A little light reading.
Multiple jabs cause the body to respond to COVID-1984 with the IgG4 antibody, which does not neutralize, but teaches the body to tolerate the jab. The IgG3 antibody, which in the unjabbed does 42% of the killing, disappears entirely after the 3rd jab. This means those with multiple jabs become reservoirs for the virus for the rest of us (and since they have no killing immune response, the virus can mutate all it wants).
{Added by me: until cardiac events or cancer no longer stopped early by the body's screwed up immune response, gets them.}
Antibodies kill by recognizing / binding to (usually) strings of amino acids (the building blocks of protein) 6-8 units long. Regular flu (real vaccines) are like 89% effective against COVID 1984. So they share many regions in common. If other respiratory viruses learn to imitate COVID 1984 immune sequences in the places where our antibodies attack them (not a certainty, for a number of reasons, but respiratory viruses do show a lot of similarities) then a LOT of respiratory viruses will become endemic.
{Added by me: you can't go substituting amino acids willy-nilly without affecting protein shape and therefore function; but you might get away with it for the spike / binding region of other respiratory viruses, if they start out close enough to COVID-1984.}
{Added by me: To quote Ian Malcolm in Jurassic Park: Your scientists were so preoccupied with finding out if they could, they never stopped to ask if they should.
Or to paraphrase the late Isaac Asimov: the scientist were experts, which means they knew everything about one small subdivisional sliver of nothing.}
{Added by me: Knowing how to manipulate the mRNA including substitution of pseudouracil so the body wouldn't recognize and chew it up easily, didn't mean they recognized all the downstream effects, including continuing protein synthesis; nor the consequences of having all that spike protein all over the body; nor how all that spike protein would change the body's immune response to EVERYTHING ELSE.}
From the comments to the above, applicable to cancer.
IgG3 Vs IgG4 and the antitumor response.
I always like to cross check these things, to the immune system cancer cells are just another pathogenic invader to destroy.
As suspected this is important and the implications aren’t good.
B Cell Orchestration of Anti-tumor Immune Responses: A Matter of Cell Localization and Communication (2021)
somatic hypermutation (SHM)
“Additionally, analysis of more than 5,000 TCGA RNA-seq samples revealed that high levels of IgG3–1 switch are associated with prolonged survival in patients with high SHM rates, whereas IgG3–1 levels are not prognostic in low SHM samples, underscoring the role of SHM in generating BCR sequences with high binding affinity to the exposed tumor antigens (Hu et al., 2019).”
https://www.frontiersin.org/articles/10.3389/fcell.2021.678127/full
***
How to select IgG subclasses in developing anti-tumor therapeutic antibodies (2020)
“IgG3 demonstrates the highest affinity binding to most FcγRs”
“IgG4 only has high affinity for FcγRI but weak affinities for all other receptors, and is a poor inducer of Fc-mediated effector functions.”
https://jhoonline.biomedcentral.com/articles/10.1186/s13045-020-00876-4
My overall comment.
Smells from here like Marek’s chickens (vaccination of chickens caused the chickens’ immune system to stop killing the virus, resulting in the vaxxed chickens becoming incubators of all kinds of new strains which killed the unvaxxed.
That being turbocharged by having zillions of chickens all in very close quarters all cooped up. (No pun intended).
Move to a smaller town, don’t travel by mass transit.
In case y’all are interested: evolutionary biologist Bret Weinstein is discussing the IgG4 buildup after three to four shots and its repercussions in the Joe Rogan Experience episode #1919 on Spotify (around 30 minutes in)
Iggy-4 is getting quite the rep here lately.