Sorry to hear about your wife, but you should be able to put together yummy food. As I see this playing out, we’ll have power for a long time, possibly right through, so having frozen/refrigerated foods (like cheese and ketchup) is possibly...and I’ve found that just putting one or both of them on just about anything works great (particularly melted American cheese...and yes, American CHEESE, not ‘Singles’)!
AT1 Receptor Blockers (“sartans”) for Severe Acute Respiratory Syndrome (SARS)The reason why the coronavirus kills in SARS is because of the exuberant host response, not because of tissue damage by the virus. Patients die of high fever and respiratory insufficiency. The lung interstitium is invaded by inflammatory cells, and alveoli fill with an inflammatory exudate. As a result, alveoli cease to become gas-exchanging units. Even in the absence of alveolar exudate, the distance between the alveolus containing oxygen-rich air and oxygen- transporting hemoglobin in the red cells of pulmonary capillaries widens because of the interstitial inflammation. Gas exchange becomes grossly impaired. Similarly, coronavirus does not cause fever; the body's immune response does. Both interleukin-1 (IL-1) and tumor necrosis factor-α (TNF-α) are the pyrogens causing the high fever. But these interleukins are made by the host's T cells and antigen-presenting cells (APCs), including activated macrophages. Decreasing the host's over-exuberant immune response to the coronavirus should reduce such symptoms. It is our belief that angiotensin II is an as yet unrecognized major stimulator of the immune response. The rate-limiting step for its synthesis is the angiotensin I-converting enzyme (ACE). ACE is present on the plasma membrane of T cells and appears on the plasma membrane of antigen presenting cells (APCs) such as monocytes and macrophages once they have become activated.
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Two treatment possibilities appear promising. One is inhibition of ACE, but effective inhibition of tissue ACE requires a very high dose of ACE inhibitor, e.g. 2 mg/kg/d quinapril. Another possibility is selective AT1R inhibition using an angiotensin II receptor blocker (“sartan”) such as valsartan (DIOVAN), irbesartan (AVAPRO), losartan (COZAAR), candesartan (ATACAND), telmisartan (MICARDIS), or eprosartan (TEVETEN). The lowest dosage should be used, and even these tablets should be split in half to minimize the danger of excessive lowering of blood pressure in volume-depleted acutely ill patients. For example, an 80 mg DIOVAN capsule can be split in half, and 40 mg given once a day while the patient is in bed (e.g. at bedtime, or q am if the patient is already hospitalized). Irbesartan (AVAPRO) comes in 75 mg tablets which can be further split in half, and ̃37 mg given to the patient once a day.
https://www.google.com/url?sa=t&rct=j&q=&esrc=s&source=web&cd=35&ved=2ahUKEwjP0OWnr8bnAhVOs54KHTUuBccQFjAiegQILBAB&url=http%3A%2F%2Fwww.medicinacomplementar.com.br%2Fbiblioteca%2Fpdfs%2FDoencas%2Fdo-1588.pdf&usg=AOvVaw31HJx29A_A4X7uyTns2JGT