” pangolin and bat SARS are not identical to SARS Cov2. And how odd this shows up, now, huh? “
The genetic similarity between SARS-2, Chinese Rhinolophus bat viruses, and Chinese pangolin coronaviruses was reported as early as March 2020. It’s not new.
The original 2002-2004 SARS outbreak was believed to have originated with Chinese miners coming into contact with Horseshoe bats and they have been subjects of interest ever since. SARS-2 is similar but not identical to the original SARS.
Bat virus RaTG13 has 96% similarity to SARS-2 but can’t infect human tissue. Pangolin viruses with 91% similarity have a spike that can infect humans. A recombination of the two viruses could produce a SARS-2 virus without human intervention. RNA viruses are unstable and can do that, much like flu.
https://bmcmedgenomics.biomedcentral.com/articles/10.1186/s12920-022-01208-w#:
https://www.nature.com/articles/s41586-020-2169-0
From your link
https://bmcmedgenomics.biomedcentral.com/articles/10.1186/s12920-022-01208-w
It’s footnote 2 claiming
“scientists think covid-1984 has a natural origin” refers back to the same Kristin Anderson.
And the reference 15 of your first paper
https://academic.oup.com/mbe/article/39/1/msab292/6382323?login=false
says this:
“Importantly, members of the relatively young (Boni et al. 2020) SARS-CoV/SARS-CoV-2 lineages (within Sarbecoviruses) do not yet appear to act as recipients in radical intertypic recombination events. They also display a very distinct AOF architecture. Thus, current evolutionary data do not favor a scenario where SARS-CoV-2 may (homologously) recombine with other currently circulating human CoVs of other subgenera/genera. Furthermore, SARS-CoV/SARS-CoV-2 do not seem to exchange accessory ORFs with other CoV subgenera or other viruses/hosts, with the exceptions of ORF3a that is an old and unresolved event and ORF7a (with some Decacoviruses).”
So this alone gives the game away: they’re just blowing smoke.
Troll.