Evidence of Antibody Dependent Enhancement? – Majority of Covid deaths are the fully vaccinated and A&E attendance is breaking records

Daily Expose ^ | 06/21/2021 | Staff

[grey_whiskers]

Hey, troll-boi.

You lied about the first study.

Your clear implication is that they goosed the concentration of spike protein in the injection, in order to be able to lie about "spike protein causes clots" since the concentration they used, isn't found in human vaccinations.

But if you bother to read THE INTRO TO THE F'ING PAPER, in the results section up top, it says:

SARS-CoV-2 and its Spike protein directly enhanced platelet activation such as platelet aggregation, PAC-1 binding, CD62P expression, α granule secretion, dense granule release, platelet spreading, and clot retraction in vitro, and thereby Spike protein enhanced thrombosis formation in wild-type mice transfused with hACE2 transgenic platelets, but this was not observed in animals transfused with wild-type platelets in vivo.

Their first part of the study was in vitro "in glass" and not even living animals.

The second part was in vivo "in living" {creatures}

Notice the live mice had been infused with human ACE2 transgenic platelets.

They then went on to say (in the very next sentence):

"Further, we provided evidence suggesting that the MAPK pathway, downstream of ACE2, mediates the potentiating role of SARS-CoV-2 on platelet activation, and that platelet ACE2 expression decreases following SARS-COV-2 stimulation. SARS-CoV-2 and its Spike protein directly stimulated platelets to facilitate the release of coagulation factors, the secretion of inflammatory factors, and the formation of leukocyte–platelet aggregates. Recombinant human ACE2 protein and anti-Spike monoclonal antibody could inhibit SARS-CoV-2 Spike protein-induced platelet activation."

1) stimulation with coof virus causes platelets to DECREASE the # of ACE2 receptors -- this has been pointed out by others, including scientists who you trolls derided.

2) They kept track of what inflammatory molecules were released by platelets when you mixed them with coof virus and spike protein.

3) They proposed ways to cut down on the platelet activation.

In other words, they were trying to study the mechanisms of clotting, not in order to find out whether there would be clotting.

Another example of a troll parroting "BECAUSE SCIENCE!"™ without any idea what in the living hell they're reading.

[palmer]

1) stimulation with coof virus causes platelets to DECREASE the # of ACE2 receptors -- this has been pointed out by others, including scientists who you trolls derided.

2) They kept track of what inflammatory molecules were released by platelets when you mixed them with coof virus and spike protein.

3) They proposed ways to cut down on the platelet activation.

When did I do #1? #2 is the same as studying the clotting. The live mice result means even less of a threat from spike protein.

[grey_whiskers]

When did I do #1?

"You trolls" is plural.

#2 is the same as studying the clotting.

Which is NOT the same (there you go with your ambiguous weasel language again) as "falsely claiming a low threshold concentration for clotting by putting your finger on the scale". Which is what you were accusing them of doing, or egging me on to do "If I had any chops"...

TROLL.

The live mice result means even less of a threat from spike protein.

The live mice result means they were not trying to model the conditions under which the threat would arise, but instead, to make SURE the clotting happened, so that they could find what pathway was causing it, and therefore how to remedy it.