Now. now, you know me better than this. And you are twisting what I said into a pretzel. There is no question that wild type covid induce a procoaguable state. You are attributing it to the spike proteins. Thrombosis is not generally diagnosed with D-Dimer and as I said, we use it as a marker of inflammation.
Please re-look at that I said, if indeed there I microcirculatory effects as is proffered in this article, D-Dimer is not the test to look at. Any inflamed state will induce a pro-coagulable milleu. There is a fair amount of nuance as to what I said, and I am certain you can understand the nuance.
"In the Covid era, we use it exactly as a marker of inflammation and to determine if anticoagulation should be considered, specifically."
To a lay person, anticoagulation == making clots go away. Low molecular weight heparin (bad with the coof, I've read), Coumadin , Eliquis (spelling), Xarelto. And you can't make clots go away unless there are clots present TO go away. So you'll have to explain specifically what's different between microclots in the capillaries (on physical grounds, that's where I'd expect them with the coof/spike protein), and generalized inflammation. We know the spike protein causes inflammation, btw, so what's the differential diagnosis other than loudly exclaiming "I DON'T BELIEVE IN CAPILLARY CLOTS, I'M A DOCTOR!" ?