Posted on 06/11/2010 4:05:34 PM PDT by Dallas59
The then-Blue Mound police officer treated his wound with hydrogen peroxide and rubbing alcohol but the damage, he would learn later, had already been done.
Bradshaw contracted hepatitis C through the bite. Last fall, he learned the virus had developed into cirrhosis of the liver and Stage 4 liver cancer.
Now 57, Bradshaw sleeps with his cellphone on his pillow, hoping for news that his wait for a liver transplant is over. He has chosen the fight song from Texas Tech — where he went to school — as his ringtone because it was the loudest one he could find.
"I want to hear it," said Bradshaw, whose 26 years in law enforcement included stints with the Tarrant County Sheriff's Department and the Willow Park Police Department.
"The anticipation kills you. Everyday you go to bed with a prayer. Everyday you wake up with a prayer."
Read more: http://www.star-telegram.com/2010/06/11/2257353/police-officer-bitten-by-drunk.html#ixzz0qaXp91d2
(Excerpt) Read more at star-telegram.com ...
There will be victims along the way to “true liberty”.
(”True liberty”: being able to do with one’s body as he or she pleases without interference from God or civil government).
I’ve had it and the treatment is horrible but it’s better than being dead. Not by much though.
You would need to show me proof the poor man didn't down a whole lot of booze. Father-in-law died of same problem and he put away a 1/2 to a whole case a day every day.
Dang if I know!!!
Booze doesn’t give a person Hep.C, a virus.
Maybe it was a gift? Personally, this gentleman has more class and strength of character than most people, considering he is a facing a certain and very painful death. Read the rest of the article!
A nurse with whom I worked for years in an ER suffered a needlestick during a code and a few months later she tested positive for Hep C and HIV.So,in health care facilities,patient/staff cross infections can run both ways.
Did not say it did, and I have seen no proof that it gives cancer to anyone 25 years after the fact. Now if you have a medical study link proving it I would be interested.
The reverse will work just as well, you’ve seen no proof that a virus could NOT be present for 25 years before causing cancer.
And if you’re not inclined to do any searching, It took me only a few minutes to locate the following to get started:
Hepatitis Central
Duke Researchers Show How Hepatitis Infection Leads To Liver Cancer
DURHAM, N.C.
Hepatitis B and C infections slowly eat away at a person’s liver, severely damaging liver function and greatly increasing the risk of liver cancer. Now researchers at Duke University Medical Center have discovered the hepatitis virus makes the liver into a cancer time bomb by converting the organ into billions of cancer-prone cells.
The finding, published in the Sept. 16 issue of the Proceedings of the National Academy of Sciences, demonstrates that once a hepatitis infection takes hold in the liver, even apparently healthy cells have lost one of two copies of a protective tumor suppressor gene called M6P/IGF2R, making them highly vulnerable to further genetic damage. Without a working copy of this suppressor gene, cancerous cell growth can’t be stopped...........................
www.hepatitis-central.com/hcv/hcc/toc.html -
South Med J. 1997 Sep;90(9):872-7.
Chronic hepatitis C.
Sharara AI.
Department of Medicine, Duke University Medical Center, Durham, NC 27710, USA.
Abstract
BACKGROUND: Infection with the hepatitis C virus (HCV) is a leading cause of chronic liver disease worldwide. Epidemiologic and natural history studies have helped to define the clinical impact of HCV infection, and molecular diagnostic assays have established clinical endpoints against which therapeutic regimens are currently tested. The absence of definitive therapy has generated controversy regarding indications and optimal candidacy for currently approved treatment. This paper reviews the epidemiology, natural history, clinical manifestations, diagnostic modalities, and current treatment of chronic HCV infection. METHODS: Search of the MEDLINE database for English-language articles and abstracts on chronic HCV infection yielded data from more than 500 original papers, reviews, and abstracts. RESULTS AND CONCLUSIONS: Hepatitis C virus is transmitted primarily through contaminated blood and less effectively by human body secretions, but a large proportion of patients have no clearly identifiable parenteral risk factors for viral acquisition. Infection with HCV results in subclinical chronic hepatitis in the majority of patients and may progress, usually over decades, to cirrhosis and hepatocellular carcinoma. Extrahepatic manifestations of HCV infection include porphyria cutanea tarda, mixed essential cryoglobulinemia, and membranoproliferative glomerulonephritis. Diagnostic modalities are accurate in estimating viral load and genotype and may be helpful in predicting and assessing response to treatment. Current therapy is limited to interferon alfa and is effective at viral eradication in only a small number of patients. The adjuvant use of drugs, such as ribavirin, in combination with interferon may hold promise at enhancing viral eradication. Understanding the mechanisms behind viral persistence and immune escape of HCV will be essential in developing effective future therapeutic and preventive strategies.
PMID: 9305294 [PubMed - indexed fo
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