family history of breast cancer, reproductive history, geographic area of residence, ethnic background, differences in study design, dose and type of hormone, or duration of use. In addition, breast cancers diagnosed in women after 10 or more years of not using OCs were less advanced than breast cancers diagnosed in women who had never used OCs. To conduct this analysis, the researchers examined the results of 54 studies. The analysis involved 53,297 women with breast cancer and 100,239 women without breast cancer. More than 200 researchers participated in this combined analysis of their original studies, which represented about 90 percent of the epidemiological studies throughout the world that had investigated the possible relationship between OCs and breast cancer (2).
The findings of the Womens Contraceptive and Reproductive Experiences (Womens CARE) study were in contrast to those described above. The Womens CARE study examined the use of OCs as a risk factor for breast cancer in women ages 35 to 64. Researchers interviewed 4,575 women who were diagnosed with breast cancer between 1994 and 1998, and 4,682 women who did not have breast cancer. Investigators collected detailed information about the participants use of OCs, reproductive history, health, and family history. The results, which were published in 2002, indicated that current or former use of OCs did not significantly increase the risk of breast cancer. The findings were similar for white and black women. Factors such as longer periods of use, higher doses of estrogen, initiation of OC use before age 20, and OC use by women with a family history of breast cancer were not associated with an increased risk of the disease (3).
In a National Cancer Institute (NCI)-sponsored study published in 2003, researchers examined risk factors for breast cancer among women ages 20 to 34 compared with women ages 35 to 54. Women diagnosed with breast cancer were asked whether they had used OCs for more than 6 months before diagnosis and, if so, whether the most recent use had been within 5 years, 5 to 10 years, or more than 10 years. The results indicated that the risk was highest for women who used OCs within 5 years prior to diagnosis, particularly in the younger group (4).
How do oral contraceptives affect ovarian and endometrial cancer risk?
Studies have consistently shown that using OCs reduces the risk of ovarian cancer. In a 1992 analysis of 20 studies of OC use and ovarian cancer, researchers from Harvard Medical School found that the risk of ovarian cancer decreased with increasing duration of OC use. Results showed a 10 to 12 percent decrease in risk after 1 year of use, and approximately a 50 percent decrease after 5 years of use (5).
Researchers have studied how the amount or type of hormones in OCs affects ovarian cancer risk reduction. One of the studies used in the Harvard analysis, the Cancer and Steroid Hormone Study (CASH), found that the reduction in ovarian cancer risk was the same regardless of the type or amount of estrogen or progestin in the pill (6). A more recent analysis of data from the CASH study, however, indicated that OC formulations with high levels of progestin reduced ovarian cancer risk more than preparations with low progestin levels (7). In another recent study, the Steroid Hormones and Reproductions (SHARE) study, researchers investigated new, lower-dose progestins that have varying androgenic properties (testosterone-like effects). They found no difference in ovarian cancer risk between androgenic and nonandrogenic pills (8).
OC use in women at increased risk of ovarian cancer due to BRCA1 and BRCA2 genetic mutations has been studied. One study showed a reduction in risk, but a more recent study showed no effect (9, 10).
The use of OCs has been shown to significantly reduce the risk of endometrial cancer. This protective effect increases with the length of time OCs are used, and continues for many years after a woman stops using OCs (11).
Evidence shows that long-term use of OCs (5 or more years) may be associated with an increased risk of cancer of the cervix (the narrow, lower portion of the uterus) (12). Although OC use may increase the risk of cervical cancer, human papillomavirus (HPV) is recognized as the major cause of this disease. Approximately 14 types of HPV have been identified as having the potential to cause cancer, and HPVs have been found in 99 percent of cervical cancer biopsy specimens worldwide (12). More information about HPV and cancer is available in Human Papillomaviruses and Cancer: Questions and Answers at http://www.cancer.gov/cancertopics/factsheet/risk/HPV on the Internet.
