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To: grey_whiskers; Tilted Irish Kilt; Qiviut; metmom

Do you have any information/link to what percentage of our immune system segments like IgG, T cells, P53, BRCA, etc. actually control? For example, the small study I saw found that loss of effectiveness of IgG4 averaged 20%, but was around 50% for a subset of subjects, who also seemed to be the ones who caught Covid. On the other hand, after the second mRNA Covid shot the IgG3 component practiclly disappeared, and that was the part with the strongest virus fighting power.

See also my Comment #81.


82 posted on 08/31/2023 12:09:42 PM PDT by gleeaikin ( Question authority!)
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To: gleeaikin

There are multiple overlapping issues; and the immune system is quite complex.

If you’re talking about the jabs, I’ve seen discussions of papers that show repeated jabs encourage (relatively) the proportion of IgG4 for the spike protein: this being the immunoglobulin that tells the immune system “hey, it’s foreign, but don’t sweat it” leading to tolerance of the antigen in question.

So, yay for no cytokine storm, but boo for allowing the virus to stay in the body (even if at levels leading to no obvious symptoms), allowing the jabbed to be incubators for mutations.

And the mutations will be more on the spike protein, not the nucleocapsid proteins, which are more highly conserved.

Leading to the new strains to infect the already-jabbed more, because (due to original antigenic sin), their bodies are primed to churn out antibodies to the spike protein from the jab—which don’t work as well against mutated strains. As opposed to the unjabbed, who will churn out antibodies to the spike, but also to the highly conserved nucleocapsid proteins—so thr antibodies will retain their effectiveness against the new strains...


84 posted on 08/31/2023 12:29:26 PM PDT by grey_whiskers ( The opinions are solely those of the author and are subject to change without notice.)
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