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Posted on 01/26/2023 10:12:07 AM PST by ransomnote
Have you seen President Trump's powerful 4 minute video message? Don't miss it!
Q is the result of the sacrifices and commitment of countless patriots to win back our captured country from the Deep State and achieve the transformation President Trump promised in this campaign video. President Trump has said the awakening of the public is key to this transformation.
Q describes this awakening as follows:
"The Great Awakening ('Freedom of Thought’), was designed and created not only as a backchannel to the public (away from the longstanding 'mind’ control of the corrupt & heavily biased media) to endure future events through transparency and regeneration of individual thought (breaking the chains of ‘group-think’), but, more importantly, aid in the construction of a vehicle (a ‘ship’) that provides the scattered (‘free thinkers’) with a ‘starter’ new social-networking platform which allows for freedom of thought, expression, and patriotism or national pride (the feeling of love, devotion and sense of attachment to a homeland and alliance with other citizens who share the same sentiment).When ‘non-dogmatic’ information becomes FREE & TRANSPARENT it becomes a threat to those who attempt to control the narrative and/or the stable.
When you are awake, you stand on the outside of the stable (‘group-think’ collective), and have ‘free thought’.
"Free thought" is a philosophical viewpoint which holds that positions regarding truth should be formed on the basis of logic, reason, and empiricism, rather than authority, tradition, revelation, or dogma.
When you are awake, you are able to clearly see.
The choice is yours, and yours alone.
Trust and put faith in yourself.
You are not alone and you are not in the minority.
Difficult truths will soon see the light of day.
WWG1WGA!!!" ~ Q (#3038)
The video, Qanon is 100% coming from the Trump Administration, is just one of many excellent responses to the all-important question, "Whom does Q serve?"
Q Boot Camp is a quick, condensed way to learn the background and basics about the Q movement.
Q has reminded us repeatedly that together, we are strong. As the false "narrative" is destroyed and the divisive machinery put in place by the Deep State fails, the fact that patriotism has no skin color or political party is exposed for all to see.
In the battle between those who strip us our constitutional rights, we can't afford to let false divisions separate us any longer. We, and our country, will be forever made stronger by diligently seeking the truth, independence and freedom of thought.
Where We Go 1, We Go All
Impossible Foods Will Cut 20% Of Its Workforce
https://www.thefinancialtrends.com/2023/02/03/impossible-foods-will-cut-20-of-its-workforce/
"Finally, to those nations who would make themselves our adversary, we offer not a pledge but a request: that both sides begin anew the quest for peace, before the dark powers of destruction unleashed by science engulf all humanity in planned or accidental self-destruction."
Oil Heads For Second Weekly Loss As China’s Reopening Falters
Switzerland Directs Energy Suppliers To Set Aside 15% Of The Nation’s Annual Consumption In Reserve
Why? So they can figure out another way to kill us?
great article. ThankQ!
That's on us if we cant light big enough fires under the butts of our elected ones especially Kevin we will get what we deserve.
Without killing off major parts of voter fraud we don't have a country any more.
Hopefully, many of us are working at it now since the dems never stop! they are criminally working to take,steal, buy, do whatever it takes to win in 24. they are already concentrating heavily on it since they know they can't win or even get close on the "merits" of what they have done under biden/obama. That's a sure loser.
If we are ready to come after moochhelle right out of the box when they announce we have a shot at taking her/it out quickly.
Basically we need to start:
NOW!!
Great find,the drivebys have successfully made a hero out of the criminal zelensky.We need to get the truth out as best we can and as often as we can. Sooner or later they will move on to something/someone else. When they do maybe the money and arms flow will slow down.
Great stuff Bob thanks for posting the more we know the better we can counter these deadly bio weapons:
heres the entire link:
NEWS
What is the ACE2 receptor?