A 2003 analysis by the International Agency for Research on Cancer (IARC) found an increased risk of cervical cancer with longer use of OCs. Researchers analyzed data from 28 studies that included 12,531 women with cervical cancer. The data suggested that the risk of cervical cancer may decrease after OC use stops (13). In another IARC report, data from eight studies were combined to assess the effect of OC use on cervical cancer risk in HPV-positive women. Researchers found a fourfold increase in risk among women who had used OCs for longer than 5 years. Risk was also increased among women who began using OCs before age 20 and women who had used OCs within the past 5 years (14). The IARC is planning a study to reanalyze all data related to OC use and cervical cancer risk (12).
Several studies have found that OCs increase the risk of liver cancer in populations usually considered low risk, such as white women in the United States and Europe who do not have liver disease. In these studies, women who used OCs for longer periods of time were found to be at increased risk for liver cancer. However, OCs did not increase the risk of liver cancer in Asian and African women, who are considered high risk for this disease. Researchers believe this is because other risk factors, such as hepatitis infection, outweigh the effect of OCs (15).
Studies have found that regular breast cancer screening with mammograms reduces the number of deaths from breast cancer for women ages 40 to 69. Women who are at increased risk for breast cancer should seek medical advice about when to begin having mammograms and how often to be screened. A high-quality mammogram, with a clinical breast exam (an exam done by a professional health care provider), is the most effective way to detect breast cancer early.
Abnormal changes in the cervix can often be detected by a Pap test and treated before cancer develops. Women who have begun to have sexual intercourse or are age 21 should check with their doctor about having a Pap test. Researchers are working on developing screening tests for ovarian and endometrial cancer.
Women who are concerned about their risk for cancer are encouraged to talk with their health care provider. More information is also available from the Cancer Information Service
Where do you feel that I implied anything even close to that?
I just think it is rotten to use specious evidence to bolster a cause, no matter how noble the cause itself may be.
Strange how smoking, which doesnt show any direct link to cancer gets so much attention, but the pill doesnt cause any concern at all.
You're joking, right? If not, where did you get the idea that there is no direct link between cigarette smoking and cancer?
By all means, tell me what the direct cancer causing agent in cigarette smoke is. After you FAIL to name it then you will know-
where I get my “idea” from.
I wish someone besides Brind would take this on. When I saw Brind 12 years ago he was very impressive. When I saw him 2 years ago he looked a bit unhinged. His Baruch College webpage lists no scientific paper published after 1996.
Mrs VS
The CDC doesn't agree with you. And that's just the first site that came up on search. I'm sure I could give you several more, but it scarcely seems necessary.
To quote a sample from the link, "Cigarette smoking is a major cause of lung cancer, the leading cause of cancer death in both men and women in the United States. Worldwide, lung cancer kills over one million people each year.2 Tobacco smoke contains more than 60 cancer-causing chemicals (carcinogens).
From the same link above
“Tobacco-specific nitrosamines (TSNAs) are a class of known carcinogens that are formed during the curing, processing, fermentation, and combustion of tobacco.4,5 They have been identified in cigarette tobacco,6 tobacco smoke,4 environmental tobacco smoke,7 smokeless tobacco8 and other tobacco products such as cigars and bidi cigarettes.9,10
There are seven known TSNAs. N’-nitrosonornicotine (NNN), (4-methylnitrosamino)-1-(3-pyridyl)-1-butanone (NNK), and N-nitrosoanatabine (NAT) generally occur in greater quantities in tobacco products than the others and NNN, NNK and N-oxide, 4-(methylnitrosamino)-1-(3-pyridyl N-oxide)-1-butanol (NNAL), a metabolic product of NNK, are clearly the most carcinogenic.11 The TSNAs, NNN, and NNK are categorized as “reasonably anticipated to be human carcinogens” by the National Toxicology Program.12 The International Agency for Research on Cancer has classified NNN and NNK as “possibly carcinogenic to humans” (Group 2B).)13”
smoking doesn’t cause cancer? How so?
The link you're trying to make is related to pregnancy ... but not the pill. Women who have baby(ies) late in life or never have a baby increase their likelihood of breast cancer.
So the pill and abortion DO increase your risk of breast cancer ... but so do miscarriage or never getting pregnant at all.
OMG Nathan! How could you make such a statement? Smoking doesn't cause cancer in ALL smokers, but it's still a risky habit for anyone who is predisposed to "having and getting" cancer.
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