I could not immediately find anything talking to its anti-inflammatory benefits, but I did see that it has some cardio-protective benefits beyond just it normal function.
I also saw that it is closest to the ARB Telmisartan, for such uses.
Ramapril is in the class of ACE inhibitors. I googled “does ramapril have anti inflammation properties”?
Here is 2014 NIH study that concludes
This study determines that a brief episode of ACE inhibitor therapy through 5 mg/day ramipril exerts comprehensive anti-inflammatory effects on vessel wall inflammation in AAA disease. The effect is followed by a general reduction of cell activation.Dec 4, 2014
https://www.ncbi.nlm.nih.gov/pmc/articles/PMC4256371/#:~:text=This%20study%20determines%20that%20a,general%20reduction%20of%20cell%20activation.
Abdominal aortic aneurysm (AAA) occurs when atherosclerosis or plaque buildup causes the walls of the abdominal aorta to become weak and bulge outward like a balloon. An AAA develops slowly over time and has few noticeable symptoms.
https://www.radiologyinfo.org/en/info/abdoaneurysm#:~:text=Abdominal%20aortic%20aneurysm%20(AAA)%20occurs,and%20has%20few%20noticeable%20symptoms.
Ibesartin is an ARB. I googled “does ibesartin have anti inflammatory properties”?
Irbesartan, an angiotensin II receptor blocker (ARB), acts as a selective PPAR-γ (peroxisome proliferator-activated receptor-γ) modulator, and thus may have anti-inflammatory and antioxidative effects, as well as beneficial effects on glucose and lipid metabolism.Feb 21, 2013
https://www.nature.com/articles/hr20133#:~:text=Irbesartan%2C%20an%20angiotensin%20II%20receptor,on%20glucose%20and%20lipid%20metabolism.
Then I googled. “ACE inhibitor inflammation vs ARB inflammation”
This randomized study tested the hypothesis that ACE inhibitors and ARBs differ in their effects on oxidative stress, inflammation, and fibrinolysis in MHD patients. The major findings are that although short-term treatment with an ACE inhibitor or ARB decreased IL-6 and IL-8, ramipril also exerted a proinflammatory effect during hemodialysis, as measured by an increase in IL-1β and a decrease in the anti-inflammatory cytokine IL-10. Both ramipril and valsartan decreased the marker of thrombosis, D-dimers, but ramipril also prevented a rise in vWf, a marker of endothelial damage, during hemodialysis. These effects are not attributable to an antihypertensive effect of ramipril or valsartan, because neither drug affected BP in the MHD studied participants.
https://www.ncbi.nlm.nih.gov/pmc/articles/PMC3269170/
While the whole was mixed above — the key take away was “ but ramipril also prevented a rise in vWf, a marker of endothelial damage”