I don’t disagree with you, but:
1. Gays really have been “out” for about a generation. Before that, they generally had a double life. One generation is not long enough to breed them out.
2. Being queer could be a genetic predisposition, like psycophrenia or some depressions. Many, many people who have the genetic predisposition don’t end up symptomatic.
3. My hunch is it is a combination of genetic and environment, just like most things.
That’s pretty much the conclusion reached in the lik I posted.
Here’s a brief look at the summary:
http://www.mygenes.co.nz/summary.pdf
Genetic content of homosexuality is minimal.
Geneticists, anthropologists, developmental psychologists, sociologists, endocrinologists, neuroanatomists,
medical researchers into gender, and twin study researchers are in broad agreement about the role of
genetics in homosexuality. Genes don’t make you do it. There is no genetic determinism, and genetic
influence at most is minor.
Is this consensus likely to change? Might some major biological link be discovered which could change
everything? After all, science is about discovery
For most of these scientific disciplines, the findings have been clearly established from facts that will
not change (eg. the diversity of homosexual practices between and within cultures; the clearly established
stages of human development; the over-riding role of upbringing in the ultimate gender choice of people with
ambiguous genitalia). But what of future studies of brain micro-structure, or detailed analysis of genes and
function? Will they reveal links between brain structure and human behaviours, or behaviours and genetic
sequences?
Of course they will. Papers will continue to be published. But we can safely conclude that even authors
wanting to find such links will almost always include the standard scientific caveats that the influence is
minor, and that the environment is important. What we can reasonably say about future research is that it will
enter new fields and come up with new links, but none of them will be definitive.
This is proved once and for all by studies of identical twins. They have identical genes, and other
influences, but if one is homosexual the identical brother or sister usually isn’t. There is only an 11 % chance
he/she is homosexual. This includes all the influences we know nothing about and are yet to discover.
All added together only have a rather weak effect.
The first edition of this book in 1999 floated a trial balloon: the genetic content of SSA will ultimately turn
out to be 10%. That is quite imprecise, and could be in the range 0-20% - allowing for a 10% margin of error.
But even if the final result is 20%, this is still a weak influence. Even at the time of writing, late 2007, this 10%
conclusion still holds, and the evidence is stronger than in 1999. And don’t forget, any genetic influence is
indirect....
What is the cause of SSA?
There is no one cause. No single genetic, hormonal, social, or environmental factor is predominant. There
are similar themes: childhood gender non-conformity, sexual abuse, peer dynamics, family dynamics,
sexual history, but the mix varies with individuals, making the individual’s response to life’s events the single
overriding factor. Random reactions of children to their circumstances are important. The overriding outcome
of these contributors is often a sense of feeling “different” and withdrawal from gender norms, along with
admiration and envy and a drive to reconnect that focuses on certain individuals and becomes confused after
puberty with genital sex. This sexual response to emotional signals can become a repeating pattern, leading
to type-casting (“I am a homosexual”), and not infrequently, to addiction.
4. Most homosexuals (incl lesbians) say they were molested as children.