How is it connected to coronavirus and why might it be key to treating COVID-19? The experts explain
Krishna Sriram Paul Insel Rohit Loomba
By Krishna Sriram, Paul Insel and Rohit Loomba
May 16, 2020
In the search for treatments for COVID-19, many researchers are focusing their attention on a specific protein that allows the virus to infect human cells. Called the angiotensin-converting enzyme 2, or ACE2 “receptor,” the protein provides the entry point for the coronavirus to hook into and infect a wide range of human cells. Might this be central in how to treat this disease?
We are scientists with expertise in pharmacology, molecular biology and biochemistry, with a strong commitment to applying these skills to the discovery of novel therapies for human disease. In particular, all three authors have experience studying angiotensin signaling in various disease settings, a biochemical pathway that appears to be central in COVID-19. Here are some of the key issues to understand about why there’s so much focus on this protein.
What is the ACE2 receptor?
THE CONVERSATION
ACE2 acts as the receptor for the SARS-CoV-2 virus and allows it to infect the cell.
ACE2 is a protein on the surface of many cell types. It is an enzyme that generates small proteins – by cutting up the larger protein angiotensinogen – that then go on to regulate functions in the cell.
Using the spike-like protein on its surface, the SARS-CoV-2 virus binds to ACE2 – like a key being inserted into a lock – prior to entry and infection of cells. Hence, ACE2 acts as a cellular doorway – a receptor – for the virus that causes COVID-19.
Where in the body is it found?
ACE2 is present in many cell types and tissues including the lungs, heart, blood vessels, kidneys, liver and gastrointestinal tract. It is present in epithelial cells, which line certain tissues and create protective barriers.
The exchange of oxygen and carbon dioxide between the lungs and blood vessels occurs across this epithelial lining in the lung. ACE2 is present in epithelium in the nose, mouth and lungs. In the lungs, ACE2 is highly abundant on type 2 pneumocytes, an important cell type present in chambers within the lung called alveoli, where oxygen is absorbed and waste carbon dioxide is released.
What is the normal role ACE2 plays in the body?
ADAPTED FROM NEJM
The ACE enzyme converts angiotensin I into angiotensin II. The main role of ACE2 is to break down angiotensin II into molecules that counteract angiotensin II’s harmful effects; but if the virus occupies the ACE2 ‘receptor’ on the surface of cells, then its role is blunted (red lines). Drugs called ACE inhibitors inhibit the formation of angiotensin II, which would otherwise interact with the angiotensin type 1 receptor to produce tissue damage and inflammation. Drugs called ARBs block angiotensin II from interacting with its receptor.
ACE2 is a vital element in a biochemical pathway that is critical to regulating processes such as blood pressure, wound healing and inflammation, called the renin-angiotensin-aldosterone system (RAAS) pathway.
ACE2 helps modulate the many activities of a protein called angiotensin II (ANG II) that increases blood pressure and inflammation, increasing damage to blood vessel linings and various types of tissue injury. ACE2 converts ANG II to other molecules that counteract the effects of ANG II.
Of greatest relevance to COVID-19, ANG II can increase inflammation and the death of cells in the alveoli which are critical for bringing oxygen into the body; these harmful effects of ANG II are reduced by ACE2.
When the SARS-CoV-2 virus binds to ACE2, it prevents ACE2 from performing its normal function to regulate ANG II signaling. Thus, ACE2 action is “inhibited,” removing the brakes from ANG II signaling and making more ANG II available to injure tissues. This “decreased braking” likely contributes to injury, especially to the lungs and heart, in COVID-19 patients.
Does everyone have the same number of ACE2 on their cells?
No. ACE2 is present in all people but the quantity can vary among individuals and in different tissues and cells. Some evidence suggests that ACE2 may be higher in patients with hypertension, diabetes and coronary heart disease. Studies have found that a lack of ACE2 (in mice) is associated with severe tissue injury in the heart, lungs and other tissue types.
Does the quantity of receptors determine whether someone gets more or less sick?
This is unclear. The SARS-CoV-2 virus requires ACE2 to infect cells but the precise relationship between ACE2 levels, viral infectivity and severity of infection are not well understood.
