Guess I'll have to cut my intake of this to just one per day.
Seems that the use of corn for ethanol feedstock makes all the more sense.
".. but Brawndo has 'Electrolytes'!"
Ya know a conspiracy kook might conclude collusion amongst agribusiness and pharmaceutical business to rape the peasants and peons here....but that would be crazy talk...
People who consume lots of sugar tend to have higher blood pressure? Could it be because they tend to be fat? Duh! Sugar is sugar, despite what the diet-loons tell you.
Guess I'll have to cut my intake of this to just one per day.
Personally I don’t like HFCS. I consume it because it’s in so many products, but generally I don’t like it. I was thinking about yogurt and how when it first started getting popular in the 70’s or 80’s, it tasted so much different. A mainstream brand not only has HFCS but sugar too.
A lot of Americans have died to keep the Fanjul family’s monopoly in place...
I won’t talk about safety, but a few years ago, Mrs. Springman and I went to Canada, she couldn’t believe how much better the pop, (soda) tasted.
They use real sugar, not HFCS.
I haven’t had it in 17 years.
Not to mention the fact that High Fructose Corn Syrup uses Mercury in it’s production, oh yes, it’s true, look it up ...
Processed sugars (heck, processed carbs in general) are the culprit, don't get fooled by the quasi-conspiracy hype.
later
Chances are you consume quite a bit of fructose. Most Americans do—in refined sugars such as sucrose or table sugar (which is half fructose) and in high-fructose corn syrup, used in products as diverse as soft drinks, protein bars, and fruit juice.
But many scientists believe that high dietary fructose contributes to the development of metabolic syndrome, a group of risk factors that predict heart disease and Type 2 diabetes.
"For this reason, it's important for scientists to understand exactly how consuming high amounts of fructose affects human health," said Nakamura, a U of I associate professor of food science and human nutrition.
Nakamura's lab is continuing to study the metabolism of fructose with an eye to making recommendations about its dietary use.
His study shows that the metabolism of fructose is more complex than the data had indicated. "Our gene-expression analysis showed that both insulin-responsive and insulin-repressive genes are induced during this process. Our bodies can do this, but it's complicated, and we may pay a price for it," he said.
According to the scientist, most carbohydrates are handled fairly simply by our bodies. They are converted quickly to glucose and used for energy or stored as fat. "When we are eating, blood sugar--and insulin production--goes up. When we sleep or fast, it goes down," he said.
The process is not so simple with fructose, he noted. "In order for fructose to be metabolized, the body has to create both fasted and fed conditions. The liver is really busy when you eat a lot of fructose."
Because, unlike glucose, fructose metabolism occurs mainly in the liver, Nakamura wanted to gain a complete picture of gene expression in the liver during fructose metabolism.
In Nakamura's study, 24 rats were fed either a 63 percent glucose or fructose diet four hours a day for two weeks; at the end of this period, half the animals fasted for 24 hours before the scientists performed a gene expression analysis; the other half were examined at the end of a four-hour feeding.
Fructose feeding not only induced a broader range of genes than had previously been identified, there were simultaneous increases in glycogen (stored glucose) and triglycerides in the liver.
"To our surprise, a key regulatory enzyme involved in the breakdown of glucose was about two times higher in the fructose-fed group than in the glucose-fed group," Nakamura said.
The study also suggests that a protein called carbohydrate response element binding protein is responsible for the fructose effect on certain genes that trigger the production of fat, he said.
"We're continuing to assess the risk of fructose insulin resistance and the consequent risk for development of diabetes," he said.
Co-authors of the study, published in a recent issue of Biochimica et Biophysica Acta, are Hyun-Young Koo, Matthew A. Wallig, Takayuki Y. Nara, and B. H. Simon Cho of the University of Illinois and Byung Hong Chung of the University of Alabama at Birmingham.
foods such as fruit juices, soft drinks, bakery products, and candy were included
In other words, as intake of processed foods has gone up, obesity has gone up. Big surprise. Many other factors influence obesity. It's absolutely silly to blame one specific food ingredient for obesity. Sorry, but there is no quick and easy fix to the obesity epidemic; if there were, there wouldn't be an obesity epidemic.
What I don't see in this study is any kind of comparison to people eating the exact same foods sweetened with a different sugar.
--HFCS can contain varying amounts of fructose, from less than 50% to 90%.
--Sucrose contains exactly 50% fructose.
--Fruits and vegetables contain varying amounts of fructose.
So where is the actual study that compares groups with different levels of fructose consumption, keeping every other factor constant (especially total sugar content), that supports a causative relationship between fructose consumption and obesity?