You'd think they'd be all like, yep, there's them old clots again. It never gets old.
You'd think they'd be all like, yep, there's them old clots again. It never gets old.
Talks about 20% incidence of Deep Vein Thrombosis among COVID sufferers.
Potentially interesting quote:
Abnormally elevated levels of proinflammatory cytokines have been found in patients infected with the novel coronavirus [[52]]. The resultant increased systemic inflammation coupled with endothelial injury triggered by attachment of the virus to the angiotensin-2 receptor of the endothelial cells and viral replication leads to a prothrombotic endothelial dysfunction [[53],[54]]. Platelet activation, immobilization, mechanical ventilation and the use of central venous catheters are other factors that contribute to a prothrombotic state in COVID-19. Earlier reports have linked coagulopathy and development of TE with an increased risk of death [[3],[52]]. Autopsy studies have provided some essential insights into this prothrombotic state in COVID-19 [[34],[44],[55]]. A recent autopsy study found that almost no organ in the body is spared of thrombosis [[56]]. Regardless of anticoagulation status and sometimes early in the disease course, significant macrovascular and microvascular thrombosis was found in multiple organs.
Immobilization, mechanical ventilation, and central venous catheters are not in common with jab recipients: but the spike protein going throughout the body (*cough*) is.
The mechanism by which the jabs cause clotting, is probably very similar to the mechanism by which COVID-1984 causes clotting: they have the spike protein in common, after all.