Even so, aside from its ability to bind the SARS-CoV-2 virus, ACE2 has protective effects against tissue injury, by mitigating the pathological effects of ANG II.
When the amount of ACE2 is reduced because the virus is occupying the receptor, individuals may be more susceptible to severe illness from COVID-19. That is because enough ACE2 is available to facilitate viral entry but the decrease in available ACE2 contributes to more ANG II-mediated injury. In particular, reducing ACE2 will increase susceptibility to inflammation, cell death and organ failure, especially in the heart and the lung.
Which organs are most severely damaged by SARS-CoV-2?
The lungs are the primary site of injury by SARS-CoV-2 infection, which causes COVID-19. The virus reaches the lungs after entry in the nose or mouth.
ANG II drives lung injury. If there is a decrease in ACE2 activity (because the virus is binding to it), then ACE2 can’t break down the ANG II protein, which means there is more of it to cause inflammation and damage in the body.
The virus also impacts other tissues that express ACE2, including the heart, where damage and inflammation (myocarditis) can occur. The kidneys, liver and digestive tract can also be injured. Blood vessels may also be a site for damage.
In a recent research paper, we argued that a key factor that determines severity of damage in patients with COVID-19 is abnormally high ANG II activity.
What are ACE inhibitors? Are they a possible treatment or prophylactic for SARS-CoV-2?
Angiotensin converting enzyme (ACE, aka ACE1) is another protein, also found in tissues such as the lung and heart, where ACE2 is present. Drugs that inhibit the actions of ACE1 are called ACE inhibitors. Examples of these drugs are ramipril, lisinopril, and enalapril. These drugs block the actions of ACE1 but not ACE2. ACE1 drives the production of ANG II. In effect, ACE1 and ACE2 have a “yin-yang” relationship; ACE1 increases the amount of ANG II, whereas ACE2 reduces ANG II.
By inhibiting ACE1, ACE inhibitors reduce the levels of ANG II and its ability to increase blood pressure and tissue injury. ACE inhibitors are commonly prescribed for patients with hypertension, heart failure and kidney disease.
Another commonly prescribed class of drugs, angiotensin receptor blockers (ARBs, e.g., losartan, valsartan, etc.) have similar effects to ACE inhibitors and may also be useful in treating COVID-19.
Evidence for a protective effect of ACE inhibitors and angiotensin receptor blockers in patients with COVID-19 was shown in recent work co-authored by one of us - Dr. Loomba.
No evidence exists to suggest prophylactic use of these drugs; we do not advise readers to take these drugs in the hopes that they will prevent COVID-19. We wish to emphasize that patients should only take these drugs as instructed by their health care provider.
New clinical trial tests ACE inhibitor against SARS-CoV-2
In collaboration with a multidisciplinary group of investigators, Dr. Loomba has initiated a multicenter (randomized, double-blinded, placebo-controlled) clinical trial to examine the efficacy of ramipril - an ACE inhibitor - compared to a placebo in reducing mortality, ICU admission or need for mechanical ventilation in patients with COVID-19.
[Get facts about coronavirus and the latest research. Sign up for The Conversation’s newsletter.]The Conversation
This article is republished from The Conversation under a Creative Commons license. Read the original article.
hmmmm
Didn’t Trump revoke the “friendly skies treaty”?
In the context I've heard it, I took it to mean "then I understood" or "then I figured it out." So, I looked it up:
The term "once the balloon has gone up" derives from the First World War.
Whenever enemy activity was expected, observations balloons would be released to monitor the enemy troop movements. As a result, the raising of these balloons, which were visible to all, soon became a sign of pending enemy action.
Someone on this thread (sorry for not remembering who) posited that the Chinese don't need a spy balloon as they have satellites (and plenty of traitors on their payroll) so this cold war artifact is unnecessary.
I find it interesting that Brandon wanted it shot down but the generals refused. Who is really commander-in-chief?
The balloon has indeed gone up. I think it may be our "sky event."
Thanks bitt. That video must have dropped after my bedtime.
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