So, here’s the data:
He had anti-spike antibody, but not anti-nucleocapsid (whole virus) antibody. It is not known in what order various antibodies appear after natural infection.
He acquired natural infection in the hospital and died shortly afterwards. It is normal to be viremic early, and the finding of diffuse organ involvement by SARS CoV 2 indicates that this was the case.
The paper does not make clear if his spike antibodies were vaccine-induced or the result of an early response to natural infection. The fact that he was PCR negative on day 18 and positive 6 days later after an exposure may indicate that the infection was early, but IgG takes a while to appear.
Biopsies of the colon indicated ischemic colitis, which is often rapidly fatal.
SO, for the flubros, he probably died WITH COVID, and not OF COVID. He was viremic before he died, consistent with early infection. He was not shown to have target organ (lung) injury from COVID, although they looked for it. Instead, he had multiple lung abscesses, probably from his rapidly dying colon.
His spike antibodies were IgG class, they probably were from his vaccine at 4 weeks out. They may in fact have prevented attachment to lung ACE-2 receptors, which is what they are supposed to do. They certainly did not prevent viremia.
Interesting case. Not conclusive regarding ADE, not impossible but <50% in my opinion.
He was old, on death’s door before he got the shot and before he got the virus.
Great analysis! Thanks for posting.
Were you saying that the spike vaccine could “protect” the ACE-2 lung receptor cells while leaving other cells (organs) open to infection?
Ischemic == no blood flow.
Is this seen often from the coof?
...or might it be a side effect of the jab, with the spike protein initially causing micro-clotting in the intestines, which then got worse?
Yes, I'm spitballing here.
Very interesting! Thank you!