“You might want to tell that to the Italians.”
It’s not hidden from them.
https://www.nature.com/articles/s41421-020-0147-1
https://link.springer.com/article/10.1007/s00134-020-05985-9
https://www.ncbi.nlm.nih.gov/pubmed/27082314
What we’re saying is that the percent difference in the genetic expression of that ONE enzyme makes NO difference in the face of an epidemic.
Once your ICU’s are full...chaos ensues.
And even 50% of your population having the genetics to overexpress ACE2 is enough to do that.
It’s not like the d32CCR5 mutation and HIV.
And in THIS coronavirus, the whole genetic predisposition thing wrt ACE2 may be moot:
https://www.sciencedirect.com/science/article/pii/S0166354220300528
“The spike glycoprotein of the new coronavirus 2019-nCoV contains a furin-like cleavage site absent in CoV of the same clade”
This virus has ‘tricky business’ not found in any other coronaviruses that are close relations. (ie, the other ‘common cold coronaviruses, mers, and sars)
Coronavirus far more likely than Sars to bond to human cells due to HIV-like mutation, scientists say
https://www.scmp.com/news/china/society/article/3052495/coronavirus-far-more-likely-sars-bond-human-cells-scientists-say