It explains a lot of the lab findings. The elevated D Dimer producing a “DIC” like lab presentation. The “circulating strands of clot” that are producing signs very similar to hemolytic anemia. Circulating micro strands of clot that are shredding red cells. Unchecked this leads to wide spread organ failure of all organ systems. But this only happens in a very small percentage of all those infected.
So what is the variable? Size of Innoculum? Immune Response? Pathogen rapidly shedding it’s pathogenicity? A combination of all of the above?
Interesting. I saw an interfering substance in blood bank too. I chalk it up to the liver damage causing abnormal proteins or side effects from the bacterial sepsis setting in.
Based on your explanations, what are the options (which may not actually exist) to treat it?
One other amateur question. There are pictures of patients lying in hospital beds in a ward in some type of self contained breathing device / environment, with the med staff in space-walking, moon landing type of suits. Why would an N-95 mask not be sufficient for the patient? For the med staff at this point?