That is from the Covid 19 Vaccine Program Director David LV Bauer who heads up the program at the main Bio Weapons Lab in London, England that said.
Here David is in his own words explaining how it the Pfizer injection neutralizes the immune system.
https://banned.video/watch?id=60f89c07b4c876043c591c8a
https://www.medrxiv.org/content/10.1101/2021.03.07.21253098v
2 Direct quote below:
“In this study we profiled vaccine-induced polyclonal antibodies as well as plasmablast derived mAbs from individuals who received SARS-CoV-2 spike mRNA vaccine.
Polyclonal antibody responses in vaccines were robust and comparable to or exceeded those seen after natural infection.
At the monoclonal level, we found that the majority of vaccine-induced antibodies did not have neutralizing activity.”
“We also found a co-dominance of mAbs targeting the NTD and RBD of SARS-CoV-2 spike and an original antigenic-sin like backboost to seasonal human coronaviruses OC43 and HKU1.”
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OC43 and HKU1 are beta coronaviruses that produce colds and flus in humans; they're considered mild these days. But one of them, OC43, is believed to have been a serious pandemic flu in the 1890 timeframe.
And then there is this study from Nature Immunology:
https://www.nature.com/articles/s41590-020-00808-x.pdf
SARS-CoV-2-derived peptides define heterologous and COVID-19-induced T cell recognition
“T cell immunity is central for the control of viral infections. To characterize T cell immunity, but also for the development of vaccines, identification of exact viral T cell epitopes is fundamental.
Here we identify and characterize multiple dominant and subdominant SARS-CoV-2 HLA class I and HLA-DR peptides as potential T cell epitopes in COVID-19 convalescent and unexposed individuals.... snip....
Cross-reactive SARS-CoV-2 peptides revealed pre-existing T cell responses in 81% of unexposed individuals and validated similarity with common cold coronaviruses, providing a functional basis for heterologous immunity in SARS-CoV-2 infection. ...snip...
This T cell-mediated immune response is even more important as studies on humoral immunity to SARS-CoV-1 provided evidence that antibody responses are short-lived and can even cause or aggravate virus-associated lung pathology”...snip...
I think perhaps that Alex is inadvertently misstating the impact on the immune system, but it is correct that the immunity from the injection is inferior to that from natural immunity.
The existing vaccines are worthless for building durable immunity since the data is that the nucleocapsid section, which the vaccines do not code, is where most of the preexisting resistance against serious disease resides.
The resistance isn't to the spike, it's almost-exclusively to the nucleocapsid portion of the virus among those with existing resistance; the largest set of reactions by far was to the nucleocapsid, not the spike.
This is very strong evidence that it is that nucleocapsid reactivity that provides effective resistance to serious disease. The existing “vaccines” do not and cannot provide this since they encode only the spike.
We don't know for sure what the actual impact on the immune system's abilities are. But it is pretty clear that the injection can not generate what is needed for durable immunity—there is way more to that than antibodies.