Q ~ Trust Trump's Plan ~ 10/31/21 Vol.378, Q Day 1465

qalerts.ne ^ | October 31, 2021 | FReeQs, FReepers, and vanity

[17strings]

I’m puzzled as to why this is news now, or what Project Veritas has to do with it. We saw those excerpts from the diaries on these threads a looooong time ago.

[ponygirl]

Nice to see the Arizona AG is asleep/slow-walking/intentionally dragging his feet on a lawsuit. And Brnovich thinks he’s going to be elected Governor. ha. ha. In fact, I think I need to send him an email....

[RitaOK]

Call me when you can ever arrive at the only 2 points of the complaint, dear.

P A C E (sluggish)

S E R I O U S C H A R G E S

[Bob Ireland]

***AS it is turning out, PR Executive -1 seems to be Charles Halliday Dolan, Jr. Mr Dolan currently serves on the board of the International Foundation for Electoral Systems***

Board of Directors:

Amb. J. Kenneth Blackwell, Chairman
Donald R. Sweitzer, Co-Chairman
June Langston DeHart, Vice Chairman
Thomas A. Devine, Vice Chairman
Amb. William C. Eacho, Treasurer
Randal Teague, Sr., Secretary
Wayne Allard
Kenneth Cutshaw
Hon. Irene Hadžiabdić
Hon. Steny Hoyer
William J. Hybl
Amb. Tom McDonald
M. Peter McPherson
Hon. Denise Nappier
Catlin O'Neill
H.E. Andrés Pastrana
Hon. Rob Portman
Daniel F. Runde
Amb. Theodore Sedgwick

[spokeshave]

[greeneyes]

This kinda makes me sick.

[bagster]

Call me when you can ever arrive at the only 2 points of the complaint, dear.

Ah, so this doom porn act of yours is just a ruse to get me to call you.

Get in line, sweetheart.

[Unrepentant VN Vet]

.

[Kalamata]

He is a profoundly evil man with an evil heritage.

[stylin19a]

as opposed to 29 armed FBI agents knew where Roger Stone was and raided his house anyway.

[stylin19a]

I wasn't quite right on this which is good for us. It puts the RATs feet to the fire which should scare those RATs up for re-election in the midterms.
https://www.washingtonexaminer.com/opinion/washington-secrets/gop-to-force-vax-mandate-vote-put-squeeze-on-moderate-dems

"The OSHA rule came down yesterday, starting a near three-week period of review. During that time, Braun plans to take advantage of the process to call a vote, which will be decided by a simple majority."

Under congressional rules, a vote is guaranteed, and that is likely to pressure Democrats forced to choose a side on the federal mandate. “Moderate Democrats in the House and the Senate will have to put on record if they support this federal overreach putting people out of work in their states,” said a Senate aide.

and this

President Biden’s vaccine mandate rule for private businesses was officially published this morning.

We are challenging this massive federal overstep in Congress.

No private company should be forced to make their employees choose between a jab and their job.— Senator Mike Braun (@SenatorBraun) November 5, 2021

[BlueMondaySkipper]

“ Not necessarily. If a red county delayed vote totals, waiting for Multnomah, it wouldn’t change anything unless the red counties had cheating methods at the ready.”

This “sort of” happened in Wisconsin with one of the Walker elections. One of the red counties reported their results officially, but the report they made to the media was missing a ton of votes. A big blue county then sent in their totals. Walker ended up winning and the left was pissed off that the red county gave the wrong totals to the media. The only reason that would matter is that they used the inaccurate totals as their guide on how many fraudulent ballots they needed, and the error left them short.

[little jeremiah]

At this point, OR is so rotten, and so many other states like CA, that arrests are the only answer.

[BlueMondaySkipper]

Mine did as well. My interview is scheduled for Monday 11/8.

A coworker was interviewed last week. She told me that they followed the same lines of questioning as Jet Blue airlines, which was discussed on the Coffee and Covid website. You might want to prepare by reading this.

https://www.coffeeandcovid.com/p/-second-religious-accommodation-primera

[BlueMondaySkipper]

Take a chill pill Eeyore.

