[StAnDeliver]

"Since the early phases of the pandemic, computational models have been used to screen for drug molecules previously approved for other pathologies and disease conditions to be potentially repurposed against COVID‐19.[ 5 , 6 ] Via these approaches, drugs such as remdesivir or toremifene have been reported to show high binding affinities against SARS‐CoV‐2 proteins, including the spike protein or main protease"

This is a very strange description, to be charitable, of what remdesivir does to COVID. Remdesivir masks itself as adenosine (an essential amino acid in RNA), so as to interrupt this Chinese-engineered COVID virus' own near-perfect genetic repair machine.

Kind of like gluing down the “A” key on the amino acid keyboard, and then the virus thinks it's reading a correct "A" and skips repairing it. That failure to repair should force the virus to expire. Trump received remdesivir as well as the Regeneron REGEN-COV monoclonal antibodies, and dexamethasone.

[grey_whiskers]

so as to interrupt this Chinese-engineered COVID virus' own near-perfect genetic repair machine.

I wasn't aware that viruses in general were known for the quality of their genetic repair; let alone the COOF virus.

Do you have a link supporting this characterization, or was it sarcasm?

[grey_whiskers]

Do we have samples of the coof from patients after remdesivir has been administered, to look at protein samples to see how they got affected?

There's an article from PubMed from six months ago which posits an entirely different mechanism: not by substitution of an amino acid in a protein, but by halting RNA synthesis.

https://pubmed.ncbi.nlm.nih.gov/33436624/

[grey_whiskers]

(an essential amino acid in RNA)

Amino acids aren’t in RNA.
...didn’t you mean “nucleotide”?

Coffee not found error, perhaps?