combat_boots wrote:
“It’s possible to get NAD as a drip. I don’t, but the mental giants who came up with NAD hit pay dirt.
Then, there’s this:
https://www.zerohedge.com/medical/something-really-strange-happening-hospitals-all-over-america
“
Rodguy911 posted this about the hospitals:
https://freerepublic.com/focus/chat/4008617/posts?page=1207#1207
Something Really Strange Is Happening At Hospitals All Over America
November 2, 2021
Which story was put up first, zerohedge or themostimportantnews.com ?
I’ve had two relatives, one now dead and one in treatment, for this villainy. To think of red states targeted, all the people seriously debilitated, babies miscarried, and now, now even little kids going to be shot up with the frankenshots….
We’re prepping, but i personally want these perpetrators put through Iron Maiden’s and wood chippers…along with all their families, friends, hangers on, and people who owe them money. Kaiser S had the right idea, fictional though that character may be. I cannot imagine what life will be in 2025, let alone 2030. It’s monstrous.
We are under Judgment. Deservedly so. It grieves my soul. God, give me. strength. God help us all.
'We may be just starting to find out what is going on.
https://www.naturalnews.com/2021-11-02-science-horror-vaccine-spike-protein-enters-cell-nuclei-suppresses-dna-repair-engine-of-the-human-body-cancer-aging.html
SCIENCE HORROR:
Vaccine spike protein enters cell nuclei, suppresses DNA repair engine of the human body, will unleash explosion of cancer, immunodeficiency, autoimmune disorders and accelerated aging I have read numerous places where the bio-weapon not only suppresses the immune system but actually turns it against us.
Creations like this would have to take genius levels of medical knowledge to produce. Yet another reason I have used the words other worldly from time to time
. ..................
https://www.mdpi.com/1999-4915/13/10/2056/htm
This is from the Swedish studies which are getting lots of notice recently.
Here is a clip:
Abstract Severe acute respiratory syndrome coronavirus 2 (SARS–CoV–2) has led to the coronavirus disease 2019 (COVID–19) pandemic, severely affecting public health and the global economy.
Adaptive immunity plays a crucial role in fighting against SARS–CoV–2 infection and directly influences the clinical outcomes of patients. Clinical studies have indicated that patients with severe COVID–19 exhibit delayed and weak adaptive immune responses; however, the mechanism by which SARS–CoV–2 impedes adaptive immunity remains unclear.
Here, by using an in vitro cell line, we report that the SARS–CoV–2 spike protein significantly inhibits DNA damage repair, which is required for effective V(D)J recombination in adaptive immunity.(WTH)
Mechanistically, we found that the spike protein localizes in the nucleus and inhibits DNA damage repair by impeding key DNA repair protein BRCA1 and 53BP1 recruitment to the damage site. Our findings reveal a potential molecular mechanism by which the spike protein might impede adaptive immunity and underscore the potential side effects of full-length spike-based vaccines.
Keywords: SARS–CoV–2; spike; DNA damage repair; V(D)J recombination; vaccine
1. Introduction
Severe acute respiratory syndrome coronavirus 2 (SARS–CoV–2) is responsible for the ongoing coronavirus disease 2019 (COVID–19) pandemic that has resulted in more than 2.3 million deaths. SARS–CoV–2 is an enveloped single positive–sense RNA virus that consists of structural and non–structural proteins
[1]. After infection, these viral proteins hijack and dysregulate the host cellular machinery to replicate, assemble, and spread progeny viruses
[2]. Recent clinical studies have shown that SARS–CoV–2 infection extraordinarily affects lymphocyte number and function [3,4,5,6]. Compared with mild and moderate survivors, patients with severe COVID–19 manifest a significantly lower number of total T cells, helper T cells, and suppressor T cells
[3,4]. Additionally, COVID–19 delays IgG and IgM levels after symptom onset
[5,6]. Collectively, these clinical observations suggest that SARS–CoV–2 affects the adaptive immune system. However, the mechanism by which SARS–CoV–2 suppresses adaptive immunity remains unclear.
As two critical host surveillance systems, the immune and DNA repair systems are the primary systems that higher organisms rely on for defense against diverse threats and tissue homeostasis. Emerging evidence indicates that these two systems are interdependent, especially during lymphocyte development and maturation
[7]. As one of the major double-strand DNA break (DSB) repair pathways, non-homologous end joining (NHEJ) repair plays a critical role in lymphocyte–specific recombination–activating gene endonuclease (RAG) –mediated V(D)J recombination, which results in a highly diverse repertoire of antibodies in B cell and T cell receptors (TCRs) in T cells
[8]. For example, loss of function of key DNA repair proteins such as ATM, DNA–PKcs, 53BP1, et al., leads to defects in the NHEJ repair which inhibit the production of functional B and T cells, leading to immunodeficiency
[7,9,10,11]. In contrast, viral infection usually induces DNA damage via different mechanisms, such as inducing reactive oxygen species (ROS) production and host cell replication stress
[12,13,14]. If DNA damage cannot be properly repaired, it will contribute to the amplification of viral infection-induced pathology. Therefore, we aimed to investigate whether SARS–CoV–2 proteins hijack the DNA damage repair system, thereby affecting adaptive immunity in vitro.