That's one of our videos with our head dentist narrating. . .
Determining the presence of periodontopathic virulence factors in short-term postmortem Alzheimer's disease brain tissue.
Demonstration of a known chronic oral-pathogen-related virulence factor reaching the human brains suggests an inflammatory role in the existing AD pathology.
http://www.ncbi.nlm.nih.gov/pubmed/23666172
Effect of periodontal therapy on the subgingival microbiota over a 2-year monitoring period. I. Overall effect and kinetics of change.
CONCLUSION:
Periodontal therapy leads to a rapid reduction in periodontal pathogens, followed by a slower reduction in other taxa that can be sustained for at least 2 years.
http://www.ncbi.nlm.nih.gov/pubmed/23710672
Altered oral viral ecology in association with periodontal disease.
http://www.ncbi.nlm.nih.gov/pubmed/24846382
The next is an important study
The connection between ruptured cerebral aneurysms and odontogenic bacteria.
CONCLUSIONS:
This is the first report showing evidence that dental infection could be a part of pathophysiology in intracranial aneurysm disease
http://www.ncbi.nlm.nih.gov/pubmed/23761916
Oral biofilms, oral and periodontal infections, and systemic disease.
http://www.ncbi.nlm.nih.gov/pubmed/24479275
Invasion of oral and aortic tissues by oral spirochete Treponema denticola in ApoE(-/-) mice causally links periodontal disease and atherosclerosis.
This is the first study examining the potential causative role of chronic T. denticola periodontal infection and vascular atherosclerosis in vivo in hyperlipidemic ApoE(-/-) mice. T. denticola is closely associated with periodontal disease and the rapid progression of atheroma in ApoE(-/-) mice. These studies confirm a causal link for active oral T. denticola infection with both atheroma and periodontal disease.
http://www.ncbi.nlm.nih.gov/pubmed/24566627