Why is it that you do nothing but complain? People are (finally) getting arrested and here you are, still whining, Relax. Durham is methodically taking them down. This is exactly how these things work.

[stars & stripes forever]

AMERICA PRAYER VIGIL, November 9, 2021

[DollyCali]

Are you still in Aurora? Rita’s advice is good

[ransomnote]

TWiV 641: COVID-19 with Dr. Anthony Fauci - YouTube (37 minute video with TRANSCRIPT BELOW)

 

CLICK HERE TO jump to the 4:32 mark in the video, where where Fauci begins explaining that running PCR 'tests' at 35 cycles and above renders meaningless results.

 

ransomnote: Computer-generated transcript follows. These transcripts may contain phonetic errors and lack punctuation

 

Click to skip to the bottom of this long post 

 

this week in virology the podcast about

viruses the kind that make you

sick

[Music]

from microbetv this is twiv this week in

virology

641 recorded on july 16th

2020 i'm vincent raqueniello

and you're listening to the podcast all

about viruses

joining me today from austin texas rich

condit hi there vincent good to be here

and our very special guest today is the

director of the national institute of

allergy and infectious diseases dr

anthony fauci welcome back

thanks vincent nice to see you nice to

see you rich you were last on twitter

in 2013. boy have things have changed

huh

yeah a little bit so we wanted to ask

you

a bunch of science questions about

sarsko v2 and

kovid19 and we have we've put them in

little sections and this first section

is all about transmission and

and testing and i wonder if i could

start by asking what your thoughts are

on uh the relative contributions of

what we know are the three routes of

transmission droplets

aerosol and contact

you know you know i don't think it's in

events that there's really

firm firm data there's the assumption

that it's mostly droplets you know just

recently there's been a buzz

about the degree of aerosol and what

that really means

um there's it's almost impossible to be

able to determine

except when you have an event you might

recall very well the famous hotel in

hong kong with

sars where we had uh infections that

went up and down

vertically and across the courtyard to

the other hotel

i mean that's a surefire aerosol if

there ever was one but when you have

both droplet and regular aerosol

uh you know it's tough to distinguish

the two i would think

as an intelligent guess that it is

almost

a very large majority of it is the

standard

aerosol wears if you stay i mean the

standard droplet

which is if you stay six feet or more

away it's unlikely

that the droplet will reach you but i

would i would not

rule out the possibility that there is a

degree of aerosol the extent of which i

don't know

surfaces we know it can

stay on surfaces hard enameled steel

plastic more than corrugated cotton

material and things like that

you know a study that was done by

vincent munster from the rocky mountain

labs

showed about you know 48 to 72 hours on

surfaces but that's pcr-able we're not

so sure if it's

replication competent so that's another

thing that's kind of

up in the air yeah so no firm

information on what we're talking about

so do you have any

any feeling for like the relative

contribution of direct

droplet uh uh transmission

and surface mediated fomite mediated

you know i don't but if you look at the

circumstances

of when crowds are together you would

you would imagine it's mostly droplet

and i'm sure if somebody coughs into the

hand and opens the door if somebody

comes 15 minutes later

and opens the same door that it you know

it's a good chance

you're gonna get transmission but i

would think that it's again if you

compare

surface to droplet i would go with

droplet predominant

it seems to me this is important because

it speaks to the effectiveness of masks

right exactly exactly

all right there have been a number of

reports of patients who

shed viral rna for weeks as determined

by pcr

doesn't seem to be infectious virus and

the real question is are they a threat

for transmission and i'm wondering

if you think we could use a cut off of

viral loads determined by pcr to say

this patient

is no longer infectious can go home can

go to a nursing facility

because right now the the physicians are

really having a hard time with that

right again a good question and what is

now

sort of uh evolving into a bit of a

standard

that if you get a cycle threshold

of 35 or more that the chances of it

being replication competent are

minuscule

so that if somebody and you know we do

we have patients and it's very

frustrating

for the patients as well as for the

physicians

somebody comes in and they repeat

their pcr and it's like 37

cycle threshold but you never if you

almost never can

