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LEARNING FROM THE PAST:

Viruses do mutate at a particularly high rate, especially ones like SARS-CoV-2 that use RNA for their genetic material.

Unlike organisms that use DNA, these viruses don’t have the ability to fix mistakes that happen when they copy their genetic code.

This means that RNA viruses tend to evolve more quickly than other organisms, says Richard Kuhn, a virologist and director of the Purdue Institute of Inflammation, Immunology and Infectious Disease in West Lafayette, Indiana.

However, the bulk of the mutations that appear as a virus spreads are either harmful to the virus itself (meaning it is less likely to survive or replicate) or don’t change how it functions.

Additionally, the two characteristics that are of most concern right now—how contagious the virus is and how harmful it is to its hosts—are controlled by multiple genes, Grubaugh says. Changing these characteristics is a complicated process, and not often the work of a single mutation. So the odds of the virus mutating in such a way that it actually becomes more lethal or contagious over the timescale of weeks, months, or even a couple years aren’t very high.

In past outbreaks of other diseases such as SARS or Ebola, scientists haven’t seen evidence that a particular virus was mutating to become more deadly.

“While there may be some mutations from time to time that arise that do have an epidemiological impact, it’s not very often that we actually find them,” Grubaugh says.

this SARS has four segments of HIV
and a locus of RNA c/w its unnatural origin.

the important questions unanswered are:

1. how many “recovered” continue to superspread virus?

2. how many “recovered” get easily reinfected by variants
which have mutated? (two scores already)

3. how many of the “recovered” now have diminished/altered
lymphatic/immune systems for the rest of their lives
as planned by those who made this atrocity, and how
many of those will get leukemia, cancer and other
susceptibilities from the PLANNED,
pandemically disseminated, Wuhan Coronavirus?

“Unlike organisms that use DNA, these viruses don’t have the ability to fix mistakes that happen when they copy their genetic code.”

This is wrong.

SARS class ACE2-binding viruses have a proof-reading subunit.

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The protein nsp15 is a 3’-5’ exoribonuclease which provides extra fidelity to the replication process. The exoribonuclease provides a proofreading function to the complex which the RNA-dependent RNA polymerase lacks. Similarly, proteins nsp7 and nsp8 form a hexadecameric sliding clamp as part of the complex which greatly increases the processivity of the RNA-dependent RNA polymerase.[46] The coronaviruses require the increased fidelity and processivity during RNA synthesis because of the relatively large genome size in comparison to other RNA viruses.[49]

https://en.wikipedia.org/wiki/Severe_acute_respiratory_syndrome-related_coronavirus