N-Acetyl Cysteine or just plain Cysteine itself 600 mg/day or 1K mg, and if one is high blood pressure treated— ACE blockers... work to block the ACE2 receptors on lung lining cells which the virus Spike S Proteins on the surface of coronavirus seek out (when virus inhaled) as the “key” to insert the S protein into the “lock” of the ACE2 receptor— which then lets the rna/dna material inside the virus particle virion— into the lung cell for infection.
Block the ACE-2 receptor with prescription Losartan or Candesartan (generic names). ACE inhibitors quinapril and others as an alternative but direct dosed ACE blockers add another layer to all the prevention of infection actions of the supplements. You’ll also never get the flu, whatever it is this coming year (which oddly during covid was a “zero” flu virus year— how convenient and how untrue).
By Francis L. Poe and Joshua Corn
Med Hypotheses. 2020 Oct; 143: 109862.
Published online 2020 May 30. doi: 10.1016/j.mehy.2020.109862
Abstract (excerpts): In vivo, in vitro, and human clinical trials have demonstrated N-acetylcysteine (NAC) as an effective method of improving redox status, especially when under oxidative stress. In human clinical trials, NAC has been used to replenish glutathione stores and increase the proliferative response of T cells. NAC has also been shown to inhibit the NLRP3 inflammasome pathway (IL1β and IL18) in vitro, and decrease plasma TNF-ɑ in human clinical trials. Mediation of the viral load could occur through NAC’s ability to increase cellular redox status via maximizing the rate limiting step of glutathione synthesis, and thereby potentially decreasing the effects of virally induced oxidative stress and cell death. We hypothesize that NAC could act as a potential therapeutic agent in the treatment of COVID-19 through a variety of potential mechanisms, including increasing glutathione, improving T cell response, and modulating inflammation.