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Ok. As a critical care anesthesiologist treating patients in this pandemic as well as numerous other pandemics, I absolutely see no difference in the presentation of this disease versus influenza ARDS. If we are to believe even the worse case scenario with a mortality of 15% for people over 80, this means that 85% of even the most vulnerable survive and recover. Additionally, as someone well educated in viral spread of disease, (the original meaning of something gone viral), I would bet that if we knew the seroconversion rate for this, we would find that the true number of cases if both significantly larger and has been around significantly longer than has been reported, which makes the true death rate and infection rate significantly less than reported currently.

I would bet that if we knew the seroconversion rate for this, we would find that the true number of cases if both significantly larger and has been around significantly longer than has been reported, which makes the true death rate and infection rate significantly less than reported currently.

Was thinking about seroconversion yesterday. Any idea when that might be known, based on your experience? I know you're not an epidemiologist, but wanted to ask.

Seroconversion rate has questionable value for this novel virus which has residues from purportedly 3 species of virus if the furin user residues identical to HIV and ebola are to be believed..as well as the SARS spike and an original report noted chimeric spikes of MErV as well dpp 4 and ACE 2..if you read much research on genetic engineering you will note that scientists have been concerned for years about the chinese reckless use of the CRSPR and a SARS escapee was reported briefly in 2017..the hybrid monkey pig and the genetic perfect baby as well as the attempt to make multivalent vaccines such as rabies and bat MERV are troubling to say the least..these papers are at pub med and are all authored by chinese..so IMHO regular view of this pathogen inciting a normal immunity curve over time will probably not be true..The micrograph show copious viral release and autoantibodies to asialo orthmuccinoids and a CNS glycoprotein as well as the long incubation with viral shed until not ambulatory.Plus its ability to infect a week after the MERV patient left th he hospital via fomite trasfer..see paper of from S Korea 2014.is a bad trait.and the derangement of the immune system which includes no permanent immunity and immunopathology T2 with eosinophila.upon secondary challenge.the typical midstage are lab results show huge interlukin 6 values and an initial leukopenia that make this a immunological nightmare..it just isn’t a normal virus so I respectfully disagree ..and people can be reinforced those papers are out there..this is after all a cold virus..but the second time can trigger the immunopathology..people keep saying its mild in 85 percent of the cases..true enough although many more european cases are seemingly severe and all ages over puberty are effected severely..

But what about reinfection?? That alone should negate glee from the 85 percent who aren’t old farts or sickly celebrating too soon..and got over this quick or so they thought...