Interesting
It explains a lot of the lab findings. The elevated D Dimer producing a “DIC” like lab presentation. The “circulating strands of clot” that are producing signs very similar to hemolytic anemia. Circulating micro strands of clot that are shredding red cells. Unchecked this leads to wide spread organ failure of all organ systems. But this only happens in a very small percentage of all those infected.
So what is the variable? Size of Innoculum? Immune Response? Pathogen rapidly shedding it’s pathogenicity? A combination of all of the above?