[T.B. Yoits]

There are only two ways to prevent catching a viral disease. One way is to completely avoid exposure. If the virus spreads through the respiratory route, the only surefire way to avoid exposure is to stay indoors where all the air is filtered. The other way is to get vaccinated.

Proving the Jim Jones Jab isn't a vaccine because people who took it still got COVID-19⁸⁴.

The third way to prevent catching a viral disease is to have natural immunity from beating one of it's related strains. People who were exposed to SARS COVID in 2003 still had the antibodies in 2019.

The Branch Covidians suppressed the truth about natural immunity.

[exDemMom]

The third way to prevent catching a viral disease is to have natural immunity from beating one of it's related strains.

Catching a disease in order to avoid catching it makes absolutely no sense. This particular piece of misinformation is so illogical and ridiculous, I don't see how people fall for it in the first place.

It's also very dangerous.

If I am in danger of exposure to rabies, would it make any sense at all to go out and catch it deliberately? Well, that *would* prevent me from catching it in the future. But probably not in the way I want.

Disease-induced immunity is not as broad or protective as vaccine-induced immunity. In addition, no immunity against coronaviruses lasts for more than a few months. Ever catch a cold, then another cold, then another cold? You keep catching them because immunity is not permanent.

[exDemMom]

People who were exposed to SARS COVID in 2003 still had the antibodies in 2019.

Really?

First, where is/are the citation(s)?

I would like to know how the researchers are completely certain that those antibodies were actually induced by SARS1 and not one of the commonly circulating coronaviruses that cause the common cold? Antibodies tend to be cross-reactive. In my experience doing western blots, antibodies against a certain protein worked on extracts from a variety of species--human, mouse, rat, guinea pig, monkey, hamster, etc. And there was considerable cross-reactivity in each sample, as well, against proteins that were not the protein I was studying.

Since coronaviruses all have pretty much the same proteins, I would expect antibodies against coronaviruses to be fairly reactive against all strains.

Western blot analysis of G proteins of wheat seedlings grown in different light regimes.

This is a random western blot image that I pulled off the internet to illustrate the cross-reactivity of antibodies. Look at how many protein bands the antibody highlighted. The only way you can tell which protein is the "right" one is through size analysis (which is not included on this blot).

[grey_whiskers]

There's another way to avoid infection: if one's innate immune system handles the virus well enough that adaptive immunity does not get ramped up. YMMV, this is something which can be predicted ab initio.