Posted on 02/09/2020 2:16:57 PM PST by janetjanet998
There are currently 40,221 confirmed cases worldwide, including 904fatalities.
Daughter just texted that. They have to evacuate the ship.
My pool guy said ‘you can’t drink the water because it has stabilizers in it’... Turns out YOU are right hardpunned... and he was wrong. Nice to know in a flat out emergency... I’m glad I read your post - might come in handy someday.
https://modernsurvivalblog.com/preps/drinking-swimming-pool-water-in-an-emergency/
from link:
Personally, I would not hesitate to drink this water “as is” in an emergency, and honestly I have done so inadvertently more times than I can remember with no ill effects.
Of course, lack of daily circulation will cause fairly rapid degradation without some sort of action on the owner’s part. This would include continued chlorination as needed, with manual agitation. Also, putting on the cover would be wise.
Using something like a Lifestraw, the similar Sawyer product, or even better a Berkey water purifier (which can remove viruses, plus bacteria and protozoa) would add another layer of protection.
I think there are at least 4 effective anti-viral flu medications that could help you. You might consider asking your doctor if one of them is appropriate. I don't know that riding it out is a good choice, but I am not a doctor... Good luck and get well!
“Everybody has a plan until they get punched in the mouth.” — Mike Tyson
AT1 Receptor Blockers (“sartans”) for Severe Acute Respiratory Syndrome (SARS)The reason why the coronavirus kills in SARS is because of the exuberant host response, not because of tissue damage by the virus. Patients die of high fever and respiratory insufficiency. The lung interstitium is invaded by inflammatory cells, and alveoli fill with an inflammatory exudate. As a result, alveoli cease to become gas-exchanging units. Even in the absence of alveolar exudate, the distance between the alveolus containing oxygen-rich air and oxygen- transporting hemoglobin in the red cells of pulmonary capillaries widens because of the interstitial inflammation. Gas exchange becomes grossly impaired. Similarly, coronavirus does not cause fever; the body's immune response does. Both interleukin-1 (IL-1) and tumor necrosis factor-α (TNF-α) are the pyrogens causing the high fever. But these interleukins are made by the host's T cells and antigen-presenting cells (APCs), including activated macrophages. Decreasing the host's over-exuberant immune response to the coronavirus should reduce such symptoms. It is our belief that angiotensin II is an as yet unrecognized major stimulator of the immune response. The rate-limiting step for its synthesis is the angiotensin I-converting enzyme (ACE). ACE is present on the plasma membrane of T cells and appears on the plasma membrane of antigen presenting cells (APCs) such as monocytes and macrophages once they have become activated.
>>>Big Snip<<<
Two treatment possibilities appear promising. One is inhibition of ACE, but effective inhibition of tissue ACE requires a very high dose of ACE inhibitor, e.g. 2 mg/kg/d quinapril. Another possibility is selective AT1R inhibition using an angiotensin II receptor blocker (“sartan”) such as valsartan (DIOVAN), irbesartan (AVAPRO), losartan (COZAAR), candesartan (ATACAND), telmisartan (MICARDIS), or eprosartan (TEVETEN). The lowest dosage should be used, and even these tablets should be split in half to minimize the danger of excessive lowering of blood pressure in volume-depleted acutely ill patients. For example, an 80 mg DIOVAN capsule can be split in half, and 40 mg given once a day while the patient is in bed (e.g. at bedtime, or q am if the patient is already hospitalized). Irbesartan (AVAPRO) comes in 75 mg tablets which can be further split in half, and ̃37 mg given to the patient once a day.
https://www.google.com/url?sa=t&rct=j&q=&esrc=s&source=web&cd=35&ved=2ahUKEwjP0OWnr8bnAhVOs54KHTUuBccQFjAiegQILBAB&url=http%3A%2F%2Fwww.medicinacomplementar.com.br%2Fbiblioteca%2Fpdfs%2FDoencas%2Fdo-1588.pdf&usg=AOvVaw31HJx29A_A4X7uyTns2JGT
Sure, but the influenza antivirals are only effective when administered very early on.
” I don’t understand only 12 cases here. “
What saved us, at least so far, has been three things: most of the migration in early Jan would have been back to China or within asia for the annual holiday; the US taking quick action to first restrict then quarantine int’l travelers by Feb 4; and an incubation of 5-7 days to symptoms/pneumonia.
The Jan 29 March ARB quarantined folks should be clear by Feb 10 if you count the day at the airport before they left and flight time into their total. From touchdown, tho, it’s Feb 12, so early this week.
If I’m thinking this right, we go from from traveler-origin high-risk to low risk about Feb 13, if everyone who arrived via aircraft between Jan 28 and Feb 3 remains virus free. Leaving the risk limited to the few person-to-person contacts of the existing 12-current-patients. Maybe we’ll be breathing easier, in the USA at least, by Feb 14?
—
13 Jan - First case in Thailand. With 7 day inoculation, he would have been infected about Jan 8. Anyone he infected by the 13th would show symptoms by the 20th. 14-days clear from Jan 8, would have been Jan 27 - after lockdown in Wuhan and well after the US began enhanced screening.
15 Jan - first case Japan - would have been infected about Jan 9th. Anyone he infected before the 15th would have shown symptoms by the 22nd. Those who stayed uninfected would be clear abt Jan 29.
17 Jan - US enhanced screening at 3 major pop center airports
21 Jan - First US case. Would have been infected approx. seven days prior. Anyone he infected by the 15th would show symptoms by Jan 22. Those who stayed unexposed from Jan 15 should be clear by Jan 29.
