Another poster said: “Some people find that once they start eating bread, cookies, chocolate, potato chips — or leftover Easter candy — they lose all sense of fullness and find it difficult to stop.”
The poster I replied to responded by saying this: "This is the metabolic mechanism of high fructose corn syrup, which is now used in everything."
I asked him to expound on the "metabolic mechanism" in HFCS that supposedly makes people lose all sense of fullness thereby preventing them from being able to stop eating. He couldn't so you linked me to some wacky website that still doesn't answer the question.
Now, are you trying to tell me that because fructose is converted into glucose by the liver that it causes people to lose control of their ability to manage their caloric intake? Is that an argument you really want to make?
People get fat because they eat more than they burn. Overeating is a learned behavior and has nothing to do with evil chemicals or food ingredients. Obese people may want to blame it on something other than overeating but that nonsense cannot be supported.
Your body processes fructose differently.
Differently than what: Galactose? Starch? Lactose? Glucose?
So what? Your liver converts fructose to glucose and it all ends up in the Krebs cycle at the same level.
I think that the jury is still out on whether it is actually harmful
Based on what, some wacky website that doesn't even understand that corn syrup is made up of glucose and not fructose? If fructose is harmful then you have to believe that fruit, fruit juice, honey and regular old table sugar are also harmful. Is that what you believe? Eating too much of anything can be bad for you. People die from drinking too much water.
but there is no argument, not even from you, on whether the human body has to work differently to process fructose.
Why is this bad? Can you also defend the statement that HFCS causes people to lose all sense of fullness and that they become unable to stop consuming food?
What I'm telling you is that fructose is metabolized in a different manner than glucose or sucrose and as such, some people have difficulty in maintaining a proper blood sugar level.
The point is that many food product manufacturers went about replacing sucrose with fructose as if it were exactly the same thing. It isn't.
People get fat because they eat more than they burn.
Absolutely
Overeating is a learned behavior and has nothing to do with evil chemicals or food ingredients. Obese people may want to blame it on something other than overeating but that nonsense cannot be supported.
And now you had to go and say something dumb.
Different sugars are processed differently by the body. The amount of that difference varies from person to person. To say in a general statement that the only reason people are obese is because they eat too much is just dumb. Granted, for many people, the way the body converts fructose versus sucrose makes a negligible difference. But for others, the difference between the body's reaction to consuming fructose and sucrose can cause all manner of difficulty.
So what? Your liver converts fructose to glucose and it all ends up in the Krebs cycle at the same level.
Yes and if I pee in your soup it's all going to end up with you peeing the liquid out.
Why is this bad? Can you also defend the statement that HFCS causes people to lose all sense of fullness and that they become unable to stop consuming food?
Well, how about this statement:
"Both plasma insulin and leptin act in the central nervous system in the long-term regulation of energy homeostasis. Because fructose does not stimulate insulin secretion from pancreatic ß cells, the consumption of foods and beverages containing fructose produces smaller postprandial insulin excursions than does consumption of glucose-containing carbohydrate. Because leptin production is regulated by insulin responses to meals, fructose consumption also reduces circulating leptin concentrations. The combined effects of lowered circulating leptin and insulin in individuals who consume diets that are high in dietary fructose could therefore increase the likelihood of weight gain and its associated metabolic sequelae. In addition, fructose, compared with glucose, is preferentially metabolized to lipid in the liver. Fructose consumption induces insulin resistance, impaired glucose tolerance, hyperinsulinemia, hypertriacylglycerolemia, and hypertension in animal models."
So, unless you want to claim that the American Journal of Clinical Nutrition is just another "wacky website" you might want to tone down your attitude and do some reading.
This isn't some conspiracy theory. My wife had the darndest time losing weight, even on an 800 calorie a day diet. She ended up in a study at Duke University Medical Center and was diagnosed by their team of endocrinologists as insulin resistant.
By eliminating all most complex carbohydrates and returning to a diet in meats, vegetables and simple sugars she lost 110 lbs.
So who am I going to believe, you, or my lying eyes, Duke University Medical Center and the American Journal of Clinical Nutrition?