culture virus from a 37

threshold cycle so i think if somebody

does come in with 37

38 even 36 you got to say you know

it's just it's just dead nucleotides

period

yeah because as you know we can't easily

culture infectious viruses you don't

have a bso3 lab everywhere

exactly exactly okay so is the the

threshold cycle uh

uh is reporting that a pretty standard

practice and doing a diagnosis now

rather than just positive or negative

okay good yeah i mean if when you go in

when i get my test you know it's

negative when someone comes in and it's

positive

they don't give them the threshold until

you go back and ask for it

okay but they they've got it they've got

it

they've got it so uh

what's your current uh perspective on

the fraction of transmission now

that's either asymptomatic or

pre-symptomatic versus symptomatic

you know richard i know you're gonna

you asked that yeah i like these

questions because

there's no right or wrong answer because

we don't know

but we can guess so long as we don't

take our guesses too seriously

so you know i think it has to be

substantial uh if you combine

asymptomatic with pre-symptomatic i

think if you put them together

and you look at the circumstances in

which

it's spread you get individual

instances like what happened on the

teddy roosevelt

nuclear aircraft carrier where you had

a few symptomatic sailors and a thousand

sailors

got infected i can't imagine

richard that one symptomatic person

coughed on a thousand people and

infected them

it would almost have to be a substantial

amount of asymptomatic

carriage and transmission okay

so um this is a question

particularly relative to schools opening

i i see

stuff in the news that implies that some

people at least

think that children uh either acquire or

transmit

the virus somehow differently than

adults

and i'm wondering if there's really any

evidence for that

yeah good question i think that's one of

those data-free zones

i mean really i i don't think that there

is

conclusive evidence either way i mean

there is

it's out there that or certainly

children

don't get sick nearly as much as right

as adults the question is do they get

infected right

and if so do they transmit right as you

said that has really

important relevance to school opening

so the answer is we don't know

but what we're doing we have a study

that we started on may 1st

called the hero studies human

epidemiology and

and i forgot what the other things stand

for but it's going to be 6

000 people kids in 2000

families looking at the rate of

infection

the transmissibility to parents and

family members

and whether or not they get sick and

what their antibody response is okay

good so we we should know the answer to

that question

soon as opposed to just guessing because

i think

my my uh impression is that in the

popular

media that the the uh

uh understanding that kids don't get

sick as much gets

sort of misinterpreted or extrapolated

to the school system to say well

since they aren't getting sick then

putting them together back in school is

not a problem and

i don't that's not a logical conclusion

from the fact they don't get sick right

you're right you're right

so so in line with that

you know on our podcast twiv we get lots

of email from

teachers who want to know what's the

safest way to open schools in the fall

and

so how can we do that in a safe way get

these kids back but

what do we have to do

you know vince it's it's really i think

it

to address that that that question i i

generally take in multiple steps so

just give me 30 seconds i'll go quickly

is that i think a fundamental principle

should be that to the best of your

capability

given the circumstances which i'll get

to in a second you should try and get

the kids back to school

because what we're seeing is that the

you know the the downstream uh

unintended um

deleterious consequences ripple effects

of kids not being in school for the kids

for the parents for society

can be substantial so if you make your

default

we're going to try and get the kids back

to school having said that

it varies greatly where you are

you know we we tend to sometimes think

of this

and when when foreign countries and and

our foreign colleagues

look at the united states they tend to

look at us as a uni-dimensional country

as you guys know it's a big country and

it's really different geographically

demographically

so there may be some counties where heck

send the kids back to school don't worry

about anything and then there are other

areas where there's enough infection

you gotta decide either it's not safe to

get the kids back to school or

if it is you've got to do things that

mitigate

risk and that could be alternate day

classes morning afternoon wearing masks

