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I don't think COVID-19 is foremost a respiratory disease; it looks like an epithelial disease. The virus is bonding with the ACE2 receptors, which is interfering with the body's ability to keep free radicals in check. This is causing O₂^. to form, which is probably combining into H₂O₂, because the ACE2 enzymes are blocked from doing their job.

The Glutathione antioxidants are supposed to provide the missing electrons to the O₂^. or help reduce the hydrogen peroxide into oxygen and water and replenish the body's supply of Glutathione, but the level of Glutathione in seniors can't keep up with the rapidity of the virus in blocking the ACE2 receptors from keeping the oxidation in check in the early stages. This is why it appears to affect seniors the most, because of their degraded ability to produce Glutathione.

Once the harmful O₂^. and H₂O₂ run wild, it presents itself as cardiovascular in the end as lung and blood issues. We know that by the time people have breathing problems and need ventilators, it's often too late.

But what this doctor is showing is what is going on before the onset of "long COVID." That's the window of opportunity to stop the oxidative distress by boosting Glutathione with IV NAC before it can do damage to the rest of the body.

MedCram is not a kook site. Watch the molecular biology videos by an MD professor and ICU doctor.

-PJ

If you had been reading at PubMed you would have known all of this well over a year ago. It’s not new, except here at FR, where kook sites and talk radio are the primary source for covid info. Go over to PubMed and use the search function and you’ll be swamped with studies. eg:

PubMed Sept 2020:

“Severe acute respiratory syndrome coronavirus 2 (SARS-CoV-2) infects host cells through angiotensin converting enzyme 2 receptors, leading to coronavirus disease (COVID-19)-related pneumonia, and also causing acute cardiac injury and chronic damage to the cardiovascular system. “

Pubmed July 2020:

“Initially, SARS-CoV-2 infects cells in the respiratory system and causes inflammation and cell death. Subsequently, the virus spreads out and damages other vital organs and tissues, triggering a complicated spectrum of pathophysiological changes and symptoms, including cardiovascular complications. Acting as the receptor for SARS-CoV entering mammalian cells, angiotensin converting enzyme-2 (ACE2) plays a pivotal role in the regulation of cardiovascular cell function. Diverse clinical manifestations and laboratory abnormalities occur in patients with cardiovascular injury in COVID-19, characterizing the development of this complication, as well as providing clues to diagnosis and treatment.”