You’re still wrong. This time you’re confusing antigens with antibodies. Antigens are pieces of foreign substances or organisms which provoke an immune response. Antibodies are part of the immune response. They bind to the invading pathogen in such a way as to disable or kill it. Two different things.
The T cell antigen receptor is used to bind to pathogen major histocompatibility complex sites and initiate biochemical responses (including recruitment of CD4 and CD8 co-receptors) so the pathogen or infected body cell can be destroyed.
It does NOT carry an antibody. That would not make any sense. If it had an antibody attached to the antigen receptor, the T cell would not be able to bind to the MHC and would become useless. What you’re describing is more along the lines of an autoimmune disease.
“This time you’re confusing antigens with antibodies.”
You’re a moron.
I am not confusing antigens with antibodies.
T cell antigen receptors are membrane associated antibodies with antigen recognition sites created via vdj rearrangement. B cell antigen receptors have two antigen recognition sites. T cell antigen receptors have one.
My use of the term antigenic determinant sites may have confused you and caused you to make your comment, and recognition rather than determinant would be better, but in the context of the phrase “vdj derived antigenic determinant” would not confuse anyone knowledgeable of molecular immunology.
(See how nice I can be you you?)