protecting the vulnerables it's not one

answer

to a question it depends on a number of

different circumstances

but the one thing that's critical is

that if you're that the first thing is

paramount you have to be

you have to be attentive to and

sensitive to

the safety and the welfare of the

children and the teachers

and the teachers the reason i say and

the teachers

because you could have the situation

where you decide you want to bring the

kids back

and the teachers don't want to come back

i mean i think that's a reality that

could happen

so we're seeing uh in austin we're

seeing uh

demonstrations from teachers saying hey

we're here too yeah yeah so

it seems to me that testing should be a

part of

uh getting back to school and yesterday

we spoke with

michael minna from the broad institute

who

is a champion of the idea of having a

one dollar daily saliva based

antigen test and he thinks it can be

done with our amazing manufacturing

capabilities have

you ever thought about that and uh is

that ever going to happen

well you know maybe not specifically

what he spoke about but something along

that line

is really what is needed is something

that you could essentially do as many

tests as you

want it's instantaneous you get the

answer bingo you're done i mean we

really do need that i mean it would

alleviate a lot of anxiety number one

and number two

it would be an important public health

tool i mean if you could do that

before you send a group together into a

class or a group together into a factory

to work

i mean how great would that be knowing

that at that given moment

everybody was negative right

yeah this game in our conversation

yesterday this came down to

uh sensitivity questions okay uh

because he's talking about uh tests that

uh generally are less sensitive than pcr

but he says they don't have to be that

sensitive the pcr

as we've already discussed in in in some

senses can be

too sensitive so if you can catch most

of the really high virus load with uh

even a test that's not as sensitive that

that may be sufficient to really

significantly mitigate absolutely as an

example of don't let the perfect be the

enemy of the good really yes interesting

so we've seen uh reports from here and

there about the

notion that the virus may cause a

systemic infection certainly there are

systemic sequelae

okay but there have been reports

occasionally

indicating that the virus itself may

cause at least a small viremia

or actually infect the vasculature

or the kidneys or even the brain and i'm

wondering about your

uh take on that situation whether it's

really

confined as a respiratory virus and

those uh

sequelae a secondary or whether the

virus is getting out

you know my sense richard is that the

virus is getting out

although we don't have you know

overwhelming proof that that's the case

that we may be seeing

downstream distal effects of things that

are essentially respiratory

but what we are going to i believe get

good data on

are autopsy studies so if you start

seeing virus in different organs

that answers your question right there i

mean if you do autopsies and you see

endothelium and kidneys that have virus

in it

or liver or brain that's it it's it's an

open and shut case

okay and we're we're awaiting those

sorts of studies is that we are

we are okay all right uh and what's your

current best guess as to an infection

fatality rate

you know in the very very beginning of

this i wrote a perspective in the new

england journal of medicine

and i figured it was somewhere about one

percent um

it's totally dependent on the number of

asymptomatic infections

you know it could be five times a

particular

level if you're not counting all the

asymptomatics

you know given just the ballpark back of

the envelope type

look at what goes on you know

it's got to be it's got to be you know

obviously

more than seasonal flu but it's it's not

like

sars which was 10 or like mares which is

37

it's got to be somewhere around 1 i

think that

that fools people you know there are two

things that determine

the ultimate impact of an outbreak a how

many people get infected and b

how many die so if you get a few people

get infected

you know with a with a very high death

rate not a lot of people

die but if you get a lot of people

infected

with a reasonably low death rate a lot

of people are going to die right

so you know it's tough to calculate

because of the asymptomatics

so uh and let's talk a little bit now

about

immunity and and vaccines what's your

best estimate for the cereal prevalence

uh in the u.s i know it's going to vary

depending on where we are but overall

where do you think we are well new

york's 22 percent

so right away you're you're leading the

pack