Jan 22 - US announces enhanced screening. Anyone infected by those passengers would show symptoms by Jan 29. Those who remained unexposed on Jan 22 would be clear by Feb 4.
23 Jan - Wuhan lockdown - Those still without symptoms would be clear by Feb 5 but too late to beat the US Feb 4 closeout
Jan 28 - US flights redirected to 11 specific airports. Those still without symptoms at airport screening on that date would be clear by Feb 12. And any exposed on that date would show symptoms by Feb 4.
Feb 4th - US flights restricted - quarantine of travelers in place. These last minute folks would show symptoms by Feb 12, without symptoms would be clear by Feb 19.
You do need to be examined for a secondary bacterial infection. Don’t let the pneumonia get started...
66 now.
ok, I finally found time to squeeze the numbers in the JAMA study, so here goes - critique away:
numbers from Zhonghan Univ Hospital, study period Jan 1-31. 138 patients, JAMA paper Feb 7.
https://jamanetwork.com/journals/jama/fullarticle/2761044?c
Did not require ICU and have not died: 102 = 74% but 85 remain hospitalized = 62%
Discharged by Feb 3: 47 = 34%; avg hosp stay 10 days
Remain hospitalized: 85 = 62%
Casualties: 6 (4.3%)(ICU deaths) (30-day period)
ICU admissions: 36 = 26%
ICU - Transferred out to general ward: 10 = 37%
ICU deaths: 6/36 = 17% (preexisting/shock/organ failure)
Overall mortality: 4.3% (ICU deaths) (hospital admitted mortality rate not total mortality rate)
To reach 600/day mortality from hospitals with the figures above, (138/6dead) could we multiply 138 x 100 for a patient load of 13,800 with 600 admitted each day to keep things static with the overall mortality still at 4.3% and a 17% mortality rate of the 26% admitted to ICU? Is this right? But we need to know how many hospitals the 600 is spread over because a daily patient load of 14K is a lot of beds and 600 new patients each day is a lot of daily admissions.
If we look at a 600/day mortality as a combination of untreated deaths at home and hospital deaths then can’t we also guestimate the number of infected overall, then number of seriously ill, and the number of untreated and the number of mild infected/recovered overall? And I accept that these numbers to start with are wonky because they are coming from CCP, but it’s a start?
We’re asking for those folks over there that will, or even if they can do nothing any longer but motion their mouth as Samuel’s mother, talk to the Greatness One, of Romans chapter 1.
“If it’s been more than 14 days since the departure (from China) and no crew members are showing symptoms,” Newberry said, “then the ships are allowed to enter the port.”
If it has been less than 14 days and there are no symptoms of illness on board, ships are allowed to enter the port to conduct normal operations, he said, but with restrictions for crew members.
Those restrictions include no open shore leave, Newberry said, though crew members — which usually number 15 to 20 per vessel — can leave the ship to procure provisions and conduct tasks related to vessel operations.
But if any crew member shows symptoms before entering the port, Newberry said, that person’s vessel will be denied entry....
https://www.dailybreeze.com/2020/02/04/new-federal-regulations-battling-spread-of-coronavirus-could-affect-cargo-cruise-ship-industries-at-ports-of-la-and-long-beach/
If I get to drinking the pool water it means the local water supply has stopped or is contaminated. If that’s the case, electricity is probably gone as well. I’m assuming my pool water, without circulation, filtration and chemicals (including stabilizer) will degrade fairly rapidly. I’d planned on using my life straw gravity feed system for drinking and cooking. In the situation above the pool water will be vital for replacing the water for toilets. Losing sanitation services would quickly becomes a huge problem.
BNO Newsroom
@BNODesk
·
3h
BREAKING: 60 new cases of coronavirus on cruise ship near Tokyo, raising ship’s total to 130 - TBS
BNO Newsroom
@BNODesk
Japan’s health minister says they’re now considering to test all 3,700 passengers and crew members for coronavirus -
https://twitter.com/BNODesk/status/1226738251366567936
According this young man, there were no extra screenings or instructions to self-quarantine at the LAX airport on Feb 6.
Flying in from Taiwan, previously in Hong Kong, previously in China, he was asked at departure in Taiwan if he had been in China in the last 14 days and on arrival at LAX if he had been in China in the last 30 days. He said nobody verified what he verbalized. His is not the first video posted about this topic; there was another one about a Chicago-bound traveler who was also has claimed there is nothing special said to them and nobody is taking temps or ‘enhanced screening’, and noone is telling them about a self-quarantine. That entry into the US was ‘anti-climatic’ compared to the checks they’d experienced on their routes here. This particular fellow intends to continue on to Mexico City from LA.
It would seem this is a critical issue that needs to be addressed tootsweet if we are to protect ourselves from citizens/residents who may try to play the system and inadvertently spread infection. Our BP/customs folks need to make it clear to entering passengers that they do need to self-quarantine.
https://www.youtube.com/watch?v=IiL0l2pAFFI
The PTB’s have essentially given up control of this. They’ve determined we will all get this regardless of what is done. So what has ‘been done’ is for kabuki purposes only.
Thanks to you both.
Thankfully, as of this morning it feels like I might survive after all.
But should this relief prove illusory, I’ll certainly go pester my GP with it.
Fingers crossed . . .
Good timeline, thank you. It can be as much as 14 days from infection to symptoms.
Disclaimer: Opinions posted on Free Republic are those of the individual posters and do not necessarily represent the opinion of Free Republic or its management. All materials posted herein are protected by copyright law and the exemption for fair use of copyrighted works.