https://www.bmj.com/content/375/bmj.n2635
Covid-19: Researcher blows the whistle on data integrity issues in Pfizer’s vaccine trial BMJ 2021; 375 doi: https://doi.org/10.1136/bmj.n2635 (Published 02 November 2021) Cite this as: BMJ 2021;375:n2635
Money quote:
In autumn 2020 Pfizer’s chairman and chief executive, Albert Bourla, released an open letter to the billions of people around the world who were investing their hopes in a safe and effective covid-19 vaccine to end the pandemic. “As I’ve said before, we are operating at the speed of science,” Bourla wrote, explaining to the public when they could expect a Pfizer vaccine to be authorised in the United States.1
But, for researchers who were testing Pfizer’s vaccine at several sites in Texas during that autumn, speed may have come at the cost of data integrity and patient safety. A regional director who was employed at the research organisation Ventavia Research Group has told The BMJ that the company falsified data, unblinded patients, employed inadequately trained vaccinators, and was slow to follow up on adverse events reported in Pfizer’s pivotal phase III trial. Staff who conducted quality control checks were overwhelmed by the volume of problems they were finding. After repeatedly notifying Ventavia of these problems, the regional director, Brook Jackson (video 1), emailed a complaint to the US Food and Drug Administration (FDA). Ventavia fired her later the same day.
Remember the "gain-of-function" / weaponization funded by Pfauci and/or Daszak in Wuhan?
Scientists in the US have been developing more harmful strains in mice to assist "in testing".
Kinda like the rationale for Daszak had in Wuhan, right?
Note to gassy and TrollHerd.
This article was published in Nature.
https://www.nature.com/articles/d41586-021-01251-0
Money quote:
Researchers have developed a strain of SARS-CoV-2 that triggers severe COVID-19 symptoms in mice — an advance that could accelerate the creation of drugs and vaccines and help to illuminate the biology of the disease.
Mice aren’t naturally susceptible to SARS-CoV-2 infection. That’s because the virus does not recognize the mouse version of a receptor called ACE2, which the virus uses to enter cells. [snip]
To develop a more faithful model, Vineet Menachery at the University of Texas Medical Branch in Galveston, Texas, and his colleagues first infected mice with SARS-CoV-2 strains adapted to the rodents, then collected the virus from the animals’ lungs, infected more mice and repeated the process. Such ‘serial passaging’ eventually generated a SARS-CoV-2 strain that caused significant weight loss, lung damage and other signs of disease.