uh because they really suffered terribly

i feel so badly about that you know i

think it's probably a few percent

three four percent something like that

that's what i think it probably is

so we're way way far from herd immunity

uh so uh that raises the question

can uh hurt immunity actually be

achieved without a vaccine this spanish

study came out and one of the reactions

to that

was that uh uh it just

wasn't even going to happen you know

i got my feeling is rich that if if

if we want to get hurt immunity

purely on the basis of infection

excluding

vaccine an awful lot of people are going

to get sick

and boy do i not want to see that i

don't want to see herd immunity that we

get

because we had 70 percent of the people

got infected and a whole bunch of people

got really sick that's the reason why

i'm putting my stock and

you know getting that vaccine as quickly

as we possibly can

so uh along the same line and this

impacts on the notion of

hurt immunity and this is one that i'm

really

confused about is uh reports that

uh immunity wanes um

and i'm interested in your perspective

on

uh in what if you know

or your perspective on what fraction of

infections

that generate immunity

that immunity wanes how fast

and and whether or not reinfection

really occurs

at a significant rate and what the

consequences of reinfection are is it

milder

because you still have partial immunity

what's the story

that's a lot of questions but i know but

they're very much related so usually i

can't remember

when somebody gives me five questions

it's okay i'll remind you

no but i think i can now because they're

quite related

so you know when you look at the history

of coronaviruses the benign

coronaviruses

go back in the old papers in the

literature the

the four common cold coronaviruses

the immunity did not last that long you

know it was lasting you know six months

a year or so

and that's the reason why people think

you get reinfected with coronaviruses

so that's the answer to the first

question what percentage of people get

immunity i think a hundred percent get

immunity

i guess the question is what percentage

lasts a reasonable period of time

and the answer to that is we don't know

but the reason we don't know for this

coronavirus

is that we're only six months into the

whole process

so if you really want to get a good

landscape view of what it is you got to

get wait a year or two and see

what happens answer your second question

the third question

is is it possible then if your immunity

wanes that you get re-infected

of course the question is if you get

re-infected

do you have a sub-optimal immunity

that then causes enhancement which is

what you're asking

i think buried in your question does it

make it better or does it make it worse

i don't think there was any any evidence

though i'm not sure people

looked in the common cold coronaviruses

if people get infected a second time

with the coronavirus do they get a worse

cold as it were so i don't know the

answer to that so that that's a big

unknown

i'm kind of hoping this is a i'm kind of

hoping that you

uh maintain some partial level of

immunity and actually get a milder

infection so

well i would hope so my goodness yeah

but no data no no like you say it's too

early

it's too early it's too early well you

know the

the current i environment does everyone

wants an answer yesterday right

yeah you know everyone wants an answer

you say i don't know they say well give

me a guess

yeah give them a guess they come back

they say you were wrong that's right

that's right so uh this notion of waning

immunity

that does that uh have any impact on

your perspective on how a vaccine might

work

not no not how it might work it's

whether you need to get boosted

okay yeah two or three i mean if you

look

we just not we but the group that did it

just published

my team but i'm not personally involved

in it

published two days ago in the new

england journal of medicine the phase

one data

and you know it really does look good i

mean you don't want to put all your

marbles in

50 45 patients this is the moderna

vaccine right

yeah the mrna so if you look at the the

uh

the immune response in a moderate dose

100 micrograms which was the moderate

dose

you get pretty robust neutralizing

antibody against

wild-type virus to the tune of as

high as or even higher than convalescent

serum

so at least for a finite period of time

guys

i think we're going to get a good

neutralizing response

how long it lasts we don't know

but you know i don't say this in a

flipping way i'll worry about that later

let me worry now about whether you can

get protection

over a season and if you can and the

immunity wanes then we'll worry about a

booster shot

so speaking of vaccines

[Music]

it seems to me that most not all but

most of them

are focused on the spike protein and we

talk about that a lot on

twiv and we'd love to get your

perspective do you think that's a good

idea to

not pay attention to all the other viral

proteins

it's a loaded question vince um

the answer is if you look at um

you know theoretically and conceptually

the neutralizing antibody that blocks

that binding to the ace ii

i mean classically that's the way you

block infection i don't

think that antibodies to other

uh components are unimportant

but i think that against the spike

binding site

is really i you know vince we had this

conversation years ago

about hemoglobin versus nuclear protein

versus neuraminidase with flu you know

and that's still a bit of an unanswered

question

even though you'd like to get it at the

hemoglobin i think the spike protein

antibody

it works it correlates with animal

protection so it all really looks pretty

good

although i do notice that one of your

warp speed candidates does have a few

other

viral proteins in there which is good

right yes yes so

do you know why immunity wanes

this speaks to the same question because

if if immunity wanes because the virus

tinkers with the immune system then

maybe that isn't an issue

for uh for a vaccine or or uh maybe it

maybe it is you know it gets back to

what you were saying about

viremia you know if this is truly

a respiratory borne infection i would

think that the immune response

would be weaker and would not last long

enough if you get somebody

you know what i would really like to see

and we'll get these data

you know as they evolve over the next

few months

i want to take somebody who gets

infected

gets up a respiratory infection a little

pulmonary

and then gets better versus somebody

that's in the hospital in an icu

with multi-system disease and find out

if the systemic descent dissemination of

the virus

really gives you immunity that that is

you know

years worth as opposed to months worth

that's okay

really interesting question okay so

track the track the

uh immunity relative to the sort of the

pathogenesis of their individual disease

that's interesting okay i i think that's

great idea because

you know the the viruses that give us

the best durable immunity are the ones

that are systemic like measles and polio

and i don't know maybe smallpox too

right rich

oh yeah i mean that's really durable

immunity

so uh tony what uh we have these uh

these things called warp speed and

active uh which are selecting vaccines

for

um upfront um payment and production

can you can you provide any insight as

to how these are

being selected or like what are the

criteria why are you why are you picking

them

so active is a

a public-private partnership of people

from different agencies of the

government as well as outside outside

investigators

who we bring together for the purpose of

doing just what you indicated of doing

prioritization

prioritization of agents prioritization

of studies

making sure that the resources are are

adequately and appropriately divided

among the different priorities

the operation warp speed is more of an

operational group that is looking at

diagnostics therapeutics and vaccines

predominantly focusing

right now on on therapeutics and

vaccines

to actually get involved in the the

conduct not that warp speed

conducts anything our group other groups

do it but to sort of outline

these are the protocols these are the

things that are going to match we've got

to harmonize the protocols

common database common dsmb

common immunological parameters common

primary and secondary endpoints

that all goes through warp speed more at

an operational way

whereas active is more of a think type

think tank type prioritization okay and

they're

obviously complementary functions then

and talking to each other

oh all the time yeah i mean they

intimated the boxes yeah they over the

boxes overlap a lot

yeah so active is

then participating in these decisions

about what the lead candidates are

absolutely absolutely and what are the

criteria

you know the criteria are what it looks

like

maybe in animal studies uh what the need

is

for the disease like for example i i'll

give you a good

a really good example so we have two

therapies now

that seem to work well in improving

either time to

discharge or mortality dexamethasone for

ventilated

people needing oxygen it's no good early

on

rem death severe better early on because

it's an antiviral

so what we really need right now are

agents

that operate early on that prevent a

sick person from going to the hospital

so

those are the ones we want to do so we

got convalescent plasma

hyperimmune globulin direct antivirals

monoclonal antibodies all those have

been prioritized and are all going into

clinical trials right now

that was on the basis which of all

deliberations about prioritizations

okay is it safe to say that in terms of

uh an an anti-viral approach a vaccine

therapeutic diagnostic there's never

been anything like this

oh for sure i mean this is you know i

don't like the word warp speed

because it makes it look like you're

carelessly rushing but

to to do things in the way we've done it

right now

is truly remarkable it's remarkable for

somebody like me who's been doing this

now

for 40 years and have been involved in

the development of a bunch of vaccines

back when we had a

you know get the the pathogen isolate it

grow it up then and doc inactivate it

attenuated

here you know the the the sequence came

out on the database

on january 10th i called a meeting in

this

room that we're in right now of our

people saying hey

let's go all hands on deck we got to

start developing vaccine the next day

we had a meeting by phone with moderna

put it into the mrna now we're on the

15th of april

five days have gone by amazing 62 days

later

we were in a phase one trial and then

five months later we're gonna start a

phase three trial

at the end of july that's mind-boggling

guys i mean

that's that's orders of magnitude more

quickly than we were doing

a decade ago so uh a question we get a

lot

is uh uh

uh vaccine production and deployment i

mean even if we get a successful vaccine

are we going to be able to deploy it to

enough people soon enough what's

what's the horizon there no the horizon

is good richard because

what what we have is that we're doing

something at risk

not at risk to the patient not at risk

to the scientific integrity

but at risk to the money so what happens

is that the federal government has

invested

hundreds of millions of dollars per

candidate

and sometimes over a billion dollars

per candidate to have the companies

proceed at risk in other words instead

of

determining if you're successful here

then investing here

then determining if you're successful

here it's like you invest

right ahead you prepare the sites you do

everything

you start making doses even before you

know

that the vaccine works which means that

if it does work

you've saved many months if it doesn't

work

you've lost a lot of money so the

urgency of the situation

dictates that it's worth investing the

money

to save a lot of time so the companies

tell us

i can't promise you personally because

that's not what i do i can get you

the vaccine and i'll tell you if it

works

the companies will say that they will

have

enough vaccine by the first quarter of

2021

to be able to start distributing tens if

not hundreds of millions of doses

incredible yeah i mean with the modern

vaccine

i've been trying to get my head around

how much rna that is

yeah and how to make it really i know

it's tough are we good let's go we're

done

they're telling me we got one more

question because i gotta run to the next

okay

i have one more question then so we all

knew that

this in fact the last twit you were on

we said what do you

worry about at night and you said are we

ready for the next big one

and i would guess you're probably

thinking we weren't ready enough

but i'm wondering you know part of the

problem is that

in academia which is where we are we

can't work on viruses that aren't

circulating and infecting people it's

hard to get support for that

so do you see after this pandemic

a change maybe in the direction of niad

so

we could have extramural funding for

basic science on emerging pathogens that

are

a little bit too risky uh for the normal

funding procedure

you know vince you probably are aware

that we've already done that

even before covert 19. you know we had a

program

of prototype pathogens platform

technologies

and pre-selected microbes we put a fair

amount of money in it

so we've been doing that what i think is

going to happen now

we i think set the example to do that

now the congress realizes

that for the next one we've really got

to be prepared

so i am seeing i hope fingers crossed an

infusion of a lot of money

into this type of preparedness that

sounds great yeah

all right the last thing i want to say

uh dr fauci

is i want to thank you personally for

everything

that you have done and i i probably

speak for rich condit and many

uh of us virologists out there we're

we're 100

200 behind you and we look forward to

your leadership

for many years to come thank you for

joining us today dr fauci

yeah absolutely thank you appreciate it

always good to be with you guys

thank you thank you thanks so much they

say you too

well that is a fabulous twiv

641 what do you think rich i just

i love that guy you know and i love

being able to talk to him like this as

i've

i i think uh you you see

the real tony fauci uh on this he's a

he's a scientist and he's a clinician he

cares about the science

he cares deeply about the science and he

cares deeply about the people

and about uh getting us done right and

he's not afraid to say

i don't know he's not afraid to say it's

a moving target

uh you know it's just a great person and

a great scientist and it's a privilege

to be able to spare

share some time with yeah we we really

appreciate

him taking the time to join us

i guess we are a bona fide thing and

wow of course he was on twit uh back in

2013 as you heard uh yeah that's uh

actually

something you need to put in the show

notes or even up front in the show

but at least here that seven years ago

we spent a whole hour with him in his

office and that's

uh definitely worth listening to i

re-listen to it it gives his whole

history

um which is uh actually quite

interesting yeah the history of his

involvement with the

aids epidemic uh and winds up with that

ringer question what keeps you up at

night and it was

this the big one yeah i know i think

it's uh it's a it's an interview you

won't

find anywhere else basically so yeah

check

who does our interviews right uh this is

twiv

641 you can find the show notes at dot

tv

slash twitter that will include the

links to

the old podcast send your questions and

comments it's with

microbe dot tv if you like what we do

consider supporting us microbe dot tv

slash contribute

our guest today of course from the

national institute of

allergy and infectious diseases was dr

anthony fauci

rich conditt rich conditt

is an emeritus professor university of

florida gainesville currently residing

in austin texas thank you rich you're

quite welcome i'll say it again it's a

privilege i'm

i feel very grateful for being able to

do this

bad for retirement right i'll tell you

retirement is

just rocks i never i never thought i

would do this much science in retirement

and it's some of them that's some of the

uh

best and most interesting science i've

ever done from my chair with a

headset on yeah we'll keep you busy

don't worry

great stuff great stuff i'm vincent

dracanello you can find me at

virology.blog i'd like to thank the

american society for

virology and the american society for

microbiology for their support of twiv

and ronald yankees

for the music this episode was recorded

edited and posted by me vincent

raqueniello

you've been listening to this week in

virology thanks for joining us we'll be

back

next week another twiv

is viral

[Music]